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Items: 1 to 20 of 96

1.

Synergistic role of ADP and Ca(2+) in diastolic myocardial stiffness.

Sequeira V, Najafi A, McConnell M, Fowler ED, Bollen IA, Wüst RC, dos Remedios C, Helmes M, White E, Stienen GJ, Tardiff J, Kuster DW, van der Velden J.

J Physiol. 2015 Sep 1;593(17):3899-916. doi: 10.1113/JP270354. Epub 2015 Jul 14.

2.

Decreased creatine kinase is linked to diastolic dysfunction in rats with right heart failure induced by pulmonary artery hypertension.

Fowler ED, Benoist D, Drinkhill MJ, Stones R, Helmes M, Wüst RC, Stienen GJ, Steele DS, White E.

J Mol Cell Cardiol. 2015 Sep;86:1-8. doi: 10.1016/j.yjmcc.2015.06.016. Epub 2015 Jun 24.

3.

Mouse intact cardiac myocyte mechanics: cross-bridge and titin-based stress in unactivated cells.

King NM, Methawasin M, Nedrud J, Harrell N, Chung CS, Helmes M, Granzier H.

J Gen Physiol. 2011 Jan;137(1):81-91. doi: 10.1085/jgp.201010499.

4.

Inhibition of the creatine kinase reaction decreases the contractile reserve of isolated rat hearts.

Hamman BL, Bittl JA, Jacobus WE, Allen PD, Spencer RS, Tian R, Ingwall JS.

Am J Physiol. 1995 Sep;269(3 Pt 2):H1030-6.

PMID:
7573498
5.

Reduced length-dependent cross-bridge recruitment in skinned fiber preparations of human failing myocardium.

Brixius K, Savidou-Zaroti P, Bloch W, Schwinger RH.

Eur J Appl Physiol. 2003 May;89(3-4):249-56. Epub 2003 Feb 28.

PMID:
12736832
6.

Failure to maintain a low ADP concentration impairs diastolic function in hypertrophied rat hearts.

Tian R, Nascimben L, Ingwall JS, Lorell BH.

Circulation. 1997 Aug 19;96(4):1313-9.

PMID:
9286964
7.

Role of MgADP in the development of diastolic dysfunction in the intact beating rat heart.

Tian R, Christe ME, Spindler M, Hopkins JC, Halow JM, Camacho SA, Ingwall JS.

J Clin Invest. 1997 Feb 15;99(4):745-51.

8.

Alterations of cross-bridge kinetics in human atrial and ventricular myocardium.

Ruf T, Schulte-Baukloh H, Lüdemann J, Posival H, Beyersdorf F, Just H, Holubarsch C.

Cardiovasc Res. 1998 Dec;40(3):580-90.

PMID:
10070500
9.

Creatine kinase inhibitor iodoacetamide antagonizes calcium-stimulated inotropy in cardiomyocytes.

Ren J, Davidoff AJ, Ingwall JS.

Clin Exp Pharmacol Physiol. 2009 Feb;36(2):141-5. doi: 10.1111/j.1440-1681.2008.05034.x. Epub 2008 Aug 29.

PMID:
18761665
10.

Ranolazine improves diastolic dysfunction in isolated myocardium from failing human hearts--role of late sodium current and intracellular ion accumulation.

Sossalla S, Wagner S, Rasenack EC, Ruff H, Weber SL, Schöndube FA, Tirilomis T, Tenderich G, Hasenfuss G, Belardinelli L, Maier LS.

J Mol Cell Cardiol. 2008 Jul;45(1):32-43. doi: 10.1016/j.yjmcc.2008.03.006. Epub 2008 Mar 14.

PMID:
18439620
11.

Nitric oxide's role in the heart: control of beating or breathing?

Paulus WJ, Bronzwaer JG.

Am J Physiol Heart Circ Physiol. 2004 Jul;287(1):H8-13. Review.

12.

Ca2+-independent alterations in diastolic sarcomere length and relaxation kinetics in a mouse model of lipotoxic diabetic cardiomyopathy.

Flagg TP, Cazorla O, Remedi MS, Haim TE, Tones MA, Bahinski A, Numann RE, Kovacs A, Schaffer JE, Nichols CG, Nerbonne JM.

Circ Res. 2009 Jan 2;104(1):95-103. doi: 10.1161/CIRCRESAHA.108.186809. Epub 2008 Nov 20.

PMID:
19023131
13.

In vitro model to study the effects of matrix stiffening on Ca2+ handling and myofilament function in isolated adult rat cardiomyocytes.

van Deel ED, Najafi A, Fontoura D, Valent E, Goebel M, Kardux K, Falcão-Pires I, van der Velden J.

J Physiol. 2017 Jul 15;595(14):4597-4610. doi: 10.1113/JP274460. Epub 2017 Jun 21.

14.

Ca(2+)-dependence of passive properties of cardiac sarcomeres.

Stuyvers BD, Miura M, ter Keurs HE.

Adv Exp Med Biol. 2000;481:353-66; discussion 367-70.

PMID:
10987083
15.

CaMKII-dependent SR Ca leak contributes to doxorubicin-induced impaired Ca handling in isolated cardiac myocytes.

Sag CM, Köhler AC, Anderson ME, Backs J, Maier LS.

J Mol Cell Cardiol. 2011 Nov;51(5):749-59. doi: 10.1016/j.yjmcc.2011.07.016. Epub 2011 Jul 26.

16.

Crucial role for Ca2(+)/calmodulin-dependent protein kinase-II in regulating diastolic stress of normal and failing hearts via titin phosphorylation.

Hamdani N, Krysiak J, Kreusser MM, Neef S, Dos Remedios CG, Maier LS, Krüger M, Backs J, Linke WA.

Circ Res. 2013 Feb 15;112(4):664-74. doi: 10.1161/CIRCRESAHA.111.300105. Epub 2013 Jan 2.

PMID:
23283722
17.

ADP-stimulated contraction: A predictor of thin-filament activation in cardiac disease.

Sequeira V, Najafi A, Wijnker PJ, Dos Remedios CG, Michels M, Kuster DW, van der Velden J.

Proc Natl Acad Sci U S A. 2015 Dec 15;112(50):E7003-12. doi: 10.1073/pnas.1513843112. Epub 2015 Nov 30.

18.

Decreased ATP production and myocardial contractile reserve in metabolic heart disease.

Luptak I, Sverdlov AL, Panagia M, Qin F, Pimentel DR, Croteau D, Siwik DA, Ingwall JS, Bachschmid MM, Balschi JA, Colucci WS.

J Mol Cell Cardiol. 2018 Mar;116:106-114. doi: 10.1016/j.yjmcc.2018.01.017. Epub 2018 Feb 1.

20.

[Metabolic and functional consequences of complete inhibition of creatine kinase by iodoacetamide in the perfused heart].

Korchazhkina OV, Lakomkin VL, Veksler VI, Shteĭnshneĭder AIa, Elizarova GV, Saks VA, Kapel'ko VI, Kupriianov VV.

Biokhimiia. 1992 Feb;57(2):201-13. Russian.

PMID:
1388056

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