Sort by

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 163


Rad GTPase deletion increases L-type calcium channel current leading to increased cardiac contraction.

Manning JR, Yin G, Kaminski CN, Magyar J, Feng HZ, Penn J, Sievert G, Thompson K, Jin JP, Andres DA, Satin J.

J Am Heart Assoc. 2013 Dec 12;2(6):e000459. doi: 10.1161/JAHA.113.000459.


Cardiac G-protein-coupled receptor kinase 2 ablation induces a novel Ca2+ handling phenotype resistant to adverse alterations and remodeling after myocardial infarction.

Raake PW, Zhang X, Vinge LE, Brinks H, Gao E, Jaleel N, Li Y, Tang M, Most P, Dorn GW 2nd, Houser SR, Katus HA, Chen X, Koch WJ.

Circulation. 2012 May 1;125(17):2108-18. doi: 10.1161/CIRCULATIONAHA.111.044255.


Enhanced basal contractility but reduced excitation-contraction coupling efficiency and beta-adrenergic reserve of hearts with increased Cav1.2 activity.

Tang M, Zhang X, Li Y, Guan Y, Ai X, Szeto C, Nakayama H, Zhang H, Ge S, Molkentin JD, Houser SR, Chen X.

Am J Physiol Heart Circ Physiol. 2010 Aug;299(2):H519-28. doi: 10.1152/ajpheart.00265.2010.


Increased β-adrenergic inotropy in ventricular myocardium from Trpm4-/- mice.

Mathar I, Kecskes M, Van der Mieren G, Jacobs G, Camacho Londoño JE, Uhl S, Flockerzi V, Voets T, Freichel M, Nilius B, Herijgers P, Vennekens R.

Circ Res. 2014 Jan 17;114(2):283-94. doi: 10.1161/CIRCRESAHA.114.302835.


Rad as a novel regulator of excitation-contraction coupling and beta-adrenergic signaling in heart.

Wang G, Zhu X, Xie W, Han P, Li K, Sun Z, Wang Y, Chen C, Song R, Cao C, Zhang J, Wu C, Liu J, Cheng H.

Circ Res. 2010 Feb 5;106(2):317-27. doi: 10.1161/CIRCRESAHA.109.208272.


Polydatin modulates Ca(2+) handling, excitation-contraction coupling and β-adrenergic signaling in rat ventricular myocytes.

Deng J, Liu W, Wang Y, Dong M, Zheng M, Liu J.

J Mol Cell Cardiol. 2012 Nov;53(5):646-56. doi: 10.1016/j.yjmcc.2012.08.009.


Rem-GTPase regulates cardiac myocyte L-type calcium current.

Magyar J, Kiper CE, Sievert G, Cai W, Shi GX, Crump SM, Li L, Niederer S, Smith N, Andres DA, Satin J.

Channels (Austin). 2012 May-Jun;6(3):166-73. doi: 10.4161/chan.20192.


Dantrolene suppresses spontaneous Ca2+ release without altering excitation-contraction coupling in cardiomyocytes of aged mice.

Domeier TL, Roberts CJ, Gibson AK, Hanft LM, McDonald KS, Segal SS.

Am J Physiol Heart Circ Physiol. 2014 Sep 15;307(6):H818-29. doi: 10.1152/ajpheart.00287.2014.


{beta}1-Adrenergic receptor activation induces mouse cardiac myocyte death through both L-type calcium channel-dependent and -independent pathways.

Wang W, Zhang H, Gao H, Kubo H, Berretta RM, Chen X, Houser SR.

Am J Physiol Heart Circ Physiol. 2010 Aug;299(2):H322-31. doi: 10.1152/ajpheart.00392.2010.


Inotropic response to beta-adrenergic receptor stimulation and anti-adrenergic effect of ACh in endothelial NO synthase-deficient mouse hearts.

Gödecke A, Heinicke T, Kamkin A, Kiseleva I, Strasser RH, Decking UK, Stumpe T, Isenberg G, Schrader J.

J Physiol. 2001 Apr 1;532(Pt 1):195-204.


Loss of Rad-GTPase produces a novel adaptive cardiac phenotype resistant to systolic decline with aging.

Manning JR, Withers CN, Levitan B, Smith JD, Andres DA, Satin J.

Am J Physiol Heart Circ Physiol. 2015 Oct;309(8):H1336-45. doi: 10.1152/ajpheart.00389.2015.


CaMKII-mediated increased lusitropic responses to beta-adrenoreceptor stimulation in ANP-receptor deficient mice.

Yurukova S, Kilić A, Völker K, Leineweber K, Dybkova N, Maier LS, Brodde OE, Kuhn M.

Cardiovasc Res. 2007 Mar 1;73(4):678-88.


Calmodulin kinase II inhibitor regulates calcium homeostasis changes caused by acute β-adrenergic receptor agonist stimulation in mouse ventricular myocytes.

Huang Y, Liu T, Wang D, Wang X, Li R, Chen Y, Tang Y, Wang T, Huang C.

In Vitro Cell Dev Biol Anim. 2016 Feb;52(2):156-62. doi: 10.1007/s11626-015-9967-y.


Ahnak1 is a tuneable modulator of cardiac Ca(v)1.2 calcium channel activity.

Pankonien I, Alvarez JL, Doller A, Köhncke C, Rotte D, Regitz-Zagrosek V, Morano I, Haase H.

J Muscle Res Cell Motil. 2011 Dec;32(4-5):281-90. doi: 10.1007/s10974-011-9269-2.


CaMKII inhibition in heart failure, beneficial, harmful, or both.

Cheng J, Xu L, Lai D, Guilbert A, Lim HJ, Keskanokwong T, Wang Y.

Am J Physiol Heart Circ Physiol. 2012 Apr 1;302(7):H1454-65. doi: 10.1152/ajpheart.00812.2011.


Blunted cardiac beta-adrenergic response as an early indication of cardiac dysfunction in Duchenne muscular dystrophy.

Li Y, Zhang S, Zhang X, Li J, Ai X, Zhang L, Yu D, Ge S, Peng Y, Chen X.

Cardiovasc Res. 2014 Jul 1;103(1):60-71. doi: 10.1093/cvr/cvu119.


Phospholemman and beta-adrenergic stimulation in the heart.

Wang J, Gao E, Song J, Zhang XQ, Li J, Koch WJ, Tucker AL, Philipson KD, Chan TO, Feldman AM, Cheung JY.

Am J Physiol Heart Circ Physiol. 2010 Mar;298(3):H807-15. doi: 10.1152/ajpheart.00877.2009.


Esmolol cardioplegia: the cellular mechanism of diastolic arrest.

Fallouh HB, Bardswell SC, McLatchie LM, Shattock MJ, Chambers DJ, Kentish JC.

Cardiovasc Res. 2010 Aug 1;87(3):552-60. doi: 10.1093/cvr/cvq058.


Basal and β-adrenergic regulation of the cardiac calcium channel CaV1.2 requires phosphorylation of serine 1700.

Fu Y, Westenbroek RE, Scheuer T, Catterall WA.

Proc Natl Acad Sci U S A. 2014 Nov 18;111(46):16598-603. doi: 10.1073/pnas.1419129111.

Items per page

Supplemental Content

Support Center