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Items: 1 to 20 of 236


Alpha-Synuclein Expression Restricts RNA Viral Infections in the Brain.

Beatman EL, Massey A, Shives KD, Burrack KS, Chamanian M, Morrison TE, Beckham JD.

J Virol. 2015 Dec 30;90(6):2767-82. doi: 10.1128/JVI.02949-15.


CCR5 limits cortical viral loads during West Nile virus infection of the central nervous system.

Durrant DM, Daniels BP, Pasieka T, Dorsey D, Klein RS.

J Neuroinflammation. 2015 Dec 15;12:233. doi: 10.1186/s12974-015-0447-9.


CD8+ T cells use TRAIL to restrict West Nile virus pathogenesis by controlling infection in neurons.

Shrestha B, Pinto AK, Green S, Bosch I, Diamond MS.

J Virol. 2012 Sep;86(17):8937-48. doi: 10.1128/JVI.00673-12. Epub 2012 Jun 27.


Viral pathogen-associated molecular patterns regulate blood-brain barrier integrity via competing innate cytokine signals.

Daniels BP, Holman DW, Cruz-Orengo L, Jujjavarapu H, Durrant DM, Klein RS.

MBio. 2014 Aug 26;5(5):e01476-14. doi: 10.1128/mBio.01476-14.


The innate immune adaptor molecule MyD88 restricts West Nile virus replication and spread in neurons of the central nervous system.

Szretter KJ, Daffis S, Patel J, Suthar MS, Klein RS, Gale M Jr, Diamond MS.

J Virol. 2010 Dec;84(23):12125-38. doi: 10.1128/JVI.01026-10. Epub 2010 Sep 29.


IPS-1 is essential for the control of West Nile virus infection and immunity.

Suthar MS, Ma DY, Thomas S, Lund JM, Zhang N, Daffis S, Rudensky AY, Bevan MJ, Clark EA, Kaja MK, Diamond MS, Gale M Jr.

PLoS Pathog. 2010 Feb 5;6(2):e1000757. doi: 10.1371/journal.ppat.1000757.


CD40-CD40 ligand interactions promote trafficking of CD8+ T cells into the brain and protection against West Nile virus encephalitis.

Sitati E, McCandless EE, Klein RS, Diamond MS.

J Virol. 2007 Sep;81(18):9801-11. Epub 2007 Jul 11.


Tumor necrosis factor alpha protects against lethal West Nile virus infection by promoting trafficking of mononuclear leukocytes into the central nervous system.

Shrestha B, Zhang B, Purtha WE, Klein RS, Diamond MS.

J Virol. 2008 Sep;82(18):8956-64. doi: 10.1128/JVI.01118-08. Epub 2008 Jul 16.


IL-1β signaling promotes CNS-intrinsic immune control of West Nile virus infection.

Ramos HJ, Lanteri MC, Blahnik G, Negash A, Suthar MS, Brassil MM, Sodhi K, Treuting PM, Busch MP, Norris PJ, Gale M Jr.

PLoS Pathog. 2012;8(11):e1003039. doi: 10.1371/journal.ppat.1003039. Epub 2012 Nov 29.


Toll-like receptor 3 has a protective role against West Nile virus infection.

Daffis S, Samuel MA, Suthar MS, Gale M Jr, Diamond MS.

J Virol. 2008 Nov;82(21):10349-58. doi: 10.1128/JVI.00935-08. Epub 2008 Aug 20.


The Interferon-Stimulated Gene Ifitm3 Restricts West Nile Virus Infection and Pathogenesis.

Gorman MJ, Poddar S, Farzan M, Diamond MS.

J Virol. 2016 Aug 26;90(18):8212-25. doi: 10.1128/JVI.00581-16. Print 2016 Sep 15.


2'-O methylation of the viral mRNA cap by West Nile virus evades ifit1-dependent and -independent mechanisms of host restriction in vivo.

Szretter KJ, Daniels BP, Cho H, Gainey MD, Yokoyama WM, Gale M Jr, Virgin HW, Klein RS, Sen GC, Diamond MS.

PLoS Pathog. 2012;8(5):e1002698. doi: 10.1371/journal.ppat.1002698. Epub 2012 May 10.


Replication and clearance of Venezuelan equine encephalitis virus from the brains of animals vaccinated with chimeric SIN/VEE viruses.

Paessler S, Ni H, Petrakova O, Fayzulin RZ, Yun N, Anishchenko M, Weaver SC, Frolov I.

J Virol. 2006 Mar;80(6):2784-96.


Differentiation of neurons restricts Arbovirus replication and increases expression of the alpha isoform of IRF-7.

Schultz KL, Vernon PS, Griffin DE.

J Virol. 2015 Jan;89(1):48-60. doi: 10.1128/JVI.02394-14. Epub 2014 Oct 15.


Cell-specific IRF-3 responses protect against West Nile virus infection by interferon-dependent and -independent mechanisms.

Daffis S, Samuel MA, Keller BC, Gale M Jr, Diamond MS.

PLoS Pathog. 2007 Jul 27;3(7):e106.


Venezuelan Equine Encephalitis Virus Induces Apoptosis through the Unfolded Protein Response Activation of EGR1.

Baer A, Lundberg L, Swales D, Waybright N, Pinkham C, Dinman JD, Jacobs JL, Kehn-Hall K.

J Virol. 2016 Jan 20;90(7):3558-72. doi: 10.1128/JVI.02827-15.

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