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Items: 1 to 20 of 98

1.

ATM specifically mediates repair of double-strand breaks with blocked DNA ends.

Álvarez-Quilón A, Serrano-Benítez A, Lieberman JA, Quintero C, Sánchez-Gutiérrez D, Escudero LM, Cortés-Ledesma F.

Nat Commun. 2014 Feb 27;5:3347. doi: 10.1038/ncomms4347.

2.

The impact of heterochromatin on DSB repair.

Goodarzi AA, Noon AT, Jeggo PA.

Biochem Soc Trans. 2009 Jun;37(Pt 3):569-76. doi: 10.1042/BST0370569.

PMID:
19442252
3.

DNA-PK triggers histone ubiquitination and signaling in response to DNA double-strand breaks produced during the repair of transcription-blocking topoisomerase I lesions.

Cristini A, Park JH, Capranico G, Legube G, Favre G, Sordet O.

Nucleic Acids Res. 2016 Feb 18;44(3):1161-78. doi: 10.1093/nar/gkv1196. Epub 2015 Nov 17.

4.

ATM prevents DSB formation by coordinating SSB repair and cell cycle progression.

Khoronenkova SV, Dianov GL.

Proc Natl Acad Sci U S A. 2015 Mar 31;112(13):3997-4002. doi: 10.1073/pnas.1416031112. Epub 2015 Mar 16.

5.

Regulation of ATM in DNA double strand break repair accounts for the radiosensitivity in human cells exposed to high linear energy transfer ionizing radiation.

Xue L, Yu D, Furusawa Y, Okayasu R, Tong J, Cao J, Fan S.

Mutat Res. 2009 Nov 2;670(1-2):15-23. doi: 10.1016/j.mrfmmm.2009.06.016. Epub 2009 Jul 5.

PMID:
19583974
6.

Ataxia telangiectasia mutated (ATM) is dispensable for endonuclease I-SceI-induced homologous recombination in mouse embryonic stem cells.

Rass E, Chandramouly G, Zha S, Alt FW, Xie A.

J Biol Chem. 2013 Mar 8;288(10):7086-95. doi: 10.1074/jbc.M112.445825. Epub 2013 Jan 26.

7.

Replication independent DNA double-strand break retention may prevent genomic instability.

Kongruttanachok N, Phuangphairoj C, Thongnak A, Ponyeam W, Rattanatanyong P, Pornthanakasem W, Mutirangura A.

Mol Cancer. 2010 Mar 31;9:70. doi: 10.1186/1476-4598-9-70.

8.

TDP2-dependent non-homologous end-joining protects against topoisomerase II-induced DNA breaks and genome instability in cells and in vivo.

Gómez-Herreros F, Romero-Granados R, Zeng Z, Alvarez-Quilón A, Quintero C, Ju L, Umans L, Vermeire L, Huylebroeck D, Caldecott KW, Cortés-Ledesma F.

PLoS Genet. 2013;9(3):e1003226. doi: 10.1371/journal.pgen.1003226. Epub 2013 Mar 7.

9.

Mutations of the Huntington's disease protein impact on the ATM-dependent signaling and repair pathways of the radiation-induced DNA double-strand breaks: corrective effect of statins and bisphosphonates.

Ferlazzo ML, Sonzogni L, Granzotto A, Bodgi L, Lartin O, Devic C, Vogin G, Pereira S, Foray N.

Mol Neurobiol. 2014 Jun;49(3):1200-11. doi: 10.1007/s12035-013-8591-7. Epub 2013 Nov 26.

PMID:
24277524
10.

Non-redundant Functions of ATM and DNA-PKcs in Response to DNA Double-Strand Breaks.

Caron P, Choudjaye J, Clouaire T, Bugler B, Daburon V, Aguirrebengoa M, Mangeat T, Iacovoni JS, Álvarez-Quilón A, Cortés-Ledesma F, Legube G.

Cell Rep. 2015 Nov 24;13(8):1598-609. doi: 10.1016/j.celrep.2015.10.024. Epub 2015 Nov 12.

11.
12.

[Mosaic forms of ataxia-telangiectasia].

Kuranova ML, Pleskach NM, Ledashcheva TA, Mikhel'son VM, Spivak IM.

Tsitologiia. 2014;56(8):619-29. Russian.

PMID:
25697008
13.

Nuclear ataxia-telangiectasia mutated (ATM) mediates the cellular response to DNA double strand breaks in human neuron-like cells.

Biton S, Dar I, Mittelman L, Pereg Y, Barzilai A, Shiloh Y.

J Biol Chem. 2006 Jun 23;281(25):17482-91. Epub 2006 Apr 20.

15.

Breaks invisible to the DNA damage response machinery accumulate in ATM-deficient cells.

Martín M, Terradas M, Iliakis G, Tusell L, Genescà A.

Genes Chromosomes Cancer. 2009 Sep;48(9):745-59. doi: 10.1002/gcc.20679.

PMID:
19455703
16.

NBN phosphorylation regulates the accumulation of MRN and ATM at sites of DNA double-strand breaks.

Wen J, Cerosaletti K, Schultz KJ, Wright JA, Concannon P.

Oncogene. 2013 Sep 12;32(37):4448-56. doi: 10.1038/onc.2012.443. Epub 2012 Nov 12.

17.

Activation of ataxia telangiectasia mutated by DNA strand break-inducing agents correlates closely with the number of DNA double strand breaks.

Ismail IH, Nyström S, Nygren J, Hammarsten O.

J Biol Chem. 2005 Feb 11;280(6):4649-55. Epub 2004 Nov 15.

19.

ATM is required for the repair of Topotecan-induced replication-associated double-strand breaks.

Köcher S, Spies-Naumann A, Kriegs M, Dahm-Daphi J, Dornreiter I.

Radiother Oncol. 2013 Sep;108(3):409-14. doi: 10.1016/j.radonc.2013.06.024. Epub 2013 Aug 5.

PMID:
23928469
20.

Persistence of unrepaired DNA double strand breaks caused by inhibition of ATM does not lead to radio-sensitisation in the absence of NF-κB activation.

Veuger SJ, Durkacz BW.

DNA Repair (Amst). 2011 Feb 7;10(2):235-44. doi: 10.1016/j.dnarep.2010.11.005. Epub 2010 Dec 8.

PMID:
21144805

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