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Items: 1 to 20 of 106

1.

Autophagy suppresses host adaptive immune responses toward Borrelia burgdorferi.

Buffen K, Oosting M, Li Y, Kanneganti TD, Netea MG, Joosten LA.

J Leukoc Biol. 2016 Sep;100(3):589-98. doi: 10.1189/jlb.4A0715-331R. Epub 2016 Apr 21.

PMID:
27101991
2.

Early production of IL-22 but not IL-17 by peripheral blood mononuclear cells exposed to live Borrelia burgdorferi: the role of monocytes and interleukin-1.

Bachmann M, Horn K, Rudloff I, Goren I, Holdener M, Christen U, Darsow N, Hunfeld KP, Koehl U, Kind P, Pfeilschifter J, Kraiczy P, Mühl H.

PLoS Pathog. 2010 Oct 14;6(10):e1001144. doi: 10.1371/journal.ppat.1001144.

3.

Adaptive and innate immune responsiveness to Borrelia burgdorferi sensu lato in exposed asymptomatic children and children with previous clinical Lyme borreliosis.

Skogman BH, Hellberg S, Ekerfelt C, Jenmalm MC, Forsberg P, Ludvigsson J, Bergström S, Ernerudh J.

Clin Dev Immunol. 2012;2012:294587. doi: 10.1155/2012/294587. Epub 2011 Dec 5.

4.

Functional and Genomic Architecture of Borrelia burgdorferi-Induced Cytokine Responses in Humans.

Oosting M, Kerstholt M, Ter Horst R, Li Y, Deelen P, Smeekens S, Jaeger M, Lachmandas E, Vrijmoeth H, Lupse M, Flonta M, Cramer RA, Kullberg BJ, Kumar V, Xavier R, Wijmenga C, Netea MG, Joosten LA.

Cell Host Microbe. 2016 Dec 14;20(6):822-833. doi: 10.1016/j.chom.2016.10.006. Epub 2016 Nov 3.

5.

Increased expression of Fas receptor and Fas ligand in the culture of the peripheral blood mononuclear cells stimulated with Borrelia burgdorferi sensu lato.

Grygorczuk S, Osada J, Moniuszko A, Świerzbińska R, Kondrusik M, Zajkowska J, Dunaj J, Dąbrowska M, Pancewicz S.

Ticks Tick Borne Dis. 2015 Mar;6(2):189-97. doi: 10.1016/j.ttbdis.2014.12.004. Epub 2014 Dec 22.

PMID:
25541498
6.

CD4+ cell-derived interleukin-17 in a model of dysregulated, Borrelia-induced arthritis.

Hansen ES, Johnson ME, Schell RF, Nardelli DT.

Pathog Dis. 2016 Oct;74(7). pii: ftw084. doi: 10.1093/femspd/ftw084. Epub 2016 Aug 21.

PMID:
27549424
7.

Synthesis of Th17 cytokines in the culture of peripheral blood mononuclear cells stimulated with Borrelia burgdorferi sensu lato.

Grygorczuk S, Świerzbińska R, Moniuszko A, Kondrusik M, Zajkowska J, Czupryna P, Dunaj J, Pancewicz S.

Ann Agric Environ Med. 2016 Jun 2;23(2):242-7. doi: 10.5604/12321966.1203884.

8.

MicroRNA-146a provides feedback regulation of lyme arthritis but not carditis during infection with Borrelia burgdorferi.

Lochhead RB, Ma Y, Zachary JF, Baltimore D, Zhao JL, Weis JH, O'Connell RM, Weis JJ.

PLoS Pathog. 2014 Jun 26;10(6):e1004212. doi: 10.1371/journal.ppat.1004212. eCollection 2014 Jun.

9.

Borrelia burgdorferi RNA induces type I and III interferons via Toll-like receptor 7 and contributes to production of NF-κB-dependent cytokines.

Love AC, Schwartz I, Petzke MM.

Infect Immun. 2014 Jun;82(6):2405-16. doi: 10.1128/IAI.01617-14. Epub 2014 Mar 24.

