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Items: 1 to 20 of 268

1.

Loss of p16(INK4a) is associated with reduced patient survival in soft tissue tumours, and indicates a senescence barrier.

Knösel T, Altendorf-Hofmann A, Lindner L, Issels R, Hermeking H, Schuebbe G, Gibis S, Siemens H, Kampmann E, Kirchner T.

J Clin Pathol. 2014 Jul;67(7):592-8. doi: 10.1136/jclinpath-2013-202106. Epub 2014 Apr 19.

PMID:
24747207
2.

Up and downregulation of p16(Ink4a) expression in BRAF-mutated polyps/adenomas indicates a senescence barrier in the serrated route to colon cancer.

Kriegl L, Neumann J, Vieth M, Greten FR, Reu S, Jung A, Kirchner T.

Mod Pathol. 2011 Jul;24(7):1015-22. doi: 10.1038/modpathol.2011.43. Epub 2011 Mar 18.

3.

Frequent alteration of p16(INK4a)/p14(ARF) and p53 pathways in the round cell component of myxoid/round cell liposarcoma: p53 gene alterations and reduced p14(ARF) expression both correlate with poor prognosis.

Oda Y, Yamamoto H, Takahira T, Kobayashi C, Kawaguchi K, Tateishi N, Nozuka Y, Tamiya S, Tanaka K, Matsuda S, Yokoyama R, Iwamoto Y, Tsuneyoshi M.

J Pathol. 2005 Dec;207(4):410-21.

PMID:
16177957
4.

DNA hypermethylation status of multiple genes in soft tissue sarcomas.

Kawaguchi K, Oda Y, Saito T, Yamamoto H, Takahira T, Kobayashi C, Tamiya S, Tateishi N, Iwamoto Y, Tsuneyoshi M.

Mod Pathol. 2006 Jan;19(1):106-14.

5.

Mechanisms of inactivation of the p16INK4a gene in leiomyosarcoma of soft tissue: decreased p16 expression correlates with promoter methylation and poor prognosis.

Kawaguchi K, Oda Y, Saito T, Yamamoto H, Tamiya S, Takahira T, Miyajima K, Iwamoto Y, Tsuneyoshi M.

J Pathol. 2003 Nov;201(3):487-95.

PMID:
14595762
6.
7.

Molecular analysis of the INK4A/INK4A-ARF gene locus in conventional (central) chondrosarcomas and enchondromas: indication of an important gene for tumour progression.

van Beerendonk HM, Rozeman LB, Taminiau AH, Sciot R, Bovée JV, Cleton-Jansen AM, Hogendoorn PC.

J Pathol. 2004 Mar;202(3):359-66.

PMID:
14991902
8.

Up-regulation of expression and lack of 5' CpG island hypermethylation of p16 INK4a in HPV-positive cervical carcinomas.

Ivanova TA, Golovina DA, Zavalishina LE, Volgareva GM, Katargin AN, Andreeva YY, Frank GA, Kisseljov FL, Kisseljova NP.

BMC Cancer. 2007 Mar 14;7:47.

9.

Role for p16(INK4a) in progression of gastrointestinal stromal tumors of the stomach: alteration of p16(INK4a) network members.

Mitomi H, Fukui N, Kishimoto I, Tanabe S, Kikuchi S, Saito T, Hayashi T, Yao T.

Hum Pathol. 2011 Oct;42(10):1505-13. doi: 10.1016/j.humpath.2011.01.005. Epub 2011 Apr 14.

PMID:
21496871
10.

Promoter hypermethylation: a common cause of reduced p16(INK4a) expression in uveal melanoma.

van der Velden PA, Metzelaar-Blok JA, Bergman W, Monique H, Hurks H, Frants RR, Gruis NA, Jager MJ.

Cancer Res. 2001 Jul 1;61(13):5303-6.

11.

Influence of methylated p15 and p16 genes on clinicopathological features in colorectal cancer.

Ishiguro A, Takahata T, Saito M, Yoshiya G, Tamura Y, Sasaki M, Munakata A.

J Gastroenterol Hepatol. 2006 Aug;21(8):1334-9.

PMID:
16872319
12.
13.

Hepatitis B virus X protein overcomes stress-induced premature senescence by repressing p16(INK4a) expression via DNA methylation.

Kim YJ, Jung JK, Lee SY, Jang KL.

Cancer Lett. 2010 Feb 28;288(2):226-35. doi: 10.1016/j.canlet.2009.07.007. Epub 2009 Aug 4.

PMID:
19656618
14.

miR-152 down-regulation is associated with MET up-regulation in leiomyosarcoma and undifferentiated pleomorphic sarcoma.

Pazzaglia L, Novello C, Conti A, Pollino S, Picci P, Benassi MS.

Cell Oncol (Dordr). 2017 Feb;40(1):77-88. doi: 10.1007/s13402-016-0306-4. Epub 2016 Nov 29.

PMID:
27900663
15.

p16INK4a and p14ARF methylation as a potential biomarker for human bladder cancer.

Kawamoto K, Enokida H, Gotanda T, Kubo H, Nishiyama K, Kawahara M, Nakagawa M.

Biochem Biophys Res Commun. 2006 Jan 20;339(3):790-6. Epub 2005 Nov 22.

PMID:
16316628
16.

CtBP2 contributes to malignant development of human esophageal squamous cell carcinoma by regulation of p16INK4A.

Guan C, Shi H, Wang H, Zhang J, Ni W, Chen B, Hou S, Yang X, Shen A, Ni R.

J Cell Biochem. 2013 Jun;114(6):1343-54. doi: 10.1002/jcb.24475.

PMID:
23255392
17.

The PAX3-FKHR fusion gene of rhabdomyosarcoma cooperates with loss of p16INK4A to promote bypass of cellular senescence.

Linardic CM, Naini S, Herndon JE 2nd, Kesserwan C, Qualman SJ, Counter CM.

Cancer Res. 2007 Jul 15;67(14):6691-9.

18.

Relationship between expression and methylation status of p16INK4a and the proliferative activity of different areas' tumour cells in human colorectal cancer.

Jie G, Zhixiang S, Lei S, Hesheng L, Xiaojun T.

Int J Clin Pract. 2007 Sep;61(9):1523-9. Epub 2007 May 30.

PMID:
17537196
19.

The in situ local immune response, tumour senescence and proliferation in colorectal cancer.

Roxburgh CS, Richards CH, Macdonald AI, Powell AG, McGlynn LM, McMillan DC, Horgan PG, Edwards J, Shiels PG.

Br J Cancer. 2013 Oct 15;109(8):2207-16. doi: 10.1038/bjc.2013.556. Epub 2013 Sep 10.

20.

Two mechanisms underlying the loss of p16(Ink4a) function are associated with distinct tumorigenic consequences for WS MEFs escaping from senescence.

Wu X, Jia S, Zhang X, Si X, Tang W, Luo Y.

Mech Ageing Dev. 2012 Aug;133(8):549-55. doi: 10.1016/j.mad.2012.07.002. Epub 2012 Jul 16.

PMID:
22813853

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