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53BP1, BRCA1, and the choice between recombination and end joining at DNA double-strand breaks.

Daley JM, Sung P.

Mol Cell Biol. 2014 Apr;34(8):1380-8. doi: 10.1128/MCB.01639-13. Epub 2014 Jan 27. Review.


A cell cycle-dependent regulatory circuit composed of 53BP1-RIF1 and BRCA1-CtIP controls DNA repair pathway choice.

Escribano-Díaz C, Orthwein A, Fradet-Turcotte A, Xing M, Young JT, Tkáč J, Cook MA, Rosebrock AP, Munro M, Canny MD, Xu D, Durocher D.

Mol Cell. 2013 Mar 7;49(5):872-83. doi: 10.1016/j.molcel.2013.01.001. Epub 2013 Jan 17.


MOF phosphorylation by ATM regulates 53BP1-mediated double-strand break repair pathway choice.

Gupta A, Hunt CR, Hegde ML, Chakraborty S, Chakraborty S, Udayakumar D, Horikoshi N, Singh M, Ramnarain DB, Hittelman WN, Namjoshi S, Asaithamby A, Hazra TK, Ludwig T, Pandita RK, Tyler JK, Pandita TK.

Cell Rep. 2014 Jul 10;8(1):177-89. doi: 10.1016/j.celrep.2014.05.044. Epub 2014 Jun 19. Erratum in: Cell Rep. 2014 Jul 10;8(1):319.


Differences in 53BP1 and BRCA1 regulation between cycling and non-cycling cells.

Croke M, Neumann MA, Grotsky DA, Kreienkamp R, Yaddanapudi SC, Gonzalo S.

Cell Cycle. 2013 Dec 1;12(23):3629-39. doi: 10.4161/cc.26582. Epub 2013 Oct 2.


Regulation of repair pathway choice at two-ended DNA double-strand breaks.

Shibata A.

Mutat Res. 2017 Oct;803-805:51-55. doi: 10.1016/j.mrfmmm.2017.07.011. Epub 2017 Jul 29. Review.


CtIP-BRCA1 modulates the choice of DNA double-strand-break repair pathway throughout the cell cycle.

Yun MH, Hiom K.

Nature. 2009 May 21;459(7245):460-3. doi: 10.1038/nature07955. Epub 2009 Apr 8.


BRCA1-associated exclusion of 53BP1 from DNA damage sites underlies temporal control of DNA repair.

Chapman JR, Sossick AJ, Boulton SJ, Jackson SP.

J Cell Sci. 2012 Aug 1;125(Pt 15):3529-34. Epub 2012 May 2.


PARP3 affects the relative contribution of homologous recombination and nonhomologous end-joining pathways.

Beck C, Boehler C, Guirouilh Barbat J, Bonnet ME, Illuzzi G, Ronde P, Gauthier LR, Magroun N, Rajendran A, Lopez BS, Scully R, Boussin FD, Schreiber V, Dantzer F.

Nucleic Acids Res. 2014 May;42(9):5616-32. doi: 10.1093/nar/gku174. Epub 2014 Mar 5.


Functional crosstalk between DNA damage response proteins 53BP1 and BRCA1 regulates double strand break repair choice.

Bakr A, Köcher S, Volquardsen J, Reimer R, Borgmann K, Dikomey E, Rothkamm K, Mansour WY.

Radiother Oncol. 2016 May;119(2):276-81. doi: 10.1016/j.radonc.2015.11.001. Epub 2015 Nov 23.


DNA double-strand break repair pathway choice and cancer.

Aparicio T, Baer R, Gautier J.

DNA Repair (Amst). 2014 Jul;19:169-75. doi: 10.1016/j.dnarep.2014.03.014. Epub 2014 Apr 18. Review.


Double-strand break repair: 53BP1 comes into focus.

Panier S, Boulton SJ.

Nat Rev Mol Cell Biol. 2014 Jan;15(1):7-18. doi: 10.1038/nrm3719. Epub 2013 Dec 11. Review.


DNA double-strand break repair: a tale of pathway choices.

Li J, Xu X.

Acta Biochim Biophys Sin (Shanghai). 2016 Jul;48(7):641-6. doi: 10.1093/abbs/gmw045. Epub 2016 May 23. Review.


RNF8 regulates assembly of RAD51 at DNA double-strand breaks in the absence of BRCA1 and 53BP1.

Nakada S, Yonamine RM, Matsuo K.

Cancer Res. 2012 Oct 1;72(19):4974-83. doi: 10.1158/0008-5472.CAN-12-1057. Epub 2012 Aug 3.


Impaired 53BP1/RIF1 DSB mediated end-protection stimulates CtIP-dependent end resection and switches the repair to PARP1-dependent end joining in G1.

Bakr A, Köcher S, Volquardsen J, Petersen C, Borgmann K, Dikomey E, Rothkamm K, Mansour WY.

Oncotarget. 2016 Sep 6;7(36):57679-57693. doi: 10.18632/oncotarget.11023.


The p53-binding protein 1-Tudor-interacting repair regulator complex participates in the DNA damage response.

Zhang A, Peng B, Huang P, Chen J, Gong Z.

J Biol Chem. 2017 Apr 21;292(16):6461-6467. doi: 10.1074/jbc.M117.777474. Epub 2017 Feb 17.


HP1 promotes tumor suppressor BRCA1 functions during the DNA damage response.

Lee YH, Kuo CY, Stark JM, Shih HM, Ann DK.

Nucleic Acids Res. 2013 Jun;41(11):5784-98. doi: 10.1093/nar/gkt231. Epub 2013 Apr 15.


53BP1 facilitates long-range DNA end-joining during V(D)J recombination.

Difilippantonio S, Gapud E, Wong N, Huang CY, Mahowald G, Chen HT, Kruhlak MJ, Callen E, Livak F, Nussenzweig MC, Sleckman BP, Nussenzweig A.

Nature. 2008 Nov 27;456(7221):529-33. doi: 10.1038/nature07476. Epub 2008 Oct 19.


Independent phosphoprotein interactions regulate 53BP1 activity.

[No authors listed]

Cancer Discov. 2013 Jul;3(7):OF19. doi: 10.1158/2159-8290.CD-RW2013-127. Epub 2013 Jun 13.


RIF1 is essential for 53BP1-dependent nonhomologous end joining and suppression of DNA double-strand break resection.

Chapman JR, Barral P, Vannier JB, Borel V, Steger M, Tomas-Loba A, Sartori AA, Adams IR, Batista FD, Boulton SJ.

Mol Cell. 2013 Mar 7;49(5):858-71. doi: 10.1016/j.molcel.2013.01.002. Epub 2013 Jan 17.


Class I histone deacetylase inhibitors inhibit the retention of BRCA1 and 53BP1 at the site of DNA damage.

Fukuda T, Wu W, Okada M, Maeda I, Kojima Y, Hayami R, Miyoshi Y, Tsugawa K, Ohta T.

Cancer Sci. 2015 Aug;106(8):1050-6. doi: 10.1111/cas.12717. Epub 2015 Jul 14.

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