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Items: 1 to 20 of 91

1.

Genetic polymorphism of APOB is associated with diabetes mellitus in sickle cell disease.

Zhang X, Zhang W, Saraf SL, Nouraie M, Han J, Gowhari M, Hassan J, Miasnikova G, Sergueeva A, Nekhai S, Kittles R, Machado RF, Garcia JG, Gladwin MT, Steinberg MH, Sebastiani P, McClain DA, Gordeuk VR.

Hum Genet. 2015 Aug;134(8):895-904. doi: 10.1007/s00439-015-1572-3. Epub 2015 May 30.

2.

Association between apolipoprotein ε4 allele, factor V Leiden, and plasma lipid and lipoprotein levels with sickle cell disease in Southern Iran.

Rahimi Z, Vaisi-Raygani A, Pourmotabbed T.

Mol Biol Rep. 2011 Feb;38(2):703-10. doi: 10.1007/s11033-010-0157-4. Epub 2010 Apr 7.

PMID:
20373031
3.

Genome-wide meta-analysis of systolic blood pressure in children with sickle cell disease.

Bhatnagar P, Barron-Casella E, Bean CJ, Milton JN, Baldwin CT, Steinberg MH, Debaun M, Casella JF, Arking DE.

PLoS One. 2013 Sep 13;8(9):e74193. doi: 10.1371/journal.pone.0074193. eCollection 2013.

4.

Several genetic polymorphisms interact with overweight/obesity to influence serum lipid levels.

Yin RX, Wu DF, Miao L, Aung LH, Cao XL, Yan TT, Long XJ, Liu WY, Zhang L, Li M.

Cardiovasc Diabetol. 2012 Oct 8;11:123. doi: 10.1186/1475-2840-11-123.

5.

Significant associations between lipoprotein(a) and corrected apolipoprotein B-100 levels in African-Americans.

Enkhmaa B, Anuurad E, Zhang W, Berglund L.

Atherosclerosis. 2014 Jul;235(1):223-9. doi: 10.1016/j.atherosclerosis.2014.04.035. Epub 2014 May 10.

6.

Do genetic modifiers of high-density lipoprotein cholesterol and triglyceride levels also modify their response to a lifestyle intervention in the setting of obesity and type-2 diabetes mellitus?: The Action for Health in Diabetes (Look AHEAD) study.

Huggins GS, Papandonatos GD, Erar B, Belalcazar LM, Brautbar A, Ballantyne C, Kitabchi AE, Wagenknecht LE, Knowler WC, Pownall HJ, Wing RR, Peter I, McCaffery JM; Genetics Subgroup of the Action for Health in Diabetes (Look AHEAD) Study.

Circ Cardiovasc Genet. 2013 Aug;6(4):391-9. doi: 10.1161/CIRCGENETICS.113.000042. Epub 2013 Jul 16.

7.

Weight status of children with sickle cell disease.

Chawla A, Sprinz PG, Welch J, Heeney M, Usmani N, Pashankar F, Kavanagh P.

Pediatrics. 2013 Apr;131(4):e1168-73. doi: 10.1542/peds.2012-2225. Epub 2013 Mar 4.

PMID:
23460681
8.

DNA polymorphisms at the BCL11A, HBS1L-MYB, and beta-globin loci associate with fetal hemoglobin levels and pain crises in sickle cell disease.

Lettre G, Sankaran VG, Bezerra MA, Araújo AS, Uda M, Sanna S, Cao A, Schlessinger D, Costa FF, Hirschhorn JN, Orkin SH.

Proc Natl Acad Sci U S A. 2008 Aug 19;105(33):11869-74. doi: 10.1073/pnas.0804799105. Epub 2008 Jul 30.

9.

Reduced cholesterol levels in African-American adults with sickle cell disease.

Shores J, Peterson J, VanderJagt D, Glew RH.

J Natl Med Assoc. 2003 Sep;95(9):813-7.

10.

Association of the MLXIPL/TBL2 rs17145738 SNP and serum lipid levels in the Guangxi Mulao and Han populations.

