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Items: 1 to 20 of 125

1.

β-Amyloid peptides display protective activity against the human Alzheimer's disease-associated herpes simplex virus-1.

Bourgade K, Garneau H, Giroux G, Le Page AY, Bocti C, Dupuis G, Frost EH, Fülöp T Jr.

Biogerontology. 2015 Feb;16(1):85-98. doi: 10.1007/s10522-014-9538-8. Epub 2014 Nov 7.

PMID:
25376108
2.

Protective Effect of Amyloid-β Peptides Against Herpes Simplex Virus-1 Infection in a Neuronal Cell Culture Model.

Bourgade K, Le Page A, Bocti C, Witkowski JM, Dupuis G, Frost EH, Fülöp T Jr.

J Alzheimers Dis. 2016;50(4):1227-41. doi: 10.3233/JAD-150652.

PMID:
26836158
4.

The helicase-primase inhibitor BAY 57-1293 reduces the Alzheimer's disease-related molecules induced by herpes simplex virus type 1.

Wozniak MA, Frost AL, Itzhaki RF.

Antiviral Res. 2013 Sep;99(3):401-4. doi: 10.1016/j.antiviral.2013.07.003. Epub 2013 Jul 16.

PMID:
23867133
5.

Anti-Viral Properties of Amyloid-β Peptides.

Bourgade K, Dupuis G, Frost EH, Fülöp T.

J Alzheimers Dis. 2016 Oct 4;54(3):859-878. Review.

PMID:
27392871
6.

Herpes simplex virus type 1 DNA is located within Alzheimer's disease amyloid plaques.

Wozniak MA, Mee AP, Itzhaki RF.

J Pathol. 2009 Jan;217(1):131-8. doi: 10.1002/path.2449.

PMID:
18973185
7.

Herpes simplex virus type 2 infection induces AD-like neurodegeneration markers in human neuroblastoma cells.

Kristen H, Santana S, Sastre I, Recuero M, Bullido MJ, Aldudo J.

Neurobiol Aging. 2015 Oct;36(10):2737-47. doi: 10.1016/j.neurobiolaging.2015.06.014. Epub 2015 Jun 18.

PMID:
26163986
8.

Antivirals reduce the formation of key Alzheimer's disease molecules in cell cultures acutely infected with herpes simplex virus type 1.

Wozniak MA, Frost AL, Preston CM, Itzhaki RF.

PLoS One. 2011;6(10):e25152. doi: 10.1371/journal.pone.0025152. Epub 2011 Oct 7.

9.

APP processing induced by herpes simplex virus type 1 (HSV-1) yields several APP fragments in human and rat neuronal cells.

De Chiara G, Marcocci ME, Civitelli L, Argnani R, Piacentini R, Ripoli C, Manservigi R, Grassi C, Garaci E, Palamara AT.

PLoS One. 2010 Nov 15;5(11):e13989. doi: 10.1371/journal.pone.0013989.

10.

Senile plaques in Alzheimer's diseased brains: possible association of beta-amyloid with herpes simplex virus type 1 (HSV-1) L-particles.

Kammerman EM, Neumann DM, Ball MJ, Lukiw W, Hill JM.

Med Hypotheses. 2006;66(2):294-9. Epub 2005 Oct 19.

PMID:
16242250
11.

Acyclovir or Aβ42 peptides attenuate HSV-1-induced miRNA-146a levels in human primary brain cells.

Lukiw WJ, Cui JG, Yuan LY, Bhattacharjee PS, Corkern M, Clement C, Kammerman EM, Ball MJ, Zhao Y, Sullivan PM, Hill JM.

Neuroreport. 2010 Oct 6;21(14):922-7. doi: 10.1097/WNR.0b013e32833da51a.

12.

Herpes simplex virus type I induces the accumulation of intracellular β-amyloid in autophagic compartments and the inhibition of the non-amyloidogenic pathway in human neuroblastoma cells.

Santana S, Recuero M, Bullido MJ, Valdivieso F, Aldudo J.

Neurobiol Aging. 2012 Feb;33(2):430.e19-33. doi: 10.1016/j.neurobiolaging.2010.12.010. Epub 2011 Jan 26.

PMID:
21272962
13.

Herpes simplex virus type 1 infection in neurons leads to production and nuclear localization of APP intracellular domain (AICD): implications for Alzheimer's disease pathogenesis.

Civitelli L, Marcocci ME, Celestino I, Piacentini R, Garaci E, Grassi C, De Chiara G, Palamara AT.

J Neurovirol. 2015 Oct;21(5):480-90. doi: 10.1007/s13365-015-0344-0. Epub 2015 Apr 30.

PMID:
25925093
14.

Herpes simplex virus infection causes cellular beta-amyloid accumulation and secretase upregulation.

Wozniak MA, Itzhaki RF, Shipley SJ, Dobson CB.

Neurosci Lett. 2007 Dec 18;429(2-3):95-100. Epub 2007 Oct 13.

PMID:
17980964
15.

Herpes simplex virus RNAi and neprilysin gene transfer vectors reduce accumulation of Alzheimer's disease-related amyloid-beta peptide in vivo.

Hong CS, Goins WF, Goss JR, Burton EA, Glorioso JC.

Gene Ther. 2006 Jul;13(14):1068-79. Epub 2006 Mar 16.

PMID:
16541122
16.

HSV-1 promotes Ca2+ -mediated APP phosphorylation and Aβ accumulation in rat cortical neurons.

Piacentini R, Civitelli L, Ripoli C, Marcocci ME, De Chiara G, Garaci E, Azzena GB, Palamara AT, Grassi C.

Neurobiol Aging. 2011 Dec;32(12):2323.e13-26. doi: 10.1016/j.neurobiolaging.2010.06.009. Epub 2010 Jul 31.

PMID:
20674092
17.

Alzheimer's Disease-Associated β-Amyloid Is Rapidly Seeded by Herpesviridae to Protect against Brain Infection.

Eimer WA, Vijaya Kumar DK, Navalpur Shanmugam NK, Rodriguez AS, Mitchell T, Washicosky KJ, György B, Breakefield XO, Tanzi RE, Moir RD.

Neuron. 2018 Jul 11;99(1):56-63.e3. doi: 10.1016/j.neuron.2018.06.030.

18.

Herpes Simplex Virus Type 1 and Other Pathogens are Key Causative Factors in Sporadic Alzheimer's Disease.

Harris SA, Harris EA.

J Alzheimers Dis. 2015;48(2):319-53. doi: 10.3233/JAD-142853. Review.

19.

Reactivation of HSV-1 in the brain of patients with familial Alzheimer's disease.

Mori I, Kimura Y, Naiki H, Matsubara R, Takeuchi T, Yokochi T, Nishiyama Y.

J Med Virol. 2004 Aug;73(4):605-11.

PMID:
15221907
20.

Activity of two synthetic amphiphilic peptides and magainin-2 against herpes simplex virus types 1 and 2.

Aboudy Y, Mendelson E, Shalit I, Bessalle R, Fridkin M.

Int J Pept Protein Res. 1994 Jun;43(6):573-82.

PMID:
7928088

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