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Items: 1 to 20 of 78

1.

A subset of five human mitochondrial formyl peptides mimics bacterial peptides and functionally deactivates human neutrophils.

Kaczmarek E, Hauser CJ, Kwon WY, Riça I, Chen L, Sandler N, Otterbein LE, Campbell Y, Cook CH, Yaffe MB, Marusich MF, Itagaki K.

J Trauma Acute Care Surg. 2018 Nov;85(5):936-943. doi: 10.1097/TA.0000000000001971.

PMID:
29787548
2.

Mitochondrial damage-associated molecular patterns from fractures suppress pulmonary immune responses via formyl peptide receptors 1 and 2.

Li H, Itagaki K, Sandler N, Gallo D, Galenkamp A, Kaczmarek E, Livingston DH, Zeng Y, Lee YT, Tang IT, Isal B, Otterbein L, Hauser CJ.

J Trauma Acute Care Surg. 2015 Feb;78(2):272-9; discussion 279-81. doi: 10.1097/TA.0000000000000509.

3.

Mitochondrial peptides are potent immune activators that activate human neutrophils via FPR-1.

Raoof M, Zhang Q, Itagaki K, Hauser CJ.

J Trauma. 2010 Jun;68(6):1328-32; discussion 1332-4. doi: 10.1097/TA.0b013e3181dcd28d.

PMID:
20539176
4.

Inhibition of human neutrophil chemotaxis by the protein kinase inhibitor, 1-(5-isoquinolinesulfonyl) piperazine.

Harvath L, McCall CE, Bass DA, McPhail LC.

J Immunol. 1987 Nov 1;139(9):3055-61.

PMID:
2822802
5.

Formyl Met-Leu-Phe-Stimulated FPR1 Phosphorylation in Plate-Adherent Human Neutrophils: Enhanced Proteolysis but Lack of Inhibition by Platelet-Activating Factor.

Jesaitis AJ, Gripentrog J, Voyich JM.

J Immunol Res. 2018 Jan 24;2018:3178970. doi: 10.1155/2018/3178970. eCollection 2018.

7.
8.

Human neutrophil formyl peptide receptor phosphorylation and the mucosal inflammatory response.

Leoni G, Gripentrog J, Lord C, Riesselman M, Sumagin R, Parkos CA, Nusrat A, Jesaitis AJ.

J Leukoc Biol. 2015 Jan;97(1):87-101. doi: 10.1189/jlb.4A0314-153R. Epub 2014 Nov 13.

10.

Identification of C-terminal phosphorylation sites of N-formyl peptide receptor-1 (FPR1) in human blood neutrophils.

Maaty WS, Lord CI, Gripentrog JM, Riesselman M, Keren-Aviram G, Liu T, Dratz EA, Bothner B, Jesaitis AJ.

J Biol Chem. 2013 Sep 20;288(38):27042-58. doi: 10.1074/jbc.M113.484113. Epub 2013 Jul 19.

11.

Mitochondrial DAMPs increase endothelial permeability through neutrophil dependent and independent pathways.

Sun S, Sursal T, Adibnia Y, Zhao C, Zheng Y, Li H, Otterbein LE, Hauser CJ, Itagaki K.

PLoS One. 2013;8(3):e59989. doi: 10.1371/journal.pone.0059989. Epub 2013 Mar 20.

12.

Mitochondrial damage-associated molecular patterns released by abdominal trauma suppress pulmonary immune responses.

Zhao C, Itagaki K, Gupta A, Odom S, Sandler N, Hauser CJ.

J Trauma Acute Care Surg. 2014 May;76(5):1222-7. doi: 10.1097/TA.0000000000000220.

13.

Mitochondrial damage associated molecular patterns from femoral reamings activate neutrophils through formyl peptide receptors and P44/42 MAP kinase.

Hauser CJ, Sursal T, Rodriguez EK, Appleton PT, Zhang Q, Itagaki K.

J Orthop Trauma. 2010 Sep;24(9):534-8. doi: 10.1097/BOT.0b013e3181ec4991.

14.

Effects of fibrinogen derivatives upon the inflammatory response. Studies with human fibrinopeptide B.

Senior RM, Skogen WF, Griffin GL, Wilner GD.

J Clin Invest. 1986 Mar;77(3):1014-9.

15.

Alteration of cellular cytosolic calcium and chemotactic peptide binding by an inhibitor of neutrophil function.

Cooper JA Jr, Bridges TA, Kennedy JI Jr, Culbreth R.

Am J Physiol. 1994 Jul;267(1 Pt 1):L71-8.

PMID:
8048544
16.
17.

Enhancement of chemotactic factor-stimulated neutrophil oxidative metabolism by leukotriene B4.

Gay JC, Beckman JK, Brash AR, Oates JA, Lukens JN.

Blood. 1984 Oct;64(4):780-5.

18.

Defective polymorphonuclear leukocyte formyl peptide receptor(s) in juvenile periodontitis.

Perez HD, Kelly E, Elfman F, Armitage G, Winkler J.

J Clin Invest. 1991 Mar;87(3):971-6.

20.

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