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Items: 1 to 20 of 98

1.

Nucleoside-Diphosphate-Kinase of P. gingivalis is Secreted from Epithelial Cells In the Absence of a Leader Sequence Through a Pannexin-1 Interactome.

Atanasova K, Lee J, Roberts J, Lee K, Ojcius DM, Yilmaz Ö.

Sci Rep. 2016 Nov 24;6:37643. doi: 10.1038/srep37643.

2.

Porphyromonas gingivalis-nucleoside-diphosphate-kinase inhibits ATP-induced reactive-oxygen-species via P2X7 receptor/NADPH-oxidase signalling and contributes to persistence.

Choi CH, Spooner R, DeGuzman J, Koutouzis T, Ojcius DM, Yilmaz Ö.

Cell Microbiol. 2013 Jun;15(6):961-76. doi: 10.1111/cmi.12089. Epub 2013 Jan 9.

3.

A novel kinase function of a nucleoside-diphosphate-kinase homologue in Porphyromonas gingivalis is critical in subversion of host cell apoptosis by targeting heat-shock protein 27.

Lee J, Roberts JS, Atanasova KR, Chowdhury N, Yilmaz Ö.

Cell Microbiol. 2018 May;20(5):e12825. doi: 10.1111/cmi.12825. Epub 2018 Feb 7.

PMID:
29359393
4.

Opportunistic Pathogen Porphyromonas gingivalis Modulates Danger Signal ATP-Mediated Antibacterial NOX2 Pathways in Primary Epithelial Cells.

Roberts JS, Atanasova KR, Lee J, Diamond G, Deguzman J, Hee Choi C, Yilmaz Ö.

Front Cell Infect Microbiol. 2017 Jul 4;7:291. doi: 10.3389/fcimb.2017.00291. eCollection 2017.

5.

Porphyromonas gingivalis attenuates ATP-mediated inflammasome activation and HMGB1 release through expression of a nucleoside-diphosphate kinase.

Johnson L, Atanasova KR, Bui PQ, Lee J, Hung SC, Yilmaz Ö, Ojcius DM.

Microbes Infect. 2015 May;17(5):369-77. doi: 10.1016/j.micinf.2015.03.010. Epub 2015 Mar 27.

6.

ATP scavenging by the intracellular pathogen Porphyromonas gingivalis inhibits P2X7-mediated host-cell apoptosis.

Yilmaz O, Yao L, Maeda K, Rose TM, Lewis EL, Duman M, Lamont RJ, Ojcius DM.

Cell Microbiol. 2008 Apr;10(4):863-75. Epub 2007 Nov 13.

7.

Porphyromonas gingivalis traffics into endoplasmic reticulum-rich-autophagosomes for successful survival in human gingival epithelial cells.

Lee K, Roberts JS, Choi CH, Atanasova KR, Yilmaz Ö.

Virulence. 2018 Dec 31;9(1):845-859. doi: 10.1080/21505594.2018.1454171.

8.

Intercellular spreading of Porphyromonas gingivalis infection in primary gingival epithelial cells.

Yilmaz O, Verbeke P, Lamont RJ, Ojcius DM.

Infect Immun. 2006 Jan;74(1):703-10.

9.

Porphyromonas gingivalis fimbriae dampen P2X7-dependent interleukin-1β secretion.

Morandini AC, Ramos-Junior ES, Potempa J, Nguyen KA, Oliveira AC, Bellio M, Ojcius DM, Scharfstein J, Coutinho-Silva R.

J Innate Immun. 2014;6(6):831-45. doi: 10.1159/000363338. Epub 2014 Jun 6.

10.

ATP-dependent activation of an inflammasome in primary gingival epithelial cells infected by Porphyromonas gingivalis.

Yilmaz O, Sater AA, Yao L, Koutouzis T, Pettengill M, Ojcius DM.

Cell Microbiol. 2010 Feb;12(2):188-98. doi: 10.1111/j.1462-5822.2009.01390.x. Epub 2009 Oct 6.

11.

P2X4 assembles with P2X7 and pannexin-1 in gingival epithelial cells and modulates ATP-induced reactive oxygen species production and inflammasome activation.

Hung SC, Choi CH, Said-Sadier N, Johnson L, Atanasova KR, Sellami H, Yilmaz Ö, Ojcius DM.

PLoS One. 2013 Jul 25;8(7):e70210. doi: 10.1371/journal.pone.0070210. Print 2013.

12.

Porphyromonas gingivalis infection sequesters pro-apoptotic Bad through Akt in primary gingival epithelial cells.

Yao L, Jermanus C, Barbetta B, Choi C, Verbeke P, Ojcius DM, Yilmaz O.

Mol Oral Microbiol. 2010 Apr;25(2):89-101. doi: 10.1111/j.2041-1014.2010.00569.x.

13.

Exit of intracellular Porphyromonas gingivalis from gingival epithelial cells is mediated by endocytic recycling pathway.

Takeuchi H, Furuta N, Morisaki I, Amano A.

Cell Microbiol. 2011 May;13(5):677-91. doi: 10.1111/j.1462-5822.2010.01564.x. Epub 2011 Jan 10.

PMID:
21155963
14.

Porphyromonas gingivalis SerB-mediated dephosphorylation of host cell cofilin modulates invasion efficiency.

Moffatt CE, Inaba H, Hirano T, Lamont RJ.

Cell Microbiol. 2012 Apr;14(4):577-88. doi: 10.1111/j.1462-5822.2011.01743.x. Epub 2012 Feb 2.

15.

Intrinsic apoptotic pathways of gingival epithelial cells modulated by Porphyromonas gingivalis.

Mao S, Park Y, Hasegawa Y, Tribble GD, James CE, Handfield M, Stavropoulos MF, Yilmaz O, Lamont RJ.

Cell Microbiol. 2007 Aug;9(8):1997-2007. Epub 2007 Apr 5.

16.

Genetic transformation of an obligate anaerobe, P. gingivalis for FMN-green fluorescent protein expression in studying host-microbe interaction.

Choi CH, DeGuzman JV, Lamont RJ, Yilmaz Ö.

PLoS One. 2011 Apr 15;6(4):e18499. doi: 10.1371/journal.pone.0018499.

17.

A Dual Role for P2X7 Receptor during Porphyromonas gingivalis Infection.

Ramos-Junior ES, Morandini AC, Almeida-da-Silva CL, Franco EJ, Potempa J, Nguyen KA, Oliveira AC, Zamboni DS, Ojcius DM, Scharfstein J, Coutinho-Silva R.

J Dent Res. 2015 Sep;94(9):1233-42. doi: 10.1177/0022034515593465. Epub 2015 Jul 7.

18.

Cytotoxic activity of nucleoside diphosphate kinase secreted from Mycobacterium tuberculosis.

Chopra P, Singh A, Koul A, Ramachandran S, Drlica K, Tyagi AK, Singh Y.

Eur J Biochem. 2003 Feb;270(4):625-34.

19.

Secretory nucleoside diphosphate kinases from both intra- and extracellular pathogenic bacteria are functionally indistinguishable.

Dar HH, Prasad D, Varshney GC, Chakraborti PK.

Microbiology. 2011 Nov;157(Pt 11):3024-35. doi: 10.1099/mic.0.049221-0. Epub 2011 Aug 4.

PMID:
21816881
20.

Gingival epithelial cell signalling and cytoskeletal responses to Porphyromonas gingivalis invasion.

Yilmaz O, Young PA, Lamont RJ, Kenny GE.

Microbiology. 2003 Sep;149(Pt 9):2417-26.

PMID:
12949167

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