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Items: 1 to 20 of 228

1.

The role of Porphyromonas gingivalis gingipains in platelet activation and innate immune modulation.

Klarström Engström K, Khalaf H, Kälvegren H, Bengtsson T.

Mol Oral Microbiol. 2015 Feb;30(1):62-73. doi: 10.1111/omi.12067. Epub 2014 Oct 3.

PMID:
25043711
2.

The periodontal pathogen Porphyromonas gingivalis sensitises human blood platelets to epinephrine.

Nylander M, Lindahl TL, Bengtsson T, Grenegård M.

Platelets. 2008 Aug;19(5):352-8. doi: 10.1080/09537100802056102.

PMID:
18791941
3.

Cleavage of protease-activated receptors on an immortalized oral epithelial cell line by Porphyromonas gingivalis gingipains.

Giacaman RA, Asrani AC, Ross KF, Herzberg MC.

Microbiology. 2009 Oct;155(Pt 10):3238-46. doi: 10.1099/mic.0.029132-0. Epub 2009 Jul 16.

4.

Suppression of inflammatory responses of human gingival fibroblasts by gingipains from Porphyromonas gingivalis.

Palm E, Khalaf H, Bengtsson T.

Mol Oral Microbiol. 2015 Feb;30(1):74-85. doi: 10.1111/omi.12073. Epub 2014 Sep 19.

PMID:
25055828
5.

Porphyromonas gingivalis-induced platelet aggregation in plasma depends on Hgp44 adhesin but not Rgp proteinase.

Naito M, Sakai E, Shi Y, Ideguchi H, Shoji M, Ohara N, Yamamoto K, Nakayama K.

Mol Microbiol. 2006 Jan;59(1):152-67.

6.

Contradictory roles of Porphyromonas gingivalis gingipains in caspase-1 activation.

Jung YJ, Jun HK, Choi BK.

Cell Microbiol. 2015 Sep;17(9):1304-19. doi: 10.1111/cmi.12435. Epub 2015 Apr 16.

PMID:
25759090
7.

Activation of protease-activated receptors by gingipains from Porphyromonas gingivalis leads to platelet aggregation: a new trait in microbial pathogenicity.

Lourbakos A, Yuan YP, Jenkins AL, Travis J, Andrade-Gordon P, Santulli R, Potempa J, Pike RN.

Blood. 2001 Jun 15;97(12):3790-7.

8.

Gingipains inactivate a cell surface ligand on Porphyromonas gingivalis that induces TLR2-and TLR4-independent signaling.

Kishimoto M, Yoshimura A, Naito M, Okamoto K, Yamamoto K, Golenbock DT, Hara Y, Nakayama K.

Microbiol Immunol. 2006;50(4):315-25.

9.

Gingipains: Critical Factors in the Development of Aspiration Pneumonia Caused by Porphyromonas gingivalis.

Benedyk M, Mydel PM, Delaleu N, Płaza K, Gawron K, Milewska A, Maresz K, Koziel J, Pyrc K, Potempa J.

J Innate Immun. 2016;8(2):185-98. doi: 10.1159/000441724. Epub 2015 Nov 28.

10.

Lipoprotein modifications by gingipains of Porphyromonas gingivalis.

Lönn J, Ljunggren S, Klarström-Engström K, Demirel I, Bengtsson T, Karlsson H.

J Periodontal Res. 2018 Jun;53(3):403-413. doi: 10.1111/jre.12527. Epub 2018 Jan 17.

11.

Gingipains from Porphyromonas gingivalis play a significant role in induction and regulation of CXCL8 in THP-1 cells.

Jayaprakash K, Khalaf H, Bengtsson T.

BMC Microbiol. 2014 Jul 18;14:193. doi: 10.1186/1471-2180-14-193.

12.

The role of gingipains in the pathogenesis of periodontal disease.

Imamura T.

J Periodontol. 2003 Jan;74(1):111-8. Review.

PMID:
12593605
13.

Gingipain-dependent augmentation by Porphyromonas gingivalis of phagocytosis of Tannerella forsythia.

Jung YJ, Jun HK, Choi BK.

Mol Oral Microbiol. 2016 Dec;31(6):457-471. doi: 10.1111/omi.12139. Epub 2015 Nov 13.

PMID:
26434368
14.

Antibody responses to Porphyromonas gingivalis infection in a murine abscess model--involvement of gingipains and responses to re-infection.

Yoneda M, Hirofuji T, Motooka N, Anan H, Hamachi T, Miura M, Ishihara Y, Maeda K.

J Periodontal Res. 2003 Dec;38(6):551-6.

PMID:
14632916
15.

Porphyromonas gingivalis gingipains selectively reduce CD14 expression, leading to macrophage hyporesponsiveness to bacterial infection.

Wilensky A, Tzach-Nahman R, Potempa J, Shapira L, Nussbaum G.

J Innate Immun. 2015;7(2):127-135. doi: 10.1159/000365970. Epub 2014 Sep 16.

16.

A role for gingipains in cellular responses and bacterial survival in Porphyromonas gingivalis-infected cells.

Kadowaki T, Takii R, Yamatake K, Kawakubo T, Tsukuba T, Yamamoto K.

Front Biosci. 2007 Sep 1;12:4800-9. Review.

PMID:
17569610
17.

The role of phagocytosis, oxidative burst and neutrophil extracellular traps in the interaction between neutrophils and the periodontal pathogen Porphyromonas gingivalis.

Jayaprakash K, Demirel I, Khalaf H, Bengtsson T.

Mol Oral Microbiol. 2015 Oct;30(5):361-75. doi: 10.1111/omi.12099. Epub 2015 May 4.

PMID:
25869817
18.

Porphyromonas gingivalis selectively up-regulates the HIV-1 coreceptor CCR5 in oral keratinocytes.

Giacaman RA, Nobbs AH, Ross KF, Herzberg MC.

J Immunol. 2007 Aug 15;179(4):2542-50.

19.

VimA is part of the maturation pathway for the major gingipains of Porphyromonas gingivalis W83.

Vanterpool E, Roy F, Zhan W, Sheets SM, Sangberg L, Fletcher HM.

Microbiology. 2006 Nov;152(Pt 11):3383-9.

PMID:
17074907
20.

Kinin danger signals proteolytically released by gingipain induce Fimbriae-specific IFN-gamma- and IL-17-producing T cells in mice infected intramucosally with Porphyromonas gingivalis.

Monteiro AC, Scovino A, Raposo S, Gaze VM, Cruz C, Svensjö E, Narciso MS, Colombo AP, Pesquero JB, Feres-Filho E, Nguyen KA, Sroka A, Potempa J, Scharfstein J.

J Immunol. 2009 Sep 15;183(6):3700-11. doi: 10.4049/jimmunol.0900895. Epub 2009 Aug 17.

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