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Controlled Mycobacterium tuberculosis infection in mice under treatment with anti-IL-17A or IL-17F antibodies, in contrast to TNFα neutralization.

Segueni N, Tritto E, Bourigault ML, Rose S, Erard F, Le Bert M, Jacobs M, Di Padova F, Stiehl DP, Moulin P, Brees D, Chibout SD, Ryffel B, Kammüller M, Quesniaux VF.

Sci Rep. 2016 Nov 17;6:36923. doi: 10.1038/srep36923.


Mice genetically inactivated in interleukin-17A receptor are defective in long-term control of Mycobacterium tuberculosis infection.

Freches D, Korf H, Denis O, Havaux X, Huygen K, Romano M.

Immunology. 2013 Oct;140(2):220-31. doi: 10.1111/imm.12130.


Antibody blockade of IL-17 family cytokines in immunity to acute murine oral mucosal candidiasis.

Whibley N, Tritto E, Traggiai E, Kolbinger F, Moulin P, Brees D, Coleman BM, Mamo AJ, Garg AV, Jaycox JR, Siebenlist U, Kammüller M, Gaffen SL.

J Leukoc Biol. 2016 Jun;99(6):1153-64. doi: 10.1189/jlb.4A0915-428R. Epub 2016 Jan 4. Erratum in: J Leukoc Biol. 2017 Apr;101(4):1065.


IL-22 is mainly produced by IFNγ-secreting cells but is dispensable for host protection against Mycobacterium tuberculosis infection.

Behrends J, Renauld JC, Ehlers S, Hölscher C.

PLoS One. 2013;8(2):e57379. doi: 10.1371/journal.pone.0057379. Epub 2013 Feb 27.


Intrahepatic innate lymphoid cells secrete IL-17A and IL-17F that are crucial for T cell priming in viral infection.

Jie Z, Liang Y, Hou L, Dong C, Iwakura Y, Soong L, Cong Y, Sun J.

J Immunol. 2014 Apr 1;192(7):3289-300. doi: 10.4049/jimmunol.1303281. Epub 2014 Mar 5.


Involvement of IL-17A-producing TCR γδ T cells in late protective immunity against pulmonary Mycobacterium tuberculosis infection.

Umemura M, Okamoto-Yoshida Y, Yahagi A, Touyama S, Nakae S, Iwakura Y, Matsuzaki G.

Immun Inflamm Dis. 2016 Aug 24;4(4):401-412. eCollection 2016 Dec.


Increased pulmonary tumor necrosis factor alpha, interleukin-6 (IL-6), and IL-17A responses compensate for decreased gamma interferon production in anti-IL-12 autovaccine-treated, Mycobacterium bovis BCG-vaccinated mice.

Freches D, Romano M, Korf H, Renauld JC, Van Snick J, Uyttenhove C, Huygen K.

Clin Vaccine Immunol. 2011 Jan;18(1):95-104. doi: 10.1128/CVI.00352-10. Epub 2010 Nov 17.


[Frontier of mycobacterium research--host vs. mycobacterium].

Okada M, Shirakawa T.

Kekkaku. 2005 Sep;80(9):613-29. Japanese.


The increased protection and pathology in Mycobacterium tuberculosis-infected IL-27R-alpha-deficient mice is supported by IL-17A and is associated with the IL-17A-induced expansion of multifunctional T cells.

Erdmann H, Behrends J, Ritter K, Hölscher A, Volz J, Rosenkrands I, Hölscher C.

Mucosal Immunol. 2018 Jul;11(4):1168-1180. doi: 10.1038/s41385-018-0026-3. Epub 2018 May 4.


A role for tumour necrosis factor-alpha, complement C5 and interleukin-6 in the initiation and development of the mycobacterial cord factor trehalose 6,6'-dimycolate induced granulomatous response.

Welsh KJ, Abbott AN, Hwang SA, Indrigo J, Armitige LY, Blackburn MR, Hunter RL Jr, Actor JK.

Microbiology. 2008 Jun;154(Pt 6):1813-24. doi: 10.1099/mic.0.2008/016923-0.


