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Items: 1 to 20 of 111

1.

Expansion of CAG repeats in Escherichia coli is controlled by single-strand DNA exonucleases of both polarities.

Jackson A, Okely EA, Leach DR.

Genetics. 2014 Oct;198(2):509-17. doi: 10.1534/genetics.114.168245. Epub 2014 Jul 31.

2.
3.

Replication restart: a pathway for (CTG).(CAG) repeat deletion in Escherichia coli.

Kim SH, Pytlos MJ, Sinden RR.

Mutat Res. 2006 Mar 20;595(1-2):5-22.

PMID:
16472829
4.

DNA tandem repeat instability in the Escherichia coli chromosome is stimulated by mismatch repair at an adjacent CAG·CTG trinucleotide repeat.

Blackwood JK, Okely EA, Zahra R, Eykelenboom JK, Leach DR.

Proc Natl Acad Sci U S A. 2010 Dec 28;107(52):22582-6. doi: 10.1073/pnas.1012906108. Epub 2010 Dec 13.

5.
6.

Two opposing effects of mismatch repair on CTG repeat instability in Escherichia coli.

Schmidt KH, Abbott CM, Leach DR.

Mol Microbiol. 2000 Jan;35(2):463-71.

7.

Long CTG.CAG repeat sequences markedly stimulate intramolecular recombination.

Napierala M, Parniewski P, Pluciennik A, Wells RD.

J Biol Chem. 2002 Sep 13;277(37):34087-100. Epub 2002 Jun 3.

8.

CTG repeats show bimodal amplification in E. coli.

Sarkar PS, Chang HC, Boudi FB, Reddy S.

Cell. 1998 Nov 13;95(4):531-40.

9.

Involvement of the nucleotide excision repair protein UvrA in instability of CAG*CTG repeat sequences in Escherichia coli.

Oussatcheva EA, Hashem VI, Zou Y, Sinden RR, Potaman VN.

J Biol Chem. 2001 Aug 17;276(33):30878-84. Epub 2001 Jun 18.

10.

Stability of a CTG/CAG trinucleotide repeat in yeast is dependent on its orientation in the genome.

Freudenreich CH, Stavenhagen JB, Zakian VA.

Mol Cell Biol. 1997 Apr;17(4):2090-8.

11.

Replication stalling and heteroduplex formation within CAG/CTG trinucleotide repeats by mismatch repair.

Viterbo D, Michoud G, Mosbach V, Dujon B, Richard GF.

DNA Repair (Amst). 2016 Jun;42:94-106. doi: 10.1016/j.dnarep.2016.03.002. Epub 2016 Mar 16.

12.

Expansion and deletion of triplet repeat sequences in Escherichia coli occur on the leading strand of DNA replication.

Iyer RR, Wells RD.

J Biol Chem. 1999 Feb 5;274(6):3865-77. Erratum in: J Biol Chem 1999 Apr 9;274(15):10668.

13.

Overexpression of the single-stranded DNA-binding protein (SSB) stabilises CAG*CTG triplet repeats in an orientation dependent manner.

Andreoni F, Darmon E, Poon WC, Leach DR.

FEBS Lett. 2010 Jan 4;584(1):153-8. doi: 10.1016/j.febslet.2009.11.042.

14.

R loops stimulate genetic instability of CTG.CAG repeats.

Lin Y, Dent SY, Wilson JH, Wells RD, Napierala M.

Proc Natl Acad Sci U S A. 2010 Jan 12;107(2):692-7. doi: 10.1073/pnas.0909740107. Epub 2009 Dec 22.

15.
16.

The Werner syndrome protein promotes CAG/CTG repeat stability by resolving large (CAG)(n)/(CTG)(n) hairpins.

Chan NL, Hou C, Zhang T, Yuan F, Machwe A, Huang J, Orren DK, Gu L, Li GM.

J Biol Chem. 2012 Aug 31;287(36):30151-6. doi: 10.1074/jbc.M112.389791. Epub 2012 Jul 11.

17.

Length of CTG.CAG repeats determines the influence of mismatch repair on genetic instability.

Parniewski P, Jaworski A, Wells RD, Bowater RP.

J Mol Biol. 2000 Jun 16;299(4):865-74.

PMID:
10843843
18.

(CAG)*(CTG) repeats associated with neurodegenerative diseases are stable in the Escherichia coli chromosome.

Kim SH, Pytlos MJ, Rosche WA, Sinden RR.

J Biol Chem. 2006 Sep 22;281(38):27950-5. Epub 2006 Jul 27.

19.

Age-, tissue- and length-dependent bidirectional somatic CAG•CTG repeat instability in an allelic series of R6/2 Huntington disease mice.

Larson E, Fyfe I, Morton AJ, Monckton DG.

Neurobiol Dis. 2015 Apr;76:98-111. doi: 10.1016/j.nbd.2015.01.004. Epub 2015 Feb 3.

PMID:
25662336

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