Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 85

1.

Quantitative proteomics and systems analysis of cultured H9C2 cardiomyoblasts during differentiation over time supports a 'function follows form' model of differentiation.

Kankeu C, Clarke K, Van Haver D, Gevaert K, Impens F, Dittrich A, Roderick HL, Passante E, Huber HJ.

Mol Omics. 2018 Jun 12;14(3):181-196. doi: 10.1039/c8mo00036k.

PMID:
29770421
2.

Proteomic analysis of F1F0-ATP synthase super-assembly in mitochondria of cardiomyoblasts undergoing differentiation to the cardiac lineage.

Bisetto E, Comelli M, Salzano AM, Picotti P, Scaloni A, Lippe G, Mavelli I.

Biochim Biophys Acta. 2013 Jul;1827(7):807-16. doi: 10.1016/j.bbabio.2013.04.002. Epub 2013 Apr 12.

3.

Gene Expression Profiling of H9c2 Myoblast Differentiation towards a Cardiac-Like Phenotype.

Branco AF, Pereira SP, Gonzalez S, Gusev O, Rizvanov AA, Oliveira PJ.

PLoS One. 2015 Jun 29;10(6):e0129303. doi: 10.1371/journal.pone.0129303. eCollection 2015.

4.

Proteomic investigation of embryonic rat heart-derived H9c2 cell line sheds new light on the molecular phenotype of the popular cell model.

Lenčo J, Lenčová-Popelová O, Link M, Jirkovská A, Tambor V, Potůčková E, Stulík J, Šimůnek T, Štěrba M.

Exp Cell Res. 2015 Dec 10;339(2):174-86. doi: 10.1016/j.yexcr.2015.10.020. Epub 2015 Nov 5. Erratum in: Exp Cell Res. 2016 May 1;343 (2):267.

PMID:
26515555
5.

Quantitative profiling of the rat heart myoblast secretome reveals differential responses to hypoxia and re-oxygenation stress.

Li X, Ren Y, Sorokin V, Poh KK, Ho HH, Lee CN, de Kleijn D, Lim SK, Tam JP, Sze SK.

J Proteomics. 2014 Feb 26;98:138-49. doi: 10.1016/j.jprot.2013.12.025. Epub 2014 Jan 9.

PMID:
24412200
6.

Cardiomyoblast (h9c2) differentiation on tunable extracellular matrix microenvironment.

Suhaeri M, Subbiah R, Van SY, Du P, Kim IG, Lee K, Park K.

Tissue Eng Part A. 2015 Jun;21(11-12):1940-51. doi: 10.1089/ten.TEA.2014.0591. Epub 2015 Apr 29.

7.

Regulation of PPAR-alpha pathway by Dicer revealed through proteomic analysis.

Sahasrabuddhe NA, Huang TC, Ahmad S, Kim MS, Yang Y, Ghosh B, Leach SD, Gowda H, Somani BL, Chaerkady R, Pandey A.

J Proteomics. 2014 Aug 28;108:306-15. doi: 10.1016/j.jprot.2014.04.027. Epub 2014 Apr 24.

PMID:
24769236
8.

Peroxisome proliferator-activated receptor delta agonists attenuated the C-reactive protein-induced pro-inflammation in cardiomyocytes and H9c2 cardiomyoblasts.

Liang YJ, Chen CY, Juang SJ, Lai LP, Shyu KG, Wang BW, Liu SY, Leu JG.

Eur J Pharmacol. 2010 Sep 15;643(1):84-92. doi: 10.1016/j.ejphar.2010.06.031. Epub 2010 Jun 28.

PMID:
20599914
9.

Activin receptor-like kinase 7 mediates high glucose-induced H9c2 cardiomyoblast apoptosis through activation of Smad2/3.

Liu L, Ding WY, Zhao J, Wang ZH, Zhong M, Zhang W, Chen YG, Zhang Y, Li L, Tang MX.

Int J Biochem Cell Biol. 2013 Sep;45(9):2027-35. doi: 10.1016/j.biocel.2013.06.018. Epub 2013 Jul 2.

PMID:
23830891
10.

Cyclin I and p53 are differentially expressed during the terminal differentiation of the postnatal mouse heart.

