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Items: 1 to 20 of 97

1.

Lead-Related Genetic Loci, Cumulative Lead Exposure and Incident Coronary Heart Disease: The Normative Aging Study.

Ding N, Wang X, Weisskopf MG, Sparrow D, Schwartz J, Hu H, Park SK.

PLoS One. 2016 Sep 1;11(9):e0161472. doi: 10.1371/journal.pone.0161472. eCollection 2016.

2.

Cumulative lead exposure is associated with reduced olfactory recognition performance in elderly men: The Normative Aging Study.

Grashow R, Sparrow D, Hu H, Weisskopf MG.

Neurotoxicology. 2015 Jul;49:158-64. doi: 10.1016/j.neuro.2015.06.006. Epub 2015 Jun 26.

3.
4.

Dietary patterns, bone lead and incident coronary heart disease among middle-aged to elderly men.

Ding N, Wang X, Tucker KL, Weisskopf MG, Sparrow D, Hu H, Park SK.

Environ Res. 2019 Jan;168:222-229. doi: 10.1016/j.envres.2018.09.035. Epub 2018 Sep 27.

PMID:
30317107
5.
6.

Modifying roles of glutathione S-transferase polymorphisms on the association between cumulative lead exposure and cognitive function.

Eum KD, Wang FT, Schwartz J, Hersh CP, Kelsey K, Wright RO, Spiro A, Sparrow D, Hu H, Weisskopf MG.

Neurotoxicology. 2013 Dec;39:65-71. doi: 10.1016/j.neuro.2013.08.002. Epub 2013 Aug 16.

7.

Effect modification by delta-aminolevulinic acid dehydratase, vitamin D receptor, and nitric oxide synthase gene polymorphisms on associations between patella lead and renal function in lead workers.

Weaver VM, Lee BK, Todd AC, Ahn KD, Shi W, Jaar BG, Kelsey KT, Lustberg ME, Silbergeld EK, Parsons PJ, Wen J, Schwartz BS.

Environ Res. 2006 Sep;102(1):61-9. Epub 2006 Feb 17.

PMID:
16487505
8.

Delta-aminolevulinic acid dehydratase polymorphism and the relation between low level lead exposure and the Mini-Mental Status Examination in older men: the Normative Aging Study.

Weuve J, Kelsey KT, Schwartz J, Bellinger D, Wright RO, Rajan P, Spiro A 3rd, Sparrow D, Aro A, Hu H.

Occup Environ Med. 2006 Nov;63(11):746-53. Epub 2006 Jun 6. Erratum in: Occup Environ Med. 2007 Apr;64(4):288.

9.

Biased Exposure-Health Effect Estimates from Selection in Cohort Studies: Are Environmental Studies at Particular Risk?

Weisskopf MG, Sparrow D, Hu H, Power MC.

Environ Health Perspect. 2015 Nov;123(11):1113-22. doi: 10.1289/ehp.1408888. Epub 2015 May 8.

10.

Effect modification by vitamin D receptor genetic polymorphisms in the association between cumulative lead exposure and pulse pressure: a longitudinal study.

Jhun MA, Hu H, Schwartz J, Weisskopf MG, Nie LH, Sparrow D, Vokonas PS, Park SK.

Environ Health. 2015 Jan 13;14:5. doi: 10.1186/1476-069X-14-5.

11.
12.

A prospective study of bone lead concentration and death from all causes, cardiovascular diseases, and cancer in the Department of Veterans Affairs Normative Aging Study.

Weisskopf MG, Jain N, Nie H, Sparrow D, Vokonas P, Schwartz J, Hu H.

Circulation. 2009 Sep 22;120(12):1056-64. doi: 10.1161/CIRCULATIONAHA.108.827121. Epub 2009 Sep 8. Erratum in: Circulation. 2014 Jul 29;130(5):e43.

13.

HFE H63D polymorphism as a modifier of the effect of cumulative lead exposure on pulse pressure: the Normative Aging Study.

Zhang A, Park SK, Wright RO, Weisskopf MG, Mukherjee B, Nie H, Sparrow D, Hu H.

Environ Health Perspect. 2010 Sep;118(9):1261-6. doi: 10.1289/ehp.1002251. Epub 2010 May 14.

14.

Amyotrophic lateral sclerosis, lead, and genetic susceptibility: polymorphisms in the delta-aminolevulinic acid dehydratase and vitamin D receptor genes.

Kamel F, Umbach DM, Lehman TA, Park LP, Munsat TL, Shefner JM, Sandler DP, Hu H, Taylor JA.

Environ Health Perspect. 2003 Aug;111(10):1335-9.

15.

Genetic background of lead and mercury metabolism in a group of medical students in Austria.

Gundacker C, Wittmann KJ, Kukuckova M, Komarnicki G, Hikkel I, Gencik M.

Environ Res. 2009 Aug;109(6):786-96. doi: 10.1016/j.envres.2009.05.003. Epub 2009 Jun 9.

PMID:
19515364
16.

GSTA1, GSTM1, GSTP1, and GSTT1 polymorphisms and susceptibility to smoking-related bladder cancer: a case-control study.

Matic M, Pekmezovic T, Djukic T, Mimic-Oka J, Dragicevic D, Krivic B, Suvakov S, Savic-Radojevic A, Pljesa-Ercegovac M, Tulic C, Coric V, Simic T.

Urol Oncol. 2013 Oct;31(7):1184-92. doi: 10.1016/j.urolonc.2011.08.005.

PMID:
24075358
17.

A genetic risk score based on direct associations with coronary heart disease improves coronary heart disease risk prediction in the Atherosclerosis Risk in Communities (ARIC), but not in the Rotterdam and Framingham Offspring, Studies.

Brautbar A, Pompeii LA, Dehghan A, Ngwa JS, Nambi V, Virani SS, Rivadeneira F, Uitterlinden AG, Hofman A, Witteman JC, Pencina MJ, Folsom AR, Cupples LA, Ballantyne CM, Boerwinkle E.

Atherosclerosis. 2012 Aug;223(2):421-6. doi: 10.1016/j.atherosclerosis.2012.05.035. Epub 2012 Jun 12.

18.

Clinical Utility of Multimarker Genetic Risk Scores for Prediction of Incident Coronary Heart Disease: A Cohort Study Among Over 51 000 Individuals of European Ancestry.

Iribarren C, Lu M, Jorgenson E, Martínez M, Lluis-Ganella C, Subirana I, Salas E, Elosua R.

Circ Cardiovasc Genet. 2016 Dec;9(6):531-540. doi: 10.1161/CIRCGENETICS.116.001522. Epub 2016 Oct 25. Erratum in: Circ Cardiovasc Genet. 2017 Apr;10 (2):. Title corrected.

PMID:
27780846
19.

The relevance of the individual genetic background for the toxicokinetics of two significant neurodevelopmental toxicants: mercury and lead.

Gundacker C, Gencik M, Hengstschläger M.

Mutat Res. 2010 Oct;705(2):130-40. doi: 10.1016/j.mrrev.2010.06.003. Epub 2010 Jun 30. Review.

PMID:
20601101
20.

Genetic predisposition to coronary heart disease and stroke using an additive genetic risk score: a population-based study in Greece.

Yiannakouris N, Katsoulis M, Dilis V, Parnell LD, Trichopoulos D, Ordovas JM, Trichopoulou A.

Atherosclerosis. 2012 May;222(1):175-9. doi: 10.1016/j.atherosclerosis.2012.02.033. Epub 2012 Feb 28.

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