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Angiogenesis, cardiomyocyte proliferation and anti-fibrotic effects underlie structural preservation post-infarction by intramyocardially-injected cardiospheres.

Tseliou E, de Couto G, Terrovitis J, Sun B, Weixin L, Marbán L, Marbán E.

PLoS One. 2014 Feb 18;9(2):e88590. doi: 10.1371/journal.pone.0088590. eCollection 2014.


Cardiospheres reverse adverse remodeling in chronic rat myocardial infarction: roles of soluble endoglin and Tgf-β signaling.

Tseliou E, Reich H, de Couto G, Terrovitis J, Sun B, Liu W, Marbán E.

Basic Res Cardiol. 2014;109(6):443. doi: 10.1007/s00395-014-0443-8. Epub 2014 Sep 23.


Allogeneic cardiospheres safely boost cardiac function and attenuate adverse remodeling after myocardial infarction in immunologically mismatched rat strains.

Tseliou E, Pollan S, Malliaras K, Terrovitis J, Sun B, Galang G, Marbán L, Luthringer D, Marbán E.

J Am Coll Cardiol. 2013 Mar 12;61(10):1108-19. doi: 10.1016/j.jacc.2012.10.052. Epub 2013 Jan 23.


p38 mitogen-activated protein kinase inhibition improves cardiac function and attenuates left ventricular remodeling following myocardial infarction in the rat.

See F, Thomas W, Way K, Tzanidis A, Kompa A, Lewis D, Itescu S, Krum H.

J Am Coll Cardiol. 2004 Oct 19;44(8):1679-89.


Angiotensin receptor neprilysin inhibitor LCZ696 attenuates cardiac remodeling and dysfunction after myocardial infarction by reducing cardiac fibrosis and hypertrophy.

von Lueder TG, Wang BH, Kompa AR, Huang L, Webb R, Jordaan P, Atar D, Krum H.

Circ Heart Fail. 2015 Jan;8(1):71-8. doi: 10.1161/CIRCHEARTFAILURE.114.001785. Epub 2014 Oct 31.


Cardiac-specific overexpression of diacylglycerol kinase zeta attenuates left ventricular remodeling and improves survival after myocardial infarction.

Niizeki T, Takeishi Y, Arimoto T, Takahashi H, Shishido T, Koyama Y, Goto K, Walsh RA, Kubota I.

Am J Physiol Heart Circ Physiol. 2007 Feb;292(2):H1105-12. Epub 2006 Oct 27.


Rat adipose tissue-derived stem cells transplantation attenuates cardiac dysfunction post infarction and biopolymers enhance cell retention.

Danoviz ME, Nakamuta JS, Marques FL, dos Santos L, Alvarenga EC, dos Santos AA, Antonio EL, Schettert IT, Tucci PJ, Krieger JE.

PLoS One. 2010 Aug 10;5(8):e12077. doi: 10.1371/journal.pone.0012077.


Transplanted human umbilical cord blood mononuclear cells improve left ventricular function through angiogenesis in myocardial infarction.

Hu CH, Wu GF, Wang XQ, Yang YH, Du ZM, He XH, Xiang P.

Chin Med J (Engl). 2006 Sep 20;119(18):1499-506.


Intramyocardial injection of autologous cardiospheres or cardiosphere-derived cells preserves function and minimizes adverse ventricular remodeling in pigs with heart failure post-myocardial infarction.

Lee ST, White AJ, Matsushita S, Malliaras K, Steenbergen C, Zhang Y, Li TS, Terrovitis J, Yee K, Simsir S, Makkar R, Marbán E.

J Am Coll Cardiol. 2011 Jan 25;57(4):455-65. doi: 10.1016/j.jacc.2010.07.049.


Qiliqiangxin improves cardiac function and attenuates cardiac remodeling in rats with experimental myocardial infarction.

Wang J, Zhou J, Ding X, Zhu L, Jiang K, Fu M, Wang S, Hu K, Ge J.

Int J Clin Exp Pathol. 2015 Jun 1;8(6):6596-606. eCollection 2015.


Exercise attenuates inflammation and limits scar thinning after myocardial infarction in mice.

Puhl SL, Müller A, Wagner M, Devaux Y, Böhm M, Wagner DR, Maack C.

Am J Physiol Heart Circ Physiol. 2015 Jul 15;309(2):H345-59. doi: 10.1152/ajpheart.00683.2014. Epub 2015 May 22.


Subtotal nephrectomy accelerates pathological cardiac remodeling post-myocardial infarction: implications for cardiorenal syndrome.

Liu S, Kompa AR, Kumfu S, Nishijima F, Kelly DJ, Krum H, Wang BH.

Int J Cardiol. 2013 Oct 3;168(3):1866-80. doi: 10.1016/j.ijcard.2012.12.065. Epub 2013 Jan 22.


Long-term but not short-term p38 mitogen-activated protein kinase inhibition improves cardiac function and reduces cardiac remodeling post-myocardial infarction.

Kompa AR, See F, Lewis DA, Adrahtas A, Cantwell DM, Wang BH, Krum H.

J Pharmacol Exp Ther. 2008 Jun;325(3):741-50. doi: 10.1124/jpet.107.133546. Epub 2008 Mar 11.


Beneficial effects of delayed ivabradine treatment on cardiac anatomical and electrical remodeling in rat severe chronic heart failure.

Milliez P, Messaoudi S, Nehme J, Rodriguez C, Samuel JL, Delcayre C.

Am J Physiol Heart Circ Physiol. 2009 Feb;296(2):H435-41. doi: 10.1152/ajpheart.00591.2008. Epub 2008 Dec 12.


Increased expression of integrin-linked kinase attenuates left ventricular remodeling and improves cardiac function after myocardial infarction.

Ding L, Dong L, Chen X, Zhang L, Xu X, Ferro A, Xu B.

Circulation. 2009 Sep 1;120(9):764-73. doi: 10.1161/CIRCULATIONAHA.109.870725. Epub 2009 Aug 17.


Early and delayed tranilast treatment reduces pathological fibrosis following myocardial infarction.

See F, Watanabe M, Kompa AR, Wang BH, Boyle AJ, Kelly DJ, Gilbert RE, Krum H.

Heart Lung Circ. 2013 Feb;22(2):122-32. doi: 10.1016/j.hlc.2012.08.054. Epub 2012 Sep 15.


The relative contribution of paracine effect versus direct differentiation on adipose-derived stem cell transplantation mediated cardiac repair.

Yang D, Wang W, Li L, Peng Y, Chen P, Huang H, Guo Y, Xia X, Wang Y, Wang H, Wang WE, Zeng C.

PLoS One. 2013;8(3):e59020. doi: 10.1371/journal.pone.0059020. Epub 2013 Mar 19.


Mitotically inactivated embryonic stem cells can be used as an in vivo feeder layer to nurse damaged myocardium after acute myocardial infarction: a preclinical study.

Burt RK, Chen YH, Verda L, Lucena C, Navale S, Johnson J, Han X, Lomasney J, Baker JM, Ngai KL, Kino A, Carr J, Kajstura J, Anversa P.

Circ Res. 2012 Oct 26;111(10):1286-96. doi: 10.1161/CIRCRESAHA.111.262584. Epub 2012 Aug 22.

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