Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 131

1.

Mono-allelic and bi-allelic ENPP1 deficiency promote post-injury neointimal hyperplasia associated with increased C/EBP homologous protein expression.

Serrano RL, Yu W, Terkeltaub R.

Atherosclerosis. 2014 Apr;233(2):493-502. doi: 10.1016/j.atherosclerosis.2014.01.003. Epub 2014 Jan 21.

2.

Connexin37 reduces smooth muscle cell proliferation and intimal hyperplasia in a mouse model of carotid artery ligation.

Allagnat F, Dubuis C, Lambelet M, Le Gal L, Alonso F, Corpataux JM, Déglise S, Haefliger JA.

Cardiovasc Res. 2017 Jun 1;113(7):805-816. doi: 10.1093/cvr/cvx079.

PMID:
28449099
3.

MFAP4 Promotes Vascular Smooth Muscle Migration, Proliferation and Accelerates Neointima Formation.

Schlosser A, Pilecki B, Hemstra LE, Kejling K, Kristmannsdottir GB, Wulf-Johansson H, Moeller JB, Füchtbauer EM, Nielsen O, Kirketerp-Møller K, Dubey LK, Hansen PB, Stubbe J, Wrede C, Hegermann J, Ochs M, Rathkolb B, Schrewe A, Bekeredjian R, Wolf E, Gailus-Durner V, Fuchs H, Hrabě de Angelis M, Lindholt JS, Holmskov U, Sorensen GL.

Arterioscler Thromb Vasc Biol. 2016 Jan;36(1):122-33. doi: 10.1161/ATVBAHA.115.306672. Epub 2015 Nov 12.

4.

Arterial calcification is driven by RAGE in Enpp1-/- mice.

Cecil DL, Terkeltaub RA.

J Vasc Res. 2011;48(3):227-35. doi: 10.1159/000318805. Epub 2010 Nov 23.

5.

Beta3-integrin mediates smooth muscle cell accumulation in neointima after carotid ligation in mice.

Choi ET, Khan MF, Leidenfrost JE, Collins ET, Boc KP, Villa BR, Novack DV, Parks WC, Abendschein DR.

Circulation. 2004 Mar 30;109(12):1564-9. Epub 2004 Mar 8.

6.

Mechanism of matrix metalloproteinase axis-induced neointimal growth.

Guo L, Ning W, Tan Z, Gong Z, Li X.

J Mol Cell Cardiol. 2014 Jan;66:116-25. doi: 10.1016/j.yjmcc.2013.11.014. Epub 2013 Nov 26.

PMID:
24291441
7.

C/EBP-Homologous Protein (CHOP) in Vascular Smooth Muscle Cells Regulates Their Proliferation in Aortic Explants and Atherosclerotic Lesions.

Zhou AX, Wang X, Lin CS, Han J, Yong J, Nadolski MJ, Borén J, Kaufman RJ, Tabas I.

Circ Res. 2015 May 22;116(11):1736-43. doi: 10.1161/CIRCRESAHA.116.305602. Epub 2015 Apr 14.

8.

Aging exacerbates neointimal formation, and increases proliferation and reduces susceptibility to apoptosis of vascular smooth muscle cells in mice.

Vazquez-Padron RI, Lasko D, Li S, Louis L, Pestana IA, Pang M, Liotta C, Fornoni A, Aitouche A, Pham SM.

J Vasc Surg. 2004 Dec;40(6):1199-207.

9.

Thrombospondin-1 activates medial smooth muscle cells and triggers neointima formation upon mouse carotid artery ligation.

Moura R, Tjwa M, Vandervoort P, Cludts K, Hoylaerts MF.

Arterioscler Thromb Vasc Biol. 2007 Oct;27(10):2163-9. Epub 2007 Aug 30.

10.

Xenogenic smooth muscle cell immunization reduces neointimal formation in balloon-injured rabbit carotid arteries.

Shinohara M, Kawashima S, Yamashita T, Takaya T, Toh R, Ishida T, Ueyama T, Inoue N, Hirata K, Yokoyama M.

Cardiovasc Res. 2005 Nov 1;68(2):249-58. Epub 2005 Jul 21.

PMID:
16039635
11.

BubR1 insufficiency inhibits neointimal hyperplasia through impaired vascular smooth muscle cell proliferation in mice.