10.

Autophagy modulates Borrelia burgdorferi-induced production of interleukin-1β (IL-1β).

Buffen K, Oosting M, Mennens S, Anand PK, Plantinga TS, Sturm P, van de Veerdonk FL, van der Meer JW, Xavier RJ, Kanneganti TD, Netea MG, Joosten LA.

J Biol Chem. 2013 Mar 22;288(12):8658-66. doi: 10.1074/jbc.M112.412841. Epub 2013 Feb 5.

11.

Interleukin-10 (IL-10) inhibits Borrelia burgdorferi-induced IL-17 production and attenuates IL-17-mediated Lyme arthritis.

Hansen ES, Medić V, Kuo J, Warner TF, Schell RF, Nardelli DT.

Infect Immun. 2013 Dec;81(12):4421-30. doi: 10.1128/IAI.01129-13. Epub 2013 Sep 16.

12.

Dual role for Fcγ receptors in host defense and disease in Borrelia burgdorferi-infected mice.

Belperron AA, Liu N, Booth CJ, Bockenstedt LK.

Front Cell Infect Microbiol. 2014 Jun 11;4:75. doi: 10.3389/fcimb.2014.00075. eCollection 2014.

13.

Induction of Interleukin 10 by Borrelia burgdorferi Is Regulated by the Action of CD14-Dependent p38 Mitogen-Activated Protein Kinase and cAMP-Mediated Chromatin Remodeling.

Sahay B, Bashant K, Nelson NLJ, Patsey RL, Gadila SK, Boohaker R, Verma A, Strle K, Sellati TJ.

Infect Immun. 2018 Mar 22;86(4). pii: e00781-17. doi: 10.1128/IAI.00781-17. Print 2018 Apr.

15.

Borrelia-primed and -infected mice deficient of interleukin-17 develop arthritis after neutralization of gamma-interferon.

Kuo J, Warner TF, Schell RF.

Pathog Dis. 2017 Mar 1;75(2). doi: 10.1093/femspd/ftx014.

PMID:
28175297
16.

Role of interleukin-23 (IL-23) receptor signaling for IL-17 responses in human Lyme disease.

Oosting M, ter Hofstede H, van de Veerdonk FL, Sturm P, Kullberg BJ, van der Meer JW, Netea MG, Joosten LA.

Infect Immun. 2011 Nov;79(11):4681-7. doi: 10.1128/IAI.05242-11. Epub 2011 Sep 6.

17.

Borrelia species induce inflammasome activation and IL-17 production through a caspase-1-dependent mechanism.

Oosting M, van de Veerdonk FL, Kanneganti TD, Sturm P, Verschueren I, Berende A, van der Meer JW, Kullberg BJ, Netea MG, Joosten LA.

Eur J Immunol. 2011 Jan;41(1):172-81. doi: 10.1002/eji.201040385. Epub 2010 Dec 9.

18.

The caspase 1 inflammasome is not required for control of murine Lyme borreliosis.

Liu N, Belperron AA, Booth CJ, Bockenstedt LK.

Infect Immun. 2009 Aug;77(8):3320-7. doi: 10.1128/IAI.00100-09. Epub 2009 Jun 1.

19.

Infection of Interleukin 17 Receptor A-Deficient C3H Mice with Borrelia burgdorferi Does Not Affect Their Development of Lyme Arthritis and Carditis.

Lasky CE, Jamison KE, Sidelinger DR, Pratt CL, Zhang G, Brown CR.

Infect Immun. 2015 Jul;83(7):2882-8. doi: 10.1128/IAI.00533-15. Epub 2015 May 4.

20.

Borrelia burgdorferi, an extracellular pathogen, circumvents osteopontin in inducing an inflammatory cytokine response.

Craig-Mylius K, Weber GF, Coburn J, Glickstein L.

J Leukoc Biol. 2005 May;77(5):710-8. Epub 2005 Feb 4.

PMID:
15695554

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