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Items: 1 to 20 of 133

1.

HSV-1 degrades, stabilizes, requires, or is stung by STING depending on ICP0, the US3 protein kinase, and cell derivation.

Kalamvoki M, Roizman B.

Proc Natl Acad Sci U S A. 2014 Feb 4;111(5):E611-7. doi: 10.1073/pnas.1323414111. Epub 2014 Jan 21.

2.

Impaired STING Pathway in Human Osteosarcoma U2OS Cells Contributes to the Growth of ICP0-Null Mutant Herpes Simplex Virus.

Deschamps T, Kalamvoki M.

J Virol. 2017 Apr 13;91(9). pii: e00006-17. doi: 10.1128/JVI.00006-17. Print 2017 May 1.

PMID:
28179534
3.

Relative Contributions of Herpes Simplex Virus 1 ICP0 and vhs to Loss of Cellular IFI16 Vary in Different Human Cell Types.

Orzalli MH, Broekema NM, Knipe DM.

J Virol. 2016 Aug 26;90(18):8351-9. doi: 10.1128/JVI.00939-16. Print 2016 Sep 15.

5.

The viral ubiquitin ligase ICP0 is neither sufficient nor necessary for degradation of the cellular DNA sensor IFI16 during herpes simplex virus 1 infection.

Cuchet-Lourenço D, Anderson G, Sloan E, Orr A, Everett RD.

J Virol. 2013 Dec;87(24):13422-32. doi: 10.1128/JVI.02474-13. Epub 2013 Oct 2.

7.

The stability of herpes simplex virus 1 ICP0 early after infection is defined by the RING finger and the UL13 protein kinase.

Zhu Z, Du T, Zhou G, Roizman B.

J Virol. 2014 May;88(10):5437-43. doi: 10.1128/JVI.00542-14. Epub 2014 Feb 26.

8.

Interactions of the Antiviral Factor Interferon Gamma-Inducible Protein 16 (IFI16) Mediate Immune Signaling and Herpes Simplex Virus-1 Immunosuppression.

Diner BA, Lum KK, Javitt A, Cristea IM.

Mol Cell Proteomics. 2015 Sep;14(9):2341-56. doi: 10.1074/mcp.M114.047068. Epub 2015 Feb 18.

9.

ICP0 and the US3 protein kinase of herpes simplex virus 1 independently block histone deacetylation to enable gene expression.

Poon AP, Gu H, Roizman B.

Proc Natl Acad Sci U S A. 2006 Jun 27;103(26):9993-8. Epub 2006 Jun 19.

10.

Roles of Us8A and Its Phosphorylation Mediated by Us3 in Herpes Simplex Virus 1 Pathogenesis.

Kato A, Ando T, Oda S, Watanabe M, Koyanagi N, Arii J, Kawaguchi Y.

J Virol. 2016 May 27;90(12):5622-35. doi: 10.1128/JVI.00446-16. Print 2016 Jun 15.

13.
14.

Interwoven roles of cyclin D3 and cdk4 recruited by ICP0 and ICP4 in the expression of herpes simplex virus genes.

Kalamvoki M, Roizman B.

J Virol. 2010 Oct;84(19):9709-17. doi: 10.1128/JVI.01050-10. Epub 2010 Jul 21.

16.

Cellular Transcriptional Coactivator RanBP10 and Herpes Simplex Virus 1 ICP0 Interact and Synergistically Promote Viral Gene Expression and Replication.

Sato Y, Kato A, Maruzuru Y, Oyama M, Kozuka-Hata H, Arii J, Kawaguchi Y.

J Virol. 2016 Jan 6;90(6):3173-86. doi: 10.1128/JVI.03043-15.

17.

Phosphorylation of a herpes simplex virus 1 dUTPase by a viral protein kinase, Us3, dictates viral pathogenicity in the central nervous system but not at the periphery.

Kato A, Shindo K, Maruzuru Y, Kawaguchi Y.

J Virol. 2014 Mar;88(5):2775-85. doi: 10.1128/JVI.03300-13. Epub 2013 Dec 18.

18.

ICP0 antagonizes Stat 1-dependent repression of herpes simplex virus: implications for the regulation of viral latency.

Halford WP, Weisend C, Grace J, Soboleski M, Carr DJ, Balliet JW, Imai Y, Margolis TP, Gebhardt BM.

Virol J. 2006 Jun 9;3:44.

19.

DNA sensing-independent inhibition of herpes simplex virus 1 replication by DAI/ZBP1.

Pham TH, Kwon KM, Kim YE, Kim KK, Ahn JH.

J Virol. 2013 Mar;87(6):3076-86. doi: 10.1128/JVI.02860-12. Epub 2013 Jan 2.

20.

Nuclear IFI16 induction of IRF-3 signaling during herpesviral infection and degradation of IFI16 by the viral ICP0 protein.

Orzalli MH, DeLuca NA, Knipe DM.

Proc Natl Acad Sci U S A. 2012 Oct 30;109(44):E3008-17. doi: 10.1073/pnas.1211302109. Epub 2012 Oct 1.

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