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Items: 1 to 20 of 133

1.

Systemic redox biomarkers and their relationship to prognostic risk markers in autosomal dominant polycystic kidney disease and IgA nephropathy.

Tariq A, Mansoor MA, Marti HP, Jonsson G, Slettan A, Weeraman P, Apeland T.

Clin Biochem. 2018 Jun;56:33-40. doi: 10.1016/j.clinbiochem.2018.04.010. Epub 2018 Apr 12.

PMID:
29655960
2.

Kidney donors and kidney transplants have abnormal aminothiol redox status, and are at increased risk of oxidative stress and reduced redox buffer capacity.

Apeland T, Holdaas H, Mansoor MA.

Clin Biochem. 2014 Apr;47(6):378-82. doi: 10.1016/j.clinbiochem.2014.02.003. Epub 2014 Feb 11.

PMID:
24530370
3.
4.
5.

Early prediction of IgA nephropathy progression: proteinuria and AOPP are strong prognostic markers.

Descamps-Latscha B, Witko-Sarsat V, Nguyen-Khoa T, Nguyen AT, Gausson V, Mothu N, Cardoso C, Noël LH, Guérin AP, London GM, Jungers P.

Kidney Int. 2004 Oct;66(4):1606-12.

6.

Elevated factor H-related protein 1 and factor H pathogenic variants decrease complement regulation in IgA nephropathy.

Tortajada A, Gutiérrez E, Goicoechea de Jorge E, Anter J, Segarra A, Espinosa M, Blasco M, Roman E, Marco H, Quintana LF, Gutiérrez J, Pinto S, Lopez-Trascasa M, Praga M, Rodriguez de Córdoba S.

Kidney Int. 2017 Oct;92(4):953-963. doi: 10.1016/j.kint.2017.03.041. Epub 2017 Jun 19.

PMID:
28637589
7.

Manganese superoxide dismutase (SOD2) polymorphisms, plasma advanced oxidation protein products (AOPP) concentration and risk of kidney complications in subjects with type 1 diabetes.

Mohammedi K, Bellili-Muñoz N, Driss F, Roussel R, Seta N, Fumeron F, Hadjadj S, Marre M, Velho G.

PLoS One. 2014 May 12;9(5):e96916. doi: 10.1371/journal.pone.0096916. eCollection 2014.

8.

Plasma gelsolin levels are decreased and correlate with fibrosis in IgA nephropathy.

Han C, Zhang L, Zhu X, Tang J, Jin X.

Exp Biol Med (Maywood). 2013 Nov 1;238(11):1318-27. doi: 10.1177/1535370213503256. Epub 2013 Sep 18.

PMID:
24047794
9.

Association between CCDC132, FDX1 and TNFSF13 gene polymorphisms and the risk of IgA nephropathy.

Niu D, Ren Y, Xie L, Sun J, Lu W, Hao Y, Zhang Y, Yin A, Li H, Lv J, Li S.

Nephrology (Carlton). 2015 Dec;20(12):908-15. doi: 10.1111/nep.12611.

PMID:
26370181
10.

Plasma oxidative stress level of IgA nephropathy in children and the effect of early intervention with angiotensin-converting enzyme inhibitors.

Pei Y, Xu Y, Ruan J, Rong L, Jiang M, Mo Y, Jiang X.

J Renin Angiotensin Aldosterone Syst. 2016 May 18;17(2):1470320316647240. doi: 10.1177/1470320316647240. Print 2016 Apr-Jun.

11.

Replication of genome-wide association study identified seven susceptibility genes, affirming the effect of rs2856717 on renal function and poor outcome of IgA nephropathy.

Wang W, Li G, Hong D, Zou Y, Fei D, Wang L.

Nephrology (Carlton). 2017 Oct;22(10):811-817. doi: 10.1111/nep.12860.

PMID:
27450519
12.

Association of soluble CD89 levels with disease progression but not susceptibility in IgA nephropathy.

Vuong MT, Hahn-Zoric M, Lundberg S, Gunnarsson I, van Kooten C, Wramner L, Seddighzadeh M, Fernström A, Hanson LÅ, Do LT, Jacobson SH, Padyukov L.

Kidney Int. 2010 Dec;78(12):1281-7. doi: 10.1038/ki.2010.314. Epub 2010 Sep 1.

13.

Oxidative stress and galactose-deficient IgA1 as markers of progression in IgA nephropathy.

Camilla R, Suzuki H, Daprà V, Loiacono E, Peruzzi L, Amore A, Ghiggeri GM, Mazzucco G, Scolari F, Gharavi AG, Appel GB, Troyanov S, Novak J, Julian BA, Coppo R.

Clin J Am Soc Nephrol. 2011 Aug;6(8):1903-11. doi: 10.2215/CJN.11571210. Epub 2011 Jul 22.

14.

Plasma reduced homocysteine and other aminothiol concentrations in patients with CKD.

Sjöberg B, Anderstam B, Suliman M, Alvestrand A.

Am J Kidney Dis. 2006 Jan;47(1):60-71.

PMID:
16377386
15.

A panel of serum biomarkers differentiates IgA nephropathy from other renal diseases.

Yanagawa H, Suzuki H, Suzuki Y, Kiryluk K, Gharavi AG, Matsuoka K, Makita Y, Julian BA, Novak J, Tomino Y.

PLoS One. 2014 May 23;9(5):e98081. doi: 10.1371/journal.pone.0098081. eCollection 2014.

16.

Malondialdehyde and advanced oxidation protein products are not increased in psoriasis: a controlled study.

Skoie IM, Dalen I, Omdal R, Jonsson G.

Arch Dermatol Res. 2019 May;311(4):299-308. doi: 10.1007/s00403-019-01903-2. Epub 2019 Mar 4.

PMID:
30830309
17.

TLR1 polymorphism rs4833095 as a risk factor for IgA nephropathy in a Chinese Han population: A case-control study.

Gao J, Wei L, Wei J, Yao G, Wang L, Wang M, Liu X, Dai C, Jin T, Dai Z, Fu R.

Oncotarget. 2016 Dec 13;7(50):83031-83039. doi: 10.18632/oncotarget.12965.

18.

Lipid peroxidation in IgA nephropathy and the effect of lipo-prostaglandin E1.

Tian J, Chen JH, Li Q, He Q, Lin WQ.

J Nephrol. 2005 May-Jun;18(3):243-8.

PMID:
16013010
19.

Early post-transplant serum IgA level is associated with IgA nephropathy recurrence after kidney transplantation.

Garnier AS, Duveau A, Demiselle J, Croué A, Subra JF, Sayegh J, Augusto JF.

PLoS One. 2018 Apr 25;13(4):e0196101. doi: 10.1371/journal.pone.0196101. eCollection 2018.

20.

Elevated Plasma α-Defensins (HNP1-3) Levels Correlated with IgA1 Glycosylation and Susceptibility to IgA Nephropathy.

Qi YY, Zhou XJ, Cheng FJ, Zhang H.

Dis Markers. 2016;2016:8123138. doi: 10.1155/2016/8123138. Epub 2016 Aug 2.

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