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Items: 1 to 20 of 99

1.

Myocyte-specific overexpressing HDAC4 promotes myocardial ischemia/reperfusion injury.

Zhang L, Wang H, Zhao Y, Wang J, Dubielecka PM, Zhuang S, Qin G, Chin YE, Kao RL, Zhao TC.

Mol Med. 2018 Jul 17;24(1):37. doi: 10.1186/s10020-018-0037-2.

2.

Transgenic overexpression of active HDAC4 in the heart attenuates cardiac function and exacerbates remodeling in infarcted myocardium.

Zhang LX, Du J, Zhao YT, Wang J, Zhang S, Dubielecka-Szczerba PM, Wei L, Zhuang S, Qin G, Chin YE, Zhao TC.

J Appl Physiol (1985). 2018 Oct 4. doi: 10.1152/japplphysiol.00006.2018. [Epub ahead of print]

PMID:
30284520
3.

HDAC4 degradation mediates HDAC inhibition-induced protective effects against hypoxia/reoxygenation injury.

Du J, Zhang L, Zhuang S, Qin GJ, Zhao TC.

J Cell Physiol. 2015 Jun;230(6):1321-31. doi: 10.1002/jcp.24871.

4.

Irisin plays a pivotal role to protect the heart against ischemia and reperfusion injury.

Wang H, Zhao YT, Zhang S, Dubielecka PM, Du J, Yano N, Chin YE, Zhuang S, Qin G, Zhao TC.

J Cell Physiol. 2017 Dec;232(12):3775-3785. doi: 10.1002/jcp.25857. Epub 2017 May 3.

PMID:
28181692
5.

Inhibition of histone deacetylases triggers pharmacologic preconditioning effects against myocardial ischemic injury.

Zhao TC, Cheng G, Zhang LX, Tseng YT, Padbury JF.

Cardiovasc Res. 2007 Dec 1;76(3):473-81. Epub 2007 Aug 23.

PMID:
17884027
6.

Specific inhibition of HDAC4 in cardiac progenitor cells enhances myocardial repairs.

Zhang LX, DeNicola M, Qin X, Du J, Ma J, Tina Zhao Y, Zhuang S, Liu PY, Wei L, Qin G, Tang Y, Zhao TC.

Am J Physiol Cell Physiol. 2014 Aug 15;307(4):C358-72. doi: 10.1152/ajpcell.00187.2013. Epub 2014 Jun 18.

7.

Cardiomyocyte-restricted overexpression of extracellular superoxide dismutase increases nitric oxide bioavailability and reduces infarct size after ischemia/reperfusion.

Obal D, Dai S, Keith R, Dimova N, Kingery J, Zheng YT, Zweier J, Velayutham M, Prabhu SD, Li Q, Conklin D, Yang D, Bhatnagar A, Bolli R, Rokosh G.

Basic Res Cardiol. 2012 Nov;107(6):305. doi: 10.1007/s00395-012-0305-1. Epub 2012 Oct 26.

8.

Cyclic nucleotide phosphodiesterase 3A1 protects the heart against ischemia-reperfusion injury.

Oikawa M, Wu M, Lim S, Knight WE, Miller CL, Cai Y, Lu Y, Blaxall BC, Takeishi Y, Abe J, Yan C.

J Mol Cell Cardiol. 2013 Nov;64:11-9. doi: 10.1016/j.yjmcc.2013.08.003. Epub 2013 Aug 27.

9.

Bacopa monnieri extract increases rat coronary flow and protects against myocardial ischemia/reperfusion injury.

Srimachai S, Devaux S, Demougeot C, Kumphune S, Ullrich ND, Niggli E, Ingkaninan K, Kamkaew N, Scholfield CN, Tapechum S, Chootip K.

BMC Complement Altern Med. 2017 Feb 20;17(1):117. doi: 10.1186/s12906-017-1637-z.

10.

Selective inhibition of class I but not class IIb histone deacetylases exerts cardiac protection from ischemia reperfusion.

Aune SE, Herr DJ, Mani SK, Menick DR.

J Mol Cell Cardiol. 2014 Jul;72:138-45. doi: 10.1016/j.yjmcc.2014.03.005. Epub 2014 Mar 13.

