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Items: 1 to 20 of 147

1.

The mouse NKR-P1B:Clr-b recognition system is a negative regulator of innate immune responses.

Rahim MM, Chen P, Mottashed AN, Mahmoud AB, Thomas MJ, Zhu Q, Brooks CG, Kartsogiannis V, Gillespie MT, Carlyle JR, Makrigiannis AP.

Blood. 2015 Apr 2;125(14):2217-27. doi: 10.1182/blood-2014-02-556142. Epub 2015 Jan 22.

2.

Genetic investigation of MHC-independent missing-self recognition by mouse NK cells using an in vivo bone marrow transplantation model.

Chen P, Aguilar OA, Rahim MM, Allan DS, Fine JH, Kirkham CL, Ma J, Tanaka M, Tu MM, Wight A, Kartsogiannis V, Gillespie MT, Makrigiannis AP, Carlyle JR.

J Immunol. 2015 Mar 15;194(6):2909-18. doi: 10.4049/jimmunol.1401523. Epub 2015 Feb 13.

3.

The Inhibitory NKR-P1B:Clr-b Recognition Axis Facilitates Detection of Oncogenic Transformation and Cancer Immunosurveillance.

Tanaka M, Fine JH, Kirkham CL, Aguilar OA, Belcheva A, Martin A, Ketela T, Moffat J, Allan DSJ, Carlyle JR.

Cancer Res. 2018 Jul 1;78(13):3589-3603. doi: 10.1158/0008-5472.CAN-17-1688. Epub 2018 Apr 24.

4.

Missing self-recognition of Ocil/Clr-b by inhibitory NKR-P1 natural killer cell receptors.

Carlyle JR, Jamieson AM, Gasser S, Clingan CS, Arase H, Raulet DH.

Proc Natl Acad Sci U S A. 2004 Mar 9;101(10):3527-32. Epub 2004 Feb 27.

5.

Expansion and Protection by a Virus-Specific NK Cell Subset Lacking Expression of the Inhibitory NKR-P1B Receptor during Murine Cytomegalovirus Infection.

Rahim MM, Wight A, Mahmoud AB, Aguilar OA, Lee SH, Vidal SM, Carlyle JR, Makrigiannis AP.

J Immunol. 2016 Sep 15;197(6):2325-37. doi: 10.4049/jimmunol.1600776. Epub 2016 Aug 10.

6.

Molecular and genetic basis for strain-dependent NK1.1 alloreactivity of mouse NK cells.

Carlyle JR, Mesci A, Ljutic B, Belanger S, Tai LH, Rousselle E, Troke AD, Proteau MF, Makrigiannis AP.

J Immunol. 2006 Jun 15;176(12):7511-24.

7.

Recognition of host Clr-b by the inhibitory NKR-P1B receptor provides a basis for missing-self recognition.

Balaji GR, Aguilar OA, Tanaka M, Shingu-Vazquez MA, Fu Z, Gully BS, Lanier LL, Carlyle JR, Rossjohn J, Berry R.

Nat Commun. 2018 Nov 5;9(1):4623. doi: 10.1038/s41467-018-06989-2.

8.

Poxvirus infection-associated downregulation of C-type lectin-related-b prevents NK cell inhibition by NK receptor protein-1B.

Williams KJ, Wilson E, Davidson CL, Aguilar OA, Fu L, Carlyle JR, Burshtyn DN.

J Immunol. 2012 May 15;188(10):4980-91. doi: 10.4049/jimmunol.1103425. Epub 2012 Apr 9.

9.

Interferon-Dependent Induction of Clr-b during Mouse Cytomegalovirus Infection Protects Bystander Cells from Natural Killer Cells via NKR-P1B-Mediated Inhibition.

Kirkham CL, Aguilar OA, Yu T, Tanaka M, Mesci A, Chu KL, Fine JH, Mossman KL, Bremner R, Allan DSJ, Carlyle JR.