Kyuragi R, Matsumoto T, Harada Y, Saito S, Onimaru M, Nakatsu Y, Tsuzuki T, Nomura M, Yonemitsu Y, Maehara Y.

Arterioscler Thromb Vasc Biol. 2015 Feb;35(2):341-7. doi: 10.1161/ATVBAHA.114.304737. Epub 2014 Dec 18. Erratum in: Arterioscler Thromb Vasc Biol. 2015 Mar;35(3):e17.

12.

[Role of cytokine-matrix metalloproteinase axis on promoting vascular neointima hyperplasia in mice].

Liu Y, Ning WH, Shen XH, Guo DL, Guo L.

Zhonghua Xin Xue Guan Bing Za Zhi. 2016 Nov 24;44(11):961-967. doi: 10.3760/cma.j.issn.0253-3758.2016.11.012. Chinese.

PMID:
27903395
13.

Inhibition of FOXO1/3 promotes vascular calcification.

Deng L, Huang L, Sun Y, Heath JM, Wu H, Chen Y.

Arterioscler Thromb Vasc Biol. 2015 Jan;35(1):175-83. doi: 10.1161/ATVBAHA.114.304786. Epub 2014 Nov 6.

14.

Npp1 promotes atherosclerosis in ApoE knockout mice.

Nitschke Y, Weissen-Plenz G, Terkeltaub R, Rutsch F.

J Cell Mol Med. 2011 Nov;15(11):2273-83. doi: 10.1111/j.1582-4934.2011.01327.x.

15.

Pyrogallol abates VSMC migration via modulation of Caveolin-1, matrix metalloproteinase and intima hyperplasia in carotid ligation mouse.

Ma YD, Thiyagarajan V, Tsai MJ, Lue SI, Chia YC, Shyue SK, Weng CF.

Environ Toxicol Pharmacol. 2016 Dec;48:63-75. doi: 10.1016/j.etap.2016.10.005. Epub 2016 Oct 15.

PMID:
27768988
16.

miRNA-221 and miRNA-222 synergistically function to promote vascular calcification.

Mackenzie NC, Staines KA, Zhu D, Genever P, Macrae VE.

Cell Biochem Funct. 2014 Mar;32(2):209-16. doi: 10.1002/cbf.3005. Epub 2013 Oct 8.

17.

20-Hydroxyeicosatetraenoic acid inhibition attenuates balloon injury-induced neointima formation and vascular remodeling in rat carotid arteries.

Orozco LD, Liu H, Perkins E, Johnson DA, Chen BB, Fan F, Baker RC, Roman RJ.

J Pharmacol Exp Ther. 2013 Jul;346(1):67-74. doi: 10.1124/jpet.113.203844. Epub 2013 May 8.

18.

Syndecan-4 deficiency limits neointimal formation after vascular injury by regulating vascular smooth muscle cell proliferation and vascular progenitor cell mobilization.

Ikesue M, Matsui Y, Ohta D, Danzaki K, Ito K, Kanayama M, Kurotaki D, Morimoto J, Kojima T, Tsutsui H, Uede T.

Arterioscler Thromb Vasc Biol. 2011 May;31(5):1066-74. doi: 10.1161/ATVBAHA.110.217703. Epub 2011 Feb 17.

19.

Overexpression of c1q/tumor necrosis factor-related protein-3 promotes phosphate-induced vascular smooth muscle cell calcification both in vivo and in vitro.

Zhou Y, Wang JY, Feng H, Wang C, Li L, Wu D, Lei H, Li H, Wu LL.

Arterioscler Thromb Vasc Biol. 2014 May;34(5):1002-10. doi: 10.1161/ATVBAHA.114.303301. Epub 2014 Feb 27.

20.

Deletion of Hyaluronan Synthase 3 Inhibits Neointimal Hyperplasia in Mice.

Kiene LS, Homann S, Suvorava T, Rabausch B, Müller J, Kojda G, Kretschmer I, Twarock S, Dai G, Deenen R, Hartwig S, Lehr S, Köhrer K, Savani RC, Grandoch M, Fischer JW.

Arterioscler Thromb Vasc Biol. 2016 Feb;36(2):e9-16. doi: 10.1161/ATVBAHA.115.306607. Epub 2015 Nov 19.

Supplemental Content

Support Center