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CD4+ T cells promote antibody production but not sustained affinity maturation during Borrelia burgdorferi infection.

Elsner RA, Hastey CJ, Baumgarth N.

Infect Immun. 2015 Jan;83(1):48-56. doi: 10.1128/IAI.02471-14. Epub 2014 Oct 13.


Suppression of Long-Lived Humoral Immunity Following Borrelia burgdorferi Infection.

Elsner RA, Hastey CJ, Olsen KJ, Baumgarth N.

PLoS Pathog. 2015 Jul 2;11(7):e1004976. doi: 10.1371/journal.ppat.1004976. eCollection 2015 Jul.


Delays and diversions mark the development of B cell responses to Borrelia burgdorferi infection.

Hastey CJ, Elsner RA, Barthold SW, Baumgarth N.

J Immunol. 2012 Jun 1;188(11):5612-22. doi: 10.4049/jimmunol.1103735. Epub 2012 Apr 30.


Marginal zone B-cell depletion impairs murine host defense against Borrelia burgdorferi infection.

Belperron AA, Dailey CM, Booth CJ, Bockenstedt LK.

Infect Immun. 2007 Jul;75(7):3354-60. Epub 2007 Apr 30.


IgE anti-Borrelia burgdorferi components (p18, p31, p34, p41, p45, p60) and increased blood CD8+CD60+ T cells in children with Lyme disease.

Bluth MH, Robin J, Ruditsky M, Norowitz KB, Chice S, Pytlak E, Nowakowski M, Durkin HG, Smith-Norowitz TA.

Scand J Immunol. 2007 Apr;65(4):376-82.


Analysis of some peripheral blood lymphocyte subsets in relation to Borrelia burgdorferi antibodies in patients with Lyme disease.

Zajkowska JM, Hermanowska-Szpakowicz T, Wysocka J, Kondrusik M, Pancewicz SA, Grygorczuk S.

Rocz Akad Med Bialymst. 2000;45:184-98.


Involvement of CD4+ T lymphocytes in induction of severe destructive Lyme arthritis in inbred LSH hamsters.

Lim LC, England DM, Glowacki NJ, DuChateau BK, Schell RF.

Infect Immun. 1995 Dec;63(12):4818-25.


Borrelia burgdorferi-infected, interleukin-6-deficient mice have decreased Th2 responses and increased lyme arthritis.

Anguita J, Rincón M, Samanta S, Barthold SW, Flavell RA, Fikrig E.

J Infect Dis. 1998 Nov;178(5):1512-5.


Lyme borreliosis in rhesus macaques: effects of corticosteroids on spirochetal load and isotype switching of anti-borrelia burgdorferi antibody.

Pachner AR, Amemiya K, Bartlett M, Schaefer H, Reddy K, Zhang WF.

Clin Diagn Lab Immunol. 2001 Mar;8(2):225-32.


Lymphoadenopathy during lyme borreliosis is caused by spirochete migration-induced specific B cell activation.

Tunev SS, Hastey CJ, Hodzic E, Feng S, Barthold SW, Baumgarth N.

PLoS Pathog. 2011 May;7(5):e1002066. doi: 10.1371/journal.ppat.1002066. Epub 2011 May 26.


Evidence of Borrelia autoimmunity-induced component of Lyme carditis and arthritis.

Raveche ES, Schutzer SE, Fernandes H, Bateman H, McCarthy BA, Nickell SP, Cunningham MW.

J Clin Microbiol. 2005 Feb;43(2):850-6.


NKT cells prevent chronic joint inflammation after infection with Borrelia burgdorferi.

Tupin E, Benhnia MR, Kinjo Y, Patsey R, Lena CJ, Haller MC, Caimano MJ, Imamura M, Wong CH, Crotty S, Radolf JD, Sellati TJ, Kronenberg M.

Proc Natl Acad Sci U S A. 2008 Dec 16;105(50):19863-8. doi: 10.1073/pnas.0810519105. Epub 2008 Dec 5.


Dual role for Fcγ receptors in host defense and disease in Borrelia burgdorferi-infected mice.

Belperron AA, Liu N, Booth CJ, Bockenstedt LK.

Front Cell Infect Microbiol. 2014 Jun 11;4:75. doi: 10.3389/fcimb.2014.00075. eCollection 2014.


Protective antibodies develop, and murine Lyme arthritis regresses, in the absence of MHC class II and CD4+ T cells.

Fikrig E, Barthold SW, Chen M, Chang CH, Flavell RA.

J Immunol. 1997 Dec 1;159(11):5682-6.


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