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Items: 1 to 20 of 72

1.

CaMKII inhibitors: from research tools to therapeutic agents.

Pellicena P, Schulman H.

Front Pharmacol. 2014 Feb 20;5:21. doi: 10.3389/fphar.2014.00021. Review.

2.

Ca2+/calmodulin-dependent protein kinase II inhibitors disrupt AKAP79-dependent PKC signaling to GluA1 AMPA receptors.

Brooks IM, Tavalin SJ.

J Biol Chem. 2011 Feb 25;286(8):6697-706. doi: 10.1074/jbc.M110.183558.

3.

Calm down when the heart is stressed: Inhibiting calmodulin-dependent protein kinase II for antiarrhythmias.

Duan DD.

Trends Cardiovasc Med. 2015 Jul;25(5):398-400. doi: 10.1016/j.tcm.2015.01.016.

4.

Mechanisms of CaMKII Activation in the Heart.

Erickson JR.

Front Pharmacol. 2014 Apr 2;5:59. doi: 10.3389/fphar.2014.00059. Review.

5.

Phosphoproteomics study based on in vivo inhibition reveals sites of calmodulin-dependent protein kinase II regulation in the heart.

Scholten A, Preisinger C, Corradini E, Bourgonje VJ, Hennrich ML, van Veen TA, Swaminathan PD, Joiner ML, Vos MA, Anderson ME, Heck AJ.

J Am Heart Assoc. 2013 Aug 7;2(4):e000318. doi: 10.1161/JAHA.113.000318.

6.

Role of CaMKII in cardiac arrhythmias.

Hund TJ, Mohler PJ.

Trends Cardiovasc Med. 2015 Jul;25(5):392-7. doi: 10.1016/j.tcm.2014.12.001. Review.

7.

Calcium/calmodulin-dependent protein kinase II (CaMKII) inhibition ameliorates arrhythmias elicited by junctin ablation under stress conditions.

Tzimas C, Terrovitis J, Lehnart SE, Kranias EG, Sanoudou D.

Heart Rhythm. 2015 Jul;12(7):1599-610. doi: 10.1016/j.hrthm.2015.03.043.

8.

Calcium/calmodulin-dependent protein kinase II contributes to cardiac arrhythmogenesis in heart failure.

Sag CM, Wadsack DP, Khabbazzadeh S, Abesser M, Grefe C, Neumann K, Opiela MK, Backs J, Olson EN, Brown JH, Neef S, Maier SK, Maier LS.

Circ Heart Fail. 2009 Nov;2(6):664-75. doi: 10.1161/CIRCHEARTFAILURE.109.865279.

9.

Sinoatrial node pacemaker activity requires Ca(2+)/calmodulin-dependent protein kinase II activation.

Vinogradova TM, Zhou YY, Bogdanov KY, Yang D, Kuschel M, Cheng H, Xiao RP.

Circ Res. 2000 Oct 27;87(9):760-7.

10.

The promise of CaMKII inhibition for heart disease: preventing heart failure and arrhythmias.

Westenbrink BD, Edwards AG, McCulloch AD, Brown JH.

Expert Opin Ther Targets. 2013 Aug;17(8):889-903. doi: 10.1517/14728222.2013.809064. Review.

PMID:
23789646
11.

Involvement of calcium/calmodulin-dependent protein kinase II (CaMKII) in meiotic maturation and activation of pig oocytes.

Fan HY, Huo LJ, Meng XQ, Zhong ZS, Hou Y, Chen DY, Sun QY.

Biol Reprod. 2003 Nov;69(5):1552-64.

12.

CaMKII in the cardiovascular system: sensing redox states.

Erickson JR, He BJ, Grumbach IM, Anderson ME.

Physiol Rev. 2011 Jul;91(3):889-915. doi: 10.1152/physrev.00018.2010. Review.

13.

CaMKII as a target for arrhythmia suppression.

Mustroph J, Neef S, Maier LS.

Pharmacol Ther. 2016 Oct 11. pii: S0163-7258(16)30184-X. doi: 10.1016/j.pharmthera.2016.10.006. [Epub ahead of print] Review.

PMID:
27742568
14.

Molecular mechanism of activation-triggered subunit exchange in Ca(2+)/calmodulin-dependent protein kinase II.

Bhattacharyya M, Stratton MM, Going CC, McSpadden ED, Huang Y, Susa AC, Elleman A, Cao YM, Pappireddi N, Burkhardt P, Gee CL, Barros T, Schulman H, Williams ER, Kuriyan J.

Elife. 2016 Mar 7;5. pii: e13405. doi: 10.7554/eLife.13405.

15.

Enzymatic activity of CaMKII is not required for its interaction with the glutamate receptor subunit GluN2B.

Barcomb K, Coultrap SJ, Bayer KU.

Mol Pharmacol. 2013 Dec;84(6):834-43. doi: 10.1124/mol.113.089045.

16.

A Single Protein Kinase A or Calmodulin Kinase II Site Does Not Control the Cardiac Pacemaker Ca2+ Clock.

Wu Y, Valdivia HH, Wehrens XH, Anderson ME.

Circ Arrhythm Electrophysiol. 2016 Feb;9(2):e003180. doi: 10.1161/CIRCEP.115.003180.

PMID:
26857906
17.

CaMKII inhibition rescues proarrhythmic phenotypes in the model of human ankyrin-B syndrome.

DeGrande S, Nixon D, Koval O, Curran JW, Wright P, Wang Q, Kashef F, Chiang D, Li N, Wehrens XH, Anderson ME, Hund TJ, Mohler PJ.

Heart Rhythm. 2012 Dec;9(12):2034-41. doi: 10.1016/j.hrthm.2012.08.026.

18.

ET-1-induced growth promoting responses involving ERK1/2 and PKB signaling and Egr-1 expression are mediated by Ca2+/CaM-dependent protein kinase-II in vascular smooth muscle cells.

Bouallegue A, Simo Cheyou ER, Anand-Srivastava MB, Srivastava AK.

Cell Calcium. 2013 Dec;54(6):428-35. doi: 10.1016/j.ceca.2013.10.002.

PMID:
24238620
19.

Activation of Ca(2+)-dependent protein kinase II during repeated contractions in single muscle fibres from mouse is dependent on the frequency of sarcoplasmic reticulum Ca(2+) release.

Aydin J, Korhonen T, Tavi P, Allen DG, Westerblad H, Bruton JD.

Acta Physiol (Oxf). 2007 Oct;191(2):131-7. Erratum in: Acta Physiol (Oxf).2008 Jun;193(2):199.

PMID:
17565565
20.

Calmodulin kinase II inhibition shortens action potential duration by upregulation of K+ currents.

Li J, Marionneau C, Zhang R, Shah V, Hell JW, Nerbonne JM, Anderson ME.

Circ Res. 2006 Nov 10;99(10):1092-9.

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