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Items: 1 to 20 of 183

1.

The fatty acid amide hydrolase inhibitor PF-3845 promotes neuronal survival, attenuates inflammation and improves functional recovery in mice with traumatic brain injury.

Tchantchou F, Tucker LB, Fu AH, Bluett RJ, McCabe JT, Patel S, Zhang Y.

Neuropharmacology. 2014 Oct;85:427-39. doi: 10.1016/j.neuropharm.2014.06.006. Epub 2014 Jun 14.

3.

The fatty acid amide hydrolase (FAAH) inhibitor PF-3845 acts in the nervous system to reverse LPS-induced tactile allodynia in mice.

Booker L, Kinsey SG, Abdullah RA, Blankman JL, Long JZ, Ezzili C, Boger DL, Cravatt BF, Lichtman AH.

Br J Pharmacol. 2012 Apr;165(8):2485-96. doi: 10.1111/j.1476-5381.2011.01445.x.

4.

CB1 and CB2 cannabinoid receptor antagonists prevent minocycline-induced neuroprotection following traumatic brain injury in mice.

Lopez-Rodriguez AB, Siopi E, Finn DP, Marchand-Leroux C, Garcia-Segura LM, Jafarian-Tehrani M, Viveros MP.

Cereb Cortex. 2015 Jan;25(1):35-45. doi: 10.1093/cercor/bht202. Epub 2013 Aug 19.

PMID:
23960212
5.

Targeting the nNOS/peroxynitrite/calpain system to confer neuroprotection and aid functional recovery in a mouse model of TBI.

Khan M, Dhammu TS, Matsuda F, Annamalai B, Dhindsa TS, Singh I, Singh AK.

Brain Res. 2016 Jan 1;1630:159-70. doi: 10.1016/j.brainres.2015.11.015. Epub 2015 Nov 17.

7.

Inhibition of FAAH reduces nitroglycerin-induced migraine-like pain and trigeminal neuronal hyperactivity in mice.

Nozaki C, Markert A, Zimmer A.

Eur Neuropsychopharmacol. 2015 Aug;25(8):1388-96. doi: 10.1016/j.euroneuro.2015.04.001. Epub 2015 Apr 13. Erratum in: Eur Neuropsychopharmacol. 2015 Nov;25(11):2186.

PMID:
25910421
8.

Mitochondrial division inhibitor 1 (Mdivi-1) offers neuroprotection through diminishing cell death and improving functional outcome in a mouse model of traumatic brain injury.

Wu Q, Xia SX, Li QQ, Gao Y, Shen X, Ma L, Zhang MY, Wang T, Li YS, Wang ZF, Luo CL, Tao LY.

Brain Res. 2016 Jan 1;1630:134-43. doi: 10.1016/j.brainres.2015.11.016. Epub 2015 Nov 17.

PMID:
26596858
9.

Experimental colitis in mice is attenuated by changes in the levels of endocannabinoid metabolites induced by selective inhibition of fatty acid amide hydrolase (FAAH).

Sałaga M, Mokrowiecka A, Zakrzewski PK, Cygankiewicz A, Leishman E, Sobczak M, Zatorski H, Małecka-Panas E, Kordek R, Storr M, Krajewska WM, Bradshaw HB, Fichna J.

J Crohns Colitis. 2014 Sep;8(9):998-1009. doi: 10.1016/j.crohns.2014.01.025. Epub 2014 Feb 14.

10.

FAAH inhibition produces antidepressant-like efforts of mice to acute stress via synaptic long-term depression.

Wang Y, Zhang X.

Behav Brain Res. 2017 May 1;324:138-145. doi: 10.1016/j.bbr.2017.01.054. Epub 2017 Feb 11.

PMID:
28193523
11.

Novel mGluR5 positive allosteric modulator improves functional recovery, attenuates neurodegeneration, and alters microglial polarization after experimental traumatic brain injury.

Loane DJ, Stoica BA, Tchantchou F, Kumar A, Barrett JP, Akintola T, Xue F, Conn PJ, Faden AI.

Neurotherapeutics. 2014 Oct;11(4):857-69. doi: 10.1007/s13311-014-0298-6.

12.

Comparison of anandamide transport in FAAH wild-type and knockout neurons: evidence for contributions by both FAAH and the CB1 receptor to anandamide uptake.

Ortega-Gutiérrez S, Hawkins EG, Viso A, López-Rodríguez ML, Cravatt BF.

Biochemistry. 2004 Jun 29;43(25):8184-90.

PMID:
15209515
13.
14.

The endocannabinoid system as a target for novel anxiolytic and antidepressant drugs.

Gaetani S, Dipasquale P, Romano A, Righetti L, Cassano T, Piomelli D, Cuomo V.

Int Rev Neurobiol. 2009;85:57-72. doi: 10.1016/S0074-7742(09)85005-8.

PMID:
19607961
15.

A pro-nociceptive phenotype unmasked in mice lacking fatty-acid amide hydrolase.

Carey LM, Slivicki RA, Leishman E, Cornett B, Mackie K, Bradshaw H, Hohmann AG.

Mol Pain. 2016 May 13;12. pii: 1744806916649192. doi: 10.1177/1744806916649192. Print 2016.

16.

A Nuclear Attack on Traumatic Brain Injury: Sequestration of Cell Death in the Nucleus.

Tajiri N, De La Peña I, Acosta SA, Kaneko Y, Tamir S, Landesman Y, Carlson R, Shacham S, Borlongan CV.

CNS Neurosci Ther. 2016 Apr;22(4):306-15. doi: 10.1111/cns.12501. Epub 2016 Feb 4.

17.

Inhibition of endocannabinoid catabolic enzymes elicits anxiolytic-like effects in the marble burying assay.

Kinsey SG, O'Neal ST, Long JZ, Cravatt BF, Lichtman AH.

Pharmacol Biochem Behav. 2011 Mar;98(1):21-7. doi: 10.1016/j.pbb.2010.12.002. Epub 2010 Dec 8.

18.

Blockade of endocannabinoid hydrolytic enzymes attenuates precipitated opioid withdrawal symptoms in mice.

Ramesh D, Ross GR, Schlosburg JE, Owens RA, Abdullah RA, Kinsey SG, Long JZ, Nomura DK, Sim-Selley LJ, Cravatt BF, Akbarali HI, Lichtman AH.

J Pharmacol Exp Ther. 2011 Oct;339(1):173-85. doi: 10.1124/jpet.111.181370. Epub 2011 Jun 30.

19.

Selective inhibition of FAAH produces antidiarrheal and antinociceptive effect mediated by endocannabinoids and cannabinoid-like fatty acid amides.

Fichna J, Sałaga M, Stuart J, Saur D, Sobczak M, Zatorski H, Timmermans JP, Bradshaw HB, Ahn K, Storr MA.

Neurogastroenterol Motil. 2014 Apr;26(4):470-81. doi: 10.1111/nmo.12272. Epub 2013 Dec 3.

PMID:
24460851
20.

Inhibition of fatty-acid amide hydrolase accelerates acquisition and extinction rates in a spatial memory task.

Varvel SA, Wise LE, Niyuhire F, Cravatt BF, Lichtman AH.

Neuropsychopharmacology. 2007 May;32(5):1032-41. Epub 2006 Oct 18.

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