Dominant-negative tumor necrosis factor protects from Mycobacterium bovis Bacillus Calmette Guérin (BCG) and endotoxin-induced liver injury without compromising host immunity to BCG and Mycobacterium tuberculosis.

Olleros ML, Vesin D, Lambou AF, Janssens JP, Ryffel B, Rose S, Frémond C, Quesniaux VF, Szymkowski DE, Garcia I.

J Infect Dis. 2009 Apr 1;199(7):1053-63. doi: 10.1086/597204.


Role of the CD137 ligand (CD137L) signaling pathway during Mycobacterium tuberculosis infection.

Martínez Gómez JM, Koh VH, Yan B, Lin W, Ang ML, Rahim SZ, Pethe K, Schwarz H, Alonso S.

Immunobiology. 2014 Jan;219(1):78-86. doi: 10.1016/j.imbio.2013.08.009. Epub 2013 Aug 23.


Programmed death-1 (PD-1)-deficient mice are extraordinarily sensitive to tuberculosis.

Lázár-Molnár E, Chen B, Sweeney KA, Wang EJ, Liu W, Lin J, Porcelli SA, Almo SC, Nathenson SG, Jacobs WR Jr.

Proc Natl Acad Sci U S A. 2010 Jul 27;107(30):13402-7. doi: 10.1073/pnas.1007394107. Epub 2010 Jul 12.


The IL-17F/IL-17RC Axis Promotes Respiratory Allergy in the Proximal Airways.

De Luca A, Pariano M, Cellini B, Costantini C, Villella VR, Jose SS, Palmieri M, Borghi M, Galosi C, Paolicelli G, Maiuri L, Fric J, Zelante T.

Cell Rep. 2017 Aug 15;20(7):1667-1680. doi: 10.1016/j.celrep.2017.07.063.


Interleukin-12 and interleukin-27 regulate macrophage control of Mycobacterium tuberculosis.

Robinson CM, Nau GJ.

J Infect Dis. 2008 Aug 1;198(3):359-66. doi: 10.1086/589774.


TNF in host resistance to tuberculosis infection.

Quesniaux VF, Jacobs M, Allie N, Grivennikov S, Nedospasov SA, Garcia I, Olleros ML, Shebzukhov Y, Kuprash D, Vasseur V, Rose S, Court N, Vacher R, Ryffel B.

Curr Dir Autoimmun. 2010;11:157-79. doi: 10.1159/000289204. Epub 2010 Feb 18. Review.


Protective CD4 T cells targeting cryptic epitopes of Mycobacterium tuberculosis resist infection-driven terminal differentiation.

Woodworth JS, Aagaard CS, Hansen PR, Cassidy JP, Agger EM, Andersen P.

J Immunol. 2014 Apr 1;192(7):3247-58. doi: 10.4049/jimmunol.1300283. Epub 2014 Feb 26.


Engagement of the Aryl Hydrocarbon Receptor in Mycobacterium tuberculosis-Infected Macrophages Has Pleiotropic Effects on Innate Immune Signaling.

Memari B, Bouttier M, Dimitrov V, Ouellette M, Behr MA, Fritz JH, White JH.

J Immunol. 2015 Nov 1;195(9):4479-91. doi: 10.4049/jimmunol.1501141. Epub 2015 Sep 28.


Innate and acquired immune responses to mycobacterial infections: involvement of IL-17A/IL-23 axis in protective immunity.

Umemura M, Matsuzaki G.

Nihon Hansenbyo Gakkai Zasshi. 2013 Dec;82(3):123-32. Review.


Containment of aerogenic Mycobacterium tuberculosis infection in mice does not require MyD88 adaptor function for TLR2, -4 and -9.

Hölscher C, Reiling N, Schaible UE, Hölscher A, Bathmann C, Korbel D, Lenz I, Sonntag T, Kröger S, Akira S, Mossmann H, Kirschning CJ, Wagner H, Freudenberg M, Ehlers S.

Eur J Immunol. 2008 Mar;38(3):680-94. doi: 10.1002/eji.200736458.

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