11.

Cardiac Overexpression of S100A6 Attenuates Cardiomyocyte Apoptosis and Reduces Infarct Size After Myocardial Ischemia-Reperfusion.

Mofid A, Newman NS, Lee PJ, Abbasi C, Matkar PN, Rudenko D, Kuliszewski MA, Chen HH, Afrasiabi K, Tsoporis JN, Gramolini AO, Connelly KA, Parker TG, Leong-Poi H.

J Am Heart Assoc. 2017 Feb 7;6(2). pii: e004738. doi: 10.1161/JAHA.116.004738.

12.

HDAC inhibition elicits myocardial protective effect through modulation of MKK3/Akt-1.

Zhao TC, Du J, Zhuang S, Liu P, Zhang LX.

PLoS One. 2013 Jun 10;8(6):e65474. doi: 10.1371/journal.pone.0065474. Print 2013. Erratum in: PLoS One. 2013;8(7). doi:10.1371/annotation/29be7977-2e22-40cd-8a71-b3fbb99de9d5. Zhuang, Shugang [corrected to Zhuang, Shougang].

13.

Insulin protects isolated hearts from ischemia/reperfusion injury: cross-talk between PI3-K/Akt and JNKs.

Liu HT, Zhang HF, Si R, Zhang QJ, Zhang KR, Guo WY, Wang HC, Gao F.

Sheng Li Xue Bao. 2007 Oct 25;59(5):651-9.

14.

Overexpression of the cardiac Na+/Ca2+ exchanger increases susceptibility to ischemia/reperfusion injury in male, but not female, transgenic mice.

Cross HR, Lu L, Steenbergen C, Philipson KD, Murphy E.

Circ Res. 1998 Dec 14-28;83(12):1215-23.

PMID:
9851938
15.

Overexpression of TIMP3 Protects Against Cardiac Ischemia/Reperfusion Injury by Inhibiting Myocardial Apoptosis Through ROS/Mapks Pathway.

Liu H, Jing X, Dong A, Bai B, Wang H.

Cell Physiol Biochem. 2017;44(3):1011-1023. doi: 10.1159/000485401. Epub 2017 Nov 27.

16.

Targeted deletion of NF-kappaB p50 diminishes the cardioprotection of histone deacetylase inhibition.

Zhang LX, Zhao Y, Cheng G, Guo TL, Chin YE, Liu PY, Zhao TC.

Am J Physiol Heart Circ Physiol. 2010 Jun;298(6):H2154-63. doi: 10.1152/ajpheart.01015.2009. Epub 2010 Apr 9.

17.

Protease-activated receptor 2-mediated protection of myocardial ischemia-reperfusion injury: role of transient receptor potential vanilloid receptors.

Zhong B, Wang DH.

Am J Physiol Regul Integr Comp Physiol. 2009 Dec;297(6):R1681-90. doi: 10.1152/ajpregu.90746.2008. Epub 2009 Oct 7.

18.

High mobility group box 1 release from hepatocytes during ischemia and reperfusion injury is mediated by decreased histone deacetylase activity.

Evankovich J, Cho SW, Zhang R, Cardinal J, Dhupar R, Zhang L, Klune JR, Zlotnicki J, Billiar T, Tsung A.

J Biol Chem. 2010 Dec 17;285(51):39888-97. doi: 10.1074/jbc.M110.128348. Epub 2010 Oct 11.

19.

Oxygen surrounding the heart during ischemic conservation determines the myocardial injury during reperfusion.

Feng Y, Bopassa JC.

Am J Cardiovasc Dis. 2015 Aug 1;5(2):127-39. eCollection 2015.

20.

Suppression of Excessive Histone Deacetylases Activity in Diabetic Hearts Attenuates Myocardial Ischemia/Reperfusion Injury via Mitochondria Apoptosis Pathway.

Wu Y, Leng Y, Meng Q, Xue R, Zhao B, Zhan L, Xia Z.

J Diabetes Res. 2017;2017:8208065. doi: 10.1155/2017/8208065. Epub 2017 Jan 16.

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