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Signaling via tumor necrosis factor receptor 1 but not Toll-like receptor 2 contributes significantly to hydrosalpinx development following Chlamydia muridarum infection.

Dong X, Liu Y, Chang X, Lei L, Zhong G.

Infect Immun. 2014 May;82(5):1833-9. doi: 10.1128/IAI.01668-13. Epub 2014 Feb 18.


Complement factor C5 but not C3 contributes significantly to hydrosalpinx development in mice infected with Chlamydia muridarum.

Yang Z, Conrad T, Zhou Z, Chen J, Dutow P, Klos A, Zhong G.

Infect Immun. 2014 Aug;82(8):3154-63. doi: 10.1128/IAI.01833-14. Epub 2014 May 19.


Lack of long-lasting hydrosalpinx in A/J mice correlates with rapid but transient chlamydial ascension and neutrophil recruitment in the oviduct following intravaginal inoculation with Chlamydia muridarum.

Zhang H, Zhou Z, Chen J, Wu G, Yang Z, Zhou Z, Baseman J, Zhang J, Reddick RL, Zhong G.

Infect Immun. 2014 Jul;82(7):2688-96. doi: 10.1128/IAI.00055-14. Epub 2014 Apr 7.


[Roles of Toll-like receptor 2 (TLR2) and TLR4 in immune responses to Chlamydia trachomatis genital tract infection in mice].

Wang Z, Chang X, Hao M, Song G, Zhu X, Zhang Y.

Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2015 Oct;31(10):1336-41. Chinese.


Tumor necrosis factor alpha production from CD8+ T cells mediates oviduct pathological sequelae following primary genital Chlamydia muridarum infection.

Murthy AK, Li W, Chaganty BK, Kamalakaran S, Guentzel MN, Seshu J, Forsthuber TG, Zhong G, Arulanandam BP.

Infect Immun. 2011 Jul;79(7):2928-35. doi: 10.1128/IAI.05022-11. Epub 2011 May 2.


The p47phox deficiency significantly attenuates the pathogenicity of Chlamydia muridarum in the mouse oviduct but not uterine tissues.

Dai J, Tang L, Chen J, Yu P, Chen Z, Zhong G.

Microbes Infect. 2016 Mar;18(3):190-8. doi: 10.1016/j.micinf.2015.11.003. Epub 2015 Dec 2.


Oviduct infection and hydrosalpinx in DBA1/j mice is induced by intracervical but not intravaginal inoculation with Chlamydia muridarum.

Tang L, Zhang H, Lei L, Gong S, Zhou Z, Baseman J, Zhong G.

PLoS One. 2013 Aug 5;8(8):e71649. doi: 10.1371/journal.pone.0071649. Print 2013.


Toll-like receptor-2, but not Toll-like receptor-4, is essential for development of oviduct pathology in chlamydial genital tract infection.

Darville T, O'Neill JM, Andrews CW Jr, Nagarajan UM, Stahl L, Ojcius DM.

J Immunol. 2003 Dec 1;171(11):6187-97.


Reduced live organism recovery and lack of hydrosalpinx in mice infected with plasmid-free Chlamydia muridarum.

Lei L, Chen J, Hou S, Ding Y, Yang Z, Zeng H, Baseman J, Zhong G.

Infect Immun. 2014 Mar;82(3):983-92. doi: 10.1128/IAI.01543-13. Epub 2013 Dec 16.


Plasmid-encoded Pgp3 is a major virulence factor for Chlamydia muridarum to induce hydrosalpinx in mice.

Liu Y, Huang Y, Yang Z, Sun Y, Gong S, Hou S, Chen C, Li Z, Liu Q, Wu Y, Baseman J, Zhong G.

Infect Immun. 2014 Dec;82(12):5327-35. doi: 10.1128/IAI.02576-14. Epub 2014 Oct 6.


Intrauterine infection with plasmid-free Chlamydia muridarum reveals a critical role of the plasmid in chlamydial ascension and establishes a model for evaluating plasmid-independent pathogenicity.

