Similar articles for PMID: 26820605

Year	Number of Results
2000	1
2002	1
2003	1
2004	1
2005	1
2006	2
2007	5
2008	3
2009	9
2010	7
2011	1
2012	11
2013	26
2014	19
2015	16
2016	21
2017	15
2018	22
2019	14
2020	13
2021	18
2022	11
2023	5
2024	0

1

A novel mutation in the SCN5A gene contributes to arrhythmogenic characteristics of early repolarization syndrome.

Guo Q, Ren L, Chen X, Hou C, Chu J, Pu J, Zhang S. Guo Q, et al. Int J Mol Med. 2016 Mar;37(3):727-33. doi: 10.3892/ijmm.2016.2468. Epub 2016 Jan 26. Int J Mol Med. 2016. PMID: 26820605 Free PMC article.

2

A heterozygous missense SCN5A mutation associated with early repolarization syndrome.

Li N, Wang R, Hou C, Zhang Y, Teng S, Pu J. Li N, et al. Int J Mol Med. 2013 Sep;32(3):661-7. doi: 10.3892/ijmm.2013.1422. Epub 2013 Jun 21. Int J Mol Med. 2013. PMID: 23799537

3

A heterozygous deletion mutation in the cardiac sodium channel gene SCN5A with loss- and gain-of-function characteristics manifests as isolated conduction disease, without signs of Brugada or long QT syndrome.

Zumhagen S, Veldkamp MW, Stallmeyer B, Baartscheer A, Eckardt L, Paul M, Remme CA, Bhuiyan ZA, Bezzina CR, Schulze-Bahr E. Zumhagen S, et al. PLoS One. 2013 Jun 28;8(6):e67963. doi: 10.1371/journal.pone.0067963. Print 2013. PLoS One. 2013. PMID: 23840796 Free PMC article.

4

De Novo Mutation in the SCN5A Gene Associated with Brugada Syndrome.

Wang L, Meng X, Yuchi Z, Zhao Z, Xu D, Fedida D, Wang Z, Huang C. Wang L, et al. Cell Physiol Biochem. 2015;36(6):2250-62. doi: 10.1159/000430189. Epub 2015 Jul 24. Cell Physiol Biochem. 2015. PMID: 26279430 Free article.

5

Enhanced fast-inactivated state stability of cardiac sodium channels by a novel voltage sensor SCN5A mutation, R1632C, as a cause of atypical Brugada syndrome.

Nakajima T, Kaneko Y, Saito A, Ota M, Iijima T, Kurabayashi M. Nakajima T, et al. Heart Rhythm. 2015 Nov;12(11):2296-304. doi: 10.1016/j.hrthm.2015.05.032. Epub 2015 May 29. Heart Rhythm. 2015. PMID: 26031372

6

Myotonic dystrophy type 1 mimics and exacerbates Brugada phenotype induced by Nav1.5 sodium channel loss-of-function mutation.

Pambrun T, Mercier A, Chatelier A, Patri S, Schott JJ, Le Scouarnec S, Chahine M, Degand B, Bois P. Pambrun T, et al. Heart Rhythm. 2014 Aug;11(8):1393-400. doi: 10.1016/j.hrthm.2014.04.026. Epub 2014 Apr 21. Heart Rhythm. 2014. PMID: 24768612

7

Relationship between sodium channel function and clinical phenotype in SCN5A variants associated with Brugada syndrome.

Pearman CM, Denham NC, Mills RW, Ding WY, Modi SS, Hall MCS, Todd DM, Mahida S. Pearman CM, et al. Hum Mutat. 2020 Dec;41(12):2195-2204. doi: 10.1002/humu.24128. Epub 2020 Nov 11. Hum Mutat. 2020. PMID: 33131149 Free PMC article.

8

Concomitant Brugada-like and short QT electrocardiogram linked to SCN5A mutation.

Hong K, Hu J, Yu J, Brugada R. Hong K, et al. Eur J Hum Genet. 2012 Nov;20(11):1189-92. doi: 10.1038/ejhg.2012.63. Epub 2012 Apr 11. Eur J Hum Genet. 2012. PMID: 22490985 Free PMC article.

9

Distinct Features of Probands With Early Repolarization and Brugada Syndromes Carrying SCN5A Pathogenic Variants.

Zhang ZH, Barajas-Martínez H, Xia H, Li B, Capra JA, Clatot J, Chen GX, Chen X, Yang B, Jiang H, Tse G, Aizawa Y, Gollob MH, Scheinman M, Antzelevitch C, Hu D. Zhang ZH, et al. J Am Coll Cardiol. 2021 Oct 19;78(16):1603-1617. doi: 10.1016/j.jacc.2021.08.024. J Am Coll Cardiol. 2021. PMID: 34649698 Free PMC article.

10

Further Insights in the Most Common SCN5A Mutation Causing Overlapping Phenotype of Long QT Syndrome, Brugada Syndrome, and Conduction Defect.

Veltmann C, Barajas-Martinez H, Wolpert C, Borggrefe M, Schimpf R, Pfeiffer R, Cáceres G, Burashnikov E, Antzelevitch C, Hu D. Veltmann C, et al. J Am Heart Assoc. 2016 Jul 5;5(7):e003379. doi: 10.1161/JAHA.116.003379. J Am Heart Assoc. 2016. PMID: 27381756 Free PMC article.

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