Liu Y, Tang MK, Cai DQ, Li M, Wong WM, Chow PH, Lee KK.

Proteomics. 2007 Jan;7(1):23-32.

PMID:
17154274
11.

An integrated approach of differential mass spectrometry and gene ontology analysis identified novel proteins regulating neuronal differentiation and survival.

Kobayashi D, Kumagai J, Morikawa T, Wilson-Morifuji M, Wilson A, Irie A, Araki N.

Mol Cell Proteomics. 2009 Oct;8(10):2350-67. doi: 10.1074/mcp.M900179-MCP200. Epub 2009 Jun 13.

12.

Methyl donor deficiency in H9c2 cardiomyoblasts induces ER stress as an important part of the proteome response.

Martinez E, Deval C, Jousse C, Mazur A, Brachet P, Comte B.

Int J Biochem Cell Biol. 2015 Feb;59:62-72. doi: 10.1016/j.biocel.2014.11.013. Epub 2014 Dec 6.

PMID:
25486180
13.

Cellular Proteome Dynamics during Differentiation of Human Primary Myoblasts.

Le Bihan MC, Barrio-Hernandez I, Mortensen TP, Henningsen J, Jensen SS, Bigot A, Blagoev B, Butler-Browne G, Kratchmarova I.

J Proteome Res. 2015 Aug 7;14(8):3348-61. doi: 10.1021/acs.jproteome.5b00397. Epub 2015 Jun 29.

PMID:
26074025
14.

Up-regulation of fatty acid oxidation in the ligament as a contributing factor of ankylosing spondylitis: A comparative proteomic study.

Xu WD, Yang XY, Li DH, Zheng KD, Qiu PC, Zhang W, Li CY, Lei KF, Yan GQ, Jin SW, Wang JG.

J Proteomics. 2015 Jan 15;113:57-72. doi: 10.1016/j.jprot.2014.09.014. Epub 2014 Oct 2.

PMID:
25281561
15.

Dilong prevents the high-KCl cardioplegic solution administration-induced apoptosis in H9c2 cardiomyoblast cells mediated by MEK.

Han CK, Kuo WW, Shen CY, Chen TS, Pai P, Tsai CH, Lo FY, Ju DT, Huang CY.

Am J Chin Med. 2014;42(6):1507-19. doi: 10.1142/S0192415X14500943.

PMID:
25482676
16.

Modulation of endothelin-A receptor, Galpha subunit, and RGS2 expression during H9c2 cardiomyoblast differentiation.

Giusti L, Gargini C, Ceccarelli F, Bacci M, Italiani P, Mazzoni MR.

J Recept Signal Transduct Res. 2004;24(4):297-317.

PMID:
15648448
17.

Isoproterenol cytotoxicity is dependent on the differentiation state of the cardiomyoblast H9c2 cell line.

Branco AF, Pereira SL, Moreira AC, Holy J, Sardão VA, Oliveira PJ.

Cardiovasc Toxicol. 2011 Sep;11(3):191-203. doi: 10.1007/s12012-011-9111-5. Erratum in: Cardiovasc Toxicol. 2011 Sep;11(3):284.

PMID:
21455642
18.

Quantitative proteomic study identified cathepsin B associated with doxorubicin-induced damage in H9c2 cardiomyocytes.

Bao GY, Wang HZ, Shang YJ, Fan HJ, Gu ML, Xia R, Qin Q, Deng AM.

Biosci Trends. 2012 Dec;6(6):283-7.

19.

Differentiation-dependent doxorubicin toxicity on H9c2 cardiomyoblasts.

Branco AF, Sampaio SF, Moreira AC, Holy J, Wallace KB, Baldeiras I, Oliveira PJ, Sardão VA.

Cardiovasc Toxicol. 2012 Dec;12(4):326-40. doi: 10.1007/s12012-012-9177-8.

PMID:
22744233
20.

Proteomics analysis of the DF-1 chicken fibroblasts infected with avian reovirus strain S1133.

Chen WT, Wu YL, Chen T, Cheng CS, Chan HL, Chou HC, Chen YW, Yin HS.

PLoS One. 2014 Mar 25;9(3):e92154. doi: 10.1371/journal.pone.0092154. eCollection 2014.

Supplemental Content

Support Center