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Items: 1 to 20 of 90

1.

Ephrin receptor A2 is a functional entry receptor for Epstein-Barr virus.

Chen J, Sathiyamoorthy K, Zhang X, Schaller S, Perez White BE, Jardetzky TS, Longnecker R.

Nat Microbiol. 2018 Feb;3(2):172-180. doi: 10.1038/s41564-017-0081-7. Epub 2018 Jan 1.

2.

Ephrin Receptor A4 is a New Kaposi's Sarcoma-Associated Herpesvirus Virus Entry Receptor.

Chen J, Zhang X, Schaller S, Jardetzky TS, Longnecker R.

MBio. 2019 Feb 19;10(1). pii: e02892-18. doi: 10.1128/mBio.02892-18.

3.

Ephrin receptor A2 is an epithelial cell receptor for Epstein-Barr virus entry.

Zhang H, Li Y, Wang HB, Zhang A, Chen ML, Fang ZX, Dong XD, Li SB, Du Y, Xiong D, He JY, Li MZ, Liu YM, Zhou AJ, Zhong Q, Zeng YX, Kieff E, Zhang Z, Gewurz BE, Zhao B, Zeng MS.

Nat Microbiol. 2018 Feb;3(2):1-8. doi: 10.1038/s41564-017-0080-8. Epub 2018 Jan 1. Erratum in: Nat Microbiol. 2018 Apr 20;:.

PMID:
29292383
4.

Fusion of epithelial cells by Epstein-Barr virus proteins is triggered by binding of viral glycoproteins gHgL to integrins alphavbeta6 or alphavbeta8.

Chesnokova LS, Nishimura SL, Hutt-Fletcher LM.

Proc Natl Acad Sci U S A. 2009 Dec 1;106(48):20464-9. doi: 10.1073/pnas.0907508106. Epub 2009 Nov 17. Erratum in: Proc Natl Acad Sci U S A. 2010 Feb 16;107(7):3275.

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Epstein-Barr virus glycoprotein gB and gHgL can mediate fusion and entry in trans, and heat can act as a partial surrogate for gHgL and trigger a conformational change in gB.

Chesnokova LS, Ahuja MK, Hutt-Fletcher LM.

J Virol. 2014 Nov;88(21):12193-201. doi: 10.1128/JVI.01597-14. Epub 2014 Aug 20.

8.

Epstein-Barr Virus Fusion with Epithelial Cells Triggered by gB Is Restricted by a gL Glycosylation Site.

Möhl BS, Chen J, Park SJ, Jardetzky TS, Longnecker R.

J Virol. 2017 Nov 14;91(23). pii: e01255-17. doi: 10.1128/JVI.01255-17. Print 2017 Dec 1.

9.

The conserved disulfide bond within domain II of Epstein-Barr virus gH has divergent roles in membrane fusion with epithelial cells and B cells.

Möhl BS, Sathiyamoorthy K, Jardetzky TS, Longnecker R.

J Virol. 2014 Dec;88(23):13570-9. doi: 10.1128/JVI.02272-14. Epub 2014 Sep 17.

10.

αvβ6- and αvβ8-integrins serve as interchangeable receptors for HSV gH/gL to promote endocytosis and activation of membrane fusion.

Gianni T, Salvioli S, Chesnokova LS, Hutt-Fletcher LM, Campadelli-Fiume G.

PLoS Pathog. 2013;9(12):e1003806. doi: 10.1371/journal.ppat.1003806. Epub 2013 Dec 19.

11.

The ephrin receptor tyrosine kinase A2 is a cellular receptor for Kaposi's sarcoma–associated herpesvirus.

Hahn AS, Kaufmann JK, Wies E, Naschberger E, Panteleev-Ivlev J, Schmidt K, Holzer A, Schmidt M, Chen J, König S, Ensser A, Myoung J, Brockmeyer NH, Stürzl M, Fleckenstein B, Neipel F.

Nat Med. 2012 Jun;18(6):961-6. doi: 10.1038/nm.2805.

12.

Structural and Mechanistic Insights into the Tropism of Epstein-Barr Virus.

Möhl BS, Chen J, Sathiyamoorthy K, Jardetzky TS, Longnecker R.

Mol Cells. 2016 Apr 30;39(4):286-91. doi: 10.14348/molcells.2016.0066. Epub 2016 Apr 6. Review.

13.

Structural basis for Epstein-Barr virus host cell tropism mediated by gp42 and gHgL entry glycoproteins.

Sathiyamoorthy K, Hu YX, Möhl BS, Chen J, Longnecker R, Jardetzky TS.

Nat Commun. 2016 Dec 8;7:13557. doi: 10.1038/ncomms13557.

14.

Assembly and architecture of the EBV B cell entry triggering complex.

Sathiyamoorthy K, Jiang J, Hu YX, Rowe CL, Möhl BS, Chen J, Jiang W, Mellins ED, Longnecker R, Zhou ZH, Jardetzky TS.

PLoS Pathog. 2014 Aug 21;10(8):e1004309. doi: 10.1371/journal.ppat.1004309. eCollection 2014 Aug.

15.

Integrins as triggers of Epstein-Barr virus fusion and epithelial cell infection.

Hutt-Fletcher LM, Chesnokova LS.

Virulence. 2010 Sep-Oct;1(5):395-8. doi: 10.4161/viru.1.5.12546. Review.

16.

Inhibition of EBV-mediated membrane fusion by anti-gHgL antibodies.

Sathiyamoorthy K, Jiang J, Möhl BS, Chen J, Zhou ZH, Longnecker R, Jardetzky TS.

Proc Natl Acad Sci U S A. 2017 Oct 10;114(41):E8703-E8710. doi: 10.1073/pnas.1704661114. Epub 2017 Sep 22.

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Modulation of Epstein-Barr virus glycoprotein B (gB) fusion activity by the gB cytoplasmic tail domain.

Garcia NJ, Chen J, Longnecker R.

MBio. 2013 Jan 22;4(1):e00571-12. doi: 10.1128/mBio.00571-12.

19.

Membrane anchoring of Epstein-Barr virus gp42 inhibits fusion with B cells even with increased flexibility allowed by engineered spacers.

Rowe CL, Chen J, Jardetzky TS, Longnecker R.

MBio. 2015 Jan 6;6(1). pii: e02285-14. doi: 10.1128/mBio.02285-14.

20.

Small molecules can selectively inhibit ephrin binding to the EphA4 and EphA2 receptors.

Noberini R, Koolpe M, Peddibhotla S, Dahl R, Su Y, Cosford ND, Roth GP, Pasquale EB.

J Biol Chem. 2008 Oct 24;283(43):29461-72. doi: 10.1074/jbc.M804103200. Epub 2008 Aug 26.

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