Chen J, Yang Z, Sun X, Tang L, Ding Y, Xue M, Zhou Z, Baseman J, Zhong G.

Infect Immun. 2015 Jun;83(6):2583-92. doi: 10.1128/IAI.00353-15. Epub 2015 Apr 13.


Mice deficient in MyD88 Develop a Th2-dominant response and severe pathology in the upper genital tract following Chlamydia muridarum infection.

Chen L, Lei L, Chang X, Li Z, Lu C, Zhang X, Wu Y, Yeh IT, Zhong G.

J Immunol. 2010 Mar 1;184(5):2602-10. doi: 10.4049/jimmunol.0901593. Epub 2010 Feb 1.


Enhanced virulence of Chlamydia muridarum respiratory infections in the absence of TLR2 activation.

He X, Nair A, Mekasha S, Alroy J, O'Connell CM, Ingalls RR.

PLoS One. 2011;6(6):e20846. doi: 10.1371/journal.pone.0020846. Epub 2011 Jun 14.


Tumor Necrosis Factor (TNF) Receptor Superfamily Member 1b on CD8+ T Cells and TNF Receptor Superfamily Member 1a on Non-CD8+ T Cells Contribute Significantly to Upper Genital Tract Pathology Following Chlamydial Infection.

Manam S, Thomas JD, Li W, Maladore A, Schripsema JH, Ramsey KH, Murthy AK.

J Infect Dis. 2015 Jun 15;211(12):2014-22. doi: 10.1093/infdis/jiu839. Epub 2014 Dec 30.


Enhanced upper genital tract pathologies by blocking Tim-3 and PD-L1 signaling pathways in mice intravaginally infected with Chlamydia muridarum.

Peng B, Lu C, Tang L, Yeh IT, He Z, Wu Y, Zhong G.

BMC Infect Dis. 2011 Dec 14;11:347. doi: 10.1186/1471-2334-11-347.


Vaginal chlamydial clearance following primary or secondary infection in mice occurs independently of TNF-α.

Kamalakaran S, Chaganty BK, Gupta R, Guentzel MN, Chambers JP, Murthy AK, Arulanandam BP.

Front Cell Infect Microbiol. 2013 Mar 11;3:11. doi: 10.3389/fcimb.2013.00011. eCollection 2013.


Plasmid-Encoded Pgp5 Is a Significant Contributor to Chlamydia muridarum Induction of Hydrosalpinx.

Huang Y, Zhang Q, Yang Z, Conrad T, Liu Y, Zhong G.

PLoS One. 2015 Apr 27;10(4):e0124840. doi: 10.1371/journal.pone.0124840. eCollection 2015.


Critical role for interleukin-1beta (IL-1beta) during Chlamydia muridarum genital infection and bacterial replication-independent secretion of IL-1beta in mouse macrophages.

Prantner D, Darville T, Sikes JD, Andrews CW Jr, Brade H, Rank RG, Nagarajan UM.

Infect Immun. 2009 Dec;77(12):5334-46. doi: 10.1128/IAI.00883-09. Epub 2009 Oct 5.


Induction of protective immunity against Chlamydia muridarum intracervical infection in DBA/1j mice.

Tang L, Yang Z, Zhang H, Zhou Z, Arulanandam B, Baseman J, Zhong G.

Vaccine. 2014 Mar 10;32(12):1407-13. doi: 10.1016/j.vaccine.2013.10.018. Epub 2013 Nov 1.


Toll-like receptor 2 activation by Chlamydia trachomatis is plasmid dependent, and plasmid-responsive chromosomal loci are coordinately regulated in response to glucose limitation by C. trachomatis but not by C. muridarum.

O'Connell CM, AbdelRahman YM, Green E, Darville HK, Saira K, Smith B, Darville T, Scurlock AM, Meyer CR, Belland RJ.

Infect Immun. 2011 Mar;79(3):1044-56. doi: 10.1128/IAI.01118-10. Epub 2011 Jan 3.

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