Zeng XN, Yin RX, Huang P, Huang KK, Wu J, Guo T, Lin QZ, Aung LH, Wu JZ, Wang YM.

Lipids Health Dis. 2013 Oct 25;12:156. doi: 10.1186/1476-511X-12-156.

11.

A comparison of type 2 diabetes risk allele load between African Americans and European Americans.

Keaton JM, Cooke Bailey JN, Palmer ND, Freedman BI, Langefeld CD, Ng MC, Bowden DW.

Hum Genet. 2014 Dec;133(12):1487-95. doi: 10.1007/s00439-014-1486-5. Epub 2014 Oct 2.

12.

Prevalence of pain-related single nucleotide polymorphisms in patients of African origin with sickle cell disease.

Jhun EH, Yao Y, He Y, Mack AK, Wilkie DJ, Molokie RE, Wang ZJ.

Pharmacogenomics. 2015 Nov;16(16):1795-806. doi: 10.2217/pgs.15.126. Epub 2015 Nov 10. Erratum in: Pharmacogenomics. 2016 Apr;17 (6):658. Kyle, Mack A [corrected to Mack, A Kyle].

13.

Endothelial nitric oxide synthase gene polymorphism is associated with sickle cell disease patients in India.

Nishank SS, Singh MP, Yadav R, Gupta RB, Gadge VS, Gwal A.

J Hum Genet. 2013 Dec;58(12):775-9. doi: 10.1038/jhg.2013.99. Epub 2013 Oct 3.

PMID:
24088668
14.

Association analysis in african americans of European-derived type 2 diabetes single nucleotide polymorphisms from whole-genome association studies.

Lewis JP, Palmer ND, Hicks PJ, Sale MM, Langefeld CD, Freedman BI, Divers J, Bowden DW.

Diabetes. 2008 Aug;57(8):2220-5. doi: 10.2337/db07-1319. Epub 2008 Apr 28.

16.

A genetic variation associated with plasma erythropoietin and a non-coding transcript of PRKAR1A in sickle cell disease.

Zhang X, Shah BN, Zhang W, Saraf SL, Miasnikova G, Sergueeva A, Ammosova T, Niu X, Nouraie M, Nekhai S, Castro O, Gladwin MT, Prchal JT, Garcia JG, Machado RF, Gordeuk VR.

Hum Mol Genet. 2016 Oct 15;25(20):4601-4609. doi: 10.1093/hmg/ddw299.

17.

Genetic variants associated with fetal hemoglobin levels show the diverse ethnic origin in Colombian patients with sickle cell anemia.

Fong C, Menzel S, Lizarralde MA, Barreto G.

Biomedica. 2015 Jul-Sep;35(3):437-43. doi: 10.7705/biomedica.v35i3.2573.

18.

Genetic variants at HbF-modifier loci moderate anemia and leukocytosis in sickle cell disease in Tanzania.

Mtatiro SN, Makani J, Mmbando B, Thein SL, Menzel S, Cox SE.

Am J Hematol. 2015 Jan;90(1):E1-4. doi: 10.1002/ajh.23859. Epub 2014 Oct 20.

19.

Original Research: A case-control genome-wide association study identifies genetic modifiers of fetal hemoglobin in sickle cell disease.

Liu L, Pertsemlidis A, Ding LH, Story MD, Steinberg MH, Sebastiani P, Hoppe C, Ballas SK, Pace BS.

Exp Biol Med (Maywood). 2016 Apr;241(7):706-18. doi: 10.1177/1535370216642047. Epub 2016 Mar 27.

20.

The Association of Type 2 Diabetes Loci Identified in Genome-Wide Association Studies with Metabolic Syndrome and Its Components in a Chinese Population with Type 2 Diabetes.

Kong X, Zhang X, Xing X, Zhang B, Hong J, Yang W.

PLoS One. 2015 Nov 24;10(11):e0143607. doi: 10.1371/journal.pone.0143607. eCollection 2015.

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