J Innate Immun. 2017;9(4):343-358. doi: 10.1159/000454926. Epub 2017 Mar 14.

10.

Chemotherapy-induced genotoxic stress promotes sensitivity to natural killer cell cytotoxicity by enabling missing-self recognition.

Fine JH, Chen P, Mesci A, Allan DS, Gasser S, Raulet DH, Carlyle JR.

Cancer Res. 2010 Sep 15;70(18):7102-13. doi: 10.1158/0008-5472.CAN-10-1316. Epub 2010 Sep 7.

11.

Two major groups of rat NKR-P1 receptors can be distinguished based on chromosomal localization, phylogenetic analysis and Clr ligand binding.

Kveberg L, Dai KZ, Westgaard IH, Daws MR, Fossum S, Naper C, Vaage JT.

Eur J Immunol. 2009 Feb;39(2):541-51. doi: 10.1002/eji.200838891.

12.

Cytomegalovirus evasion of innate immunity by subversion of the NKR-P1B:Clr-b missing-self axis.

Voigt S, Mesci A, Ettinger J, Fine JH, Chen P, Chou W, Carlyle JR.

Immunity. 2007 May;26(5):617-27. Epub 2007 Apr 26.

13.

The NKR-P1B gene product is an inhibitory receptor on SJL/J NK cells.

Kung SK, Su RC, Shannon J, Miller RG.

J Immunol. 1999 May 15;162(10):5876-87.

14.

Re-evaluation of the involvement of NK cells and C-type lectin-like NK receptors in modulation of immune responses by multivalent GlcNAc-terminated oligosaccharides.

Grobárová V, Benson V, Rozbeský D, Novák P, Cerný J.

Immunol Lett. 2013 Nov-Dec;156(1-2):110-7. doi: 10.1016/j.imlet.2013.09.009. Epub 2013 Sep 25.

PMID:
24076118
15.

Functional requirements for signaling through the stimulatory and inhibitory mouse NKR-P1 (CD161) NK cell receptors.

Ljutic B, Carlyle JR, Filipp D, Nakagawa R, Julius M, Zúñiga-Pflücker JC.

J Immunol. 2005 Apr 15;174(8):4789-96.

16.

NKR-P1B expression in gut-associated innate lymphoid cells is required for the control of gastrointestinal tract infections.

Abou-Samra E, Hickey Z, Aguilar OA, Scur M, Mahmoud AB, Pyatibrat S, Tu MM, Francispillai J, Mortha A, Carlyle JR, Rahim MMA, Makrigiannis AP.

Cell Mol Immunol. 2018 Oct 1. doi: 10.1038/s41423-018-0169-x. [Epub ahead of print]

PMID:
30275537
17.

Modulation of Clr Ligand Expression and NKR-P1 Receptor Function during Murine Cytomegalovirus Infection.

Aguilar OA, Mesci A, Ma J, Chen P, Kirkham CL, Hundrieser J, Voigt S, Allan DS, Carlyle JR.

J Innate Immun. 2015;7(6):584-600. doi: 10.1159/000382032. Epub 2015 May 29.

18.

Altering the specificity of NK:target cell interactions by genetic manipulation of NK receptor expression on primary mouse NK cells.

Tran J, Mahmood S, Carlyle JR, Kung SK.

Vaccine. 2010 May 14;28(22):3767-72. doi: 10.1016/j.vaccine.2010.03.013. Epub 2010 Mar 23.

PMID:
20338213
19.

Expression cloning and function of the rat NK activating and inhibitory receptors NKR-P1A and -P1B.

Li J, Rabinovich BA, Hurren R, Shannon J, Miller RG.

Int Immunol. 2003 Mar;15(3):411-6.

PMID:
12618485
20.

Mouse NKR-P1B, a novel NK1.1 antigen with inhibitory function.

Carlyle JR, Martin A, Mehra A, Attisano L, Tsui FW, Zúñiga-Pflücker JC.

J Immunol. 1999 May 15;162(10):5917-23.

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