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Items: 1 to 20 of 90

1.

Lack of phospholipase A2 receptor increases susceptibility to cardiac rupture after myocardial infarction.

Mishina H, Watanabe K, Tamaru S, Watanabe Y, Fujioka D, Takahashi S, Suzuki K, Nakamura T, Obata JE, Kawabata K, Yokota Y, Inoue O, Murakami M, Hanasaki K, Kugiyama K.

Circ Res. 2014 Jan 31;114(3):493-504. doi: 10.1161/CIRCRESAHA.114.302319. Epub 2013 Dec 4.

2.

Deficiency of phospholipase A2 receptor exacerbates ovalbumin-induced lung inflammation.

Tamaru S, Mishina H, Watanabe Y, Watanabe K, Fujioka D, Takahashi S, Suzuki K, Nakamura T, Obata JE, Kawabata K, Yokota Y, Murakami M, Hanasaki K, Kugiyama K.

J Immunol. 2013 Aug 1;191(3):1021-8. doi: 10.4049/jimmunol.1300738. Epub 2013 Jul 1.

3.

Deficiency in TIMP-3 increases cardiac rupture and mortality post-myocardial infarction via EGFR signaling: beneficial effects of cetuximab.

Hammoud L, Lu X, Lei M, Feng Q.

Basic Res Cardiol. 2011 May;106(3):459-71. doi: 10.1007/s00395-010-0147-7. Epub 2011 Jan 18.

PMID:
21243368
4.

[Functional analysis of phospholipase A2 receptor by gene knockout studies].

Yokota Y.

Yakugaku Zasshi. 2001 Jan;121(1):23-33. Review. Japanese.

5.

sPLA2 IB induces human podocyte apoptosis via the M-type phospholipase A2 receptor.

Pan Y, Wan J, Liu Y, Yang Q, Liang W, Singhal PC, Saleem MA, Ding G.

Sci Rep. 2014 Oct 22;4:6660. doi: 10.1038/srep06660.

6.

Interleukin-23 deficiency leads to impaired wound healing and adverse prognosis after myocardial infarction.

Savvatis K, Pappritz K, Becher PM, Lindner D, Zietsch C, Volk HD, Westermann D, Schultheiss HP, Tschöpe C.

Circ Heart Fail. 2014 Jan;7(1):161-71. doi: 10.1161/CIRCHEARTFAILURE.113.000604. Epub 2013 Dec 3.

7.

Phospholipase A2 receptor: a regulator of biological functions of secretory phospholipase A2.

Hanasaki K, Arita H.

Prostaglandins Other Lipid Mediat. 2002 Aug;68-69:71-82. Review.

PMID:
12432910
8.

Excessive tumor necrosis factor activation after infarction contributes to susceptibility of myocardial rupture and left ventricular dysfunction.

Sun M, Dawood F, Wen WH, Chen M, Dixon I, Kirshenbaum LA, Liu PP.

Circulation. 2004 Nov 16;110(20):3221-8. Epub 2004 Nov 8.

9.

Mouse group X secretory phospholipase A2 induces a potent release of arachidonic acid from spleen cells and acts as a ligand for the phospholipase A2 receptor.

Morioka Y, Saiga A, Yokota Y, Suzuki N, Ikeda M, Ono T, Nakano K, Fujii N, Ishizaki J, Arita H, Hanasaki K.

Arch Biochem Biophys. 2000 Sep 1;381(1):31-42.

PMID:
11019817
10.

Higher levels of collagen and facilitated healing protect against ventricular rupture following myocardial infarction.

Fang L, Gao XM, Samuel CS, Su Y, Lim YL, Dart AM, Du XJ.

Clin Sci (Lond). 2008 Aug;115(3):99-106. doi: 10.1042/CS20070365.

PMID:
18248324
11.

Breeding Strategy Determines Rupture Incidence in Post-Infarct Healing WARPing Cardiovascular Research.

Deckx S, Carai P, Bateman J, Heymans S, Papageorgiou AP.

PLoS One. 2015 Sep 25;10(9):e0139199. doi: 10.1371/journal.pone.0139199. eCollection 2015.

12.

Targeted deletion of class A macrophage scavenger receptor increases the risk of cardiac rupture after experimental myocardial infarction.

Tsujita K, Kaikita K, Hayasaki T, Honda T, Kobayashi H, Sakashita N, Suzuki H, Kodama T, Ogawa H, Takeya M.

Circulation. 2007 Apr 10;115(14):1904-11. Epub 2007 Mar 26.

13.

Activation of CD4+ T lymphocytes improves wound healing and survival after experimental myocardial infarction in mice.

Hofmann U, Beyersdorf N, Weirather J, Podolskaya A, Bauersachs J, Ertl G, Kerkau T, Frantz S.

Circulation. 2012 Apr 3;125(13):1652-63. doi: 10.1161/CIRCULATIONAHA.111.044164. Epub 2012 Mar 2.

14.

Suppression of murine endotoxic shock by sPLA2 inhibitor, indoxam, through group IIA sPLA2-independent mechanisms.

Yokota Y, Hanasaki K, Ono T, Nakazato H, Kobayashi T, Arita H.

Biochim Biophys Acta. 1999 May 18;1438(2):213-22.

PMID:
10320804
15.

C-type lectin-like domain and fibronectin-like type II domain of phospholipase A(2) receptor 1 modulate binding and migratory responses to collagen.

Takahashi S, Watanabe K, Watanabe Y, Fujioka D, Nakamura T, Nakamura K, Obata JE, Kugiyama K.

FEBS Lett. 2015 Mar 24;589(7):829-35. doi: 10.1016/j.febslet.2015.02.016. Epub 2015 Feb 25.

16.

Mammalian phospholipase A2: phospholipase A2 receptor.

Hanasaki K.

Biol Pharm Bull. 2004 Aug;27(8):1165-7.

17.

Akt-dependent Girdin phosphorylation regulates repair processes after acute myocardial infarction.

Hayano S, Takefuji M, Maeda K, Noda T, Ichimiya H, Kobayashi K, Enomoto A, Asai N, Takahashi M, Murohara T.

J Mol Cell Cardiol. 2015 Nov;88:55-63. doi: 10.1016/j.yjmcc.2015.09.012. Epub 2015 Sep 21.

PMID:
26393439
18.

Resistance to endotoxic shock in phospholipase A2 receptor-deficient mice.

Hanasaki K, Yokota Y, Ishizaki J, Itoh T, Arita H.

J Biol Chem. 1997 Dec 26;272(52):32792-7.

19.

Bone marrow-derived myofibroblasts contribute functionally to scar formation after myocardial infarction.

van Amerongen MJ, Bou-Gharios G, Popa E, van Ark J, Petersen AH, van Dam GM, van Luyn MJ, Harmsen MC.

J Pathol. 2008 Feb;214(3):377-86.

PMID:
18095257
20.

Lack of haptoglobin results in unbalanced VEGFα/angiopoietin-1 expression, intramural hemorrhage and impaired wound healing after myocardial infarction.

Arslan F, Smeets MB, Buttari B, Profumo E, Riganò R, Akeroyd L, Kara E, Timmers L, Sluijter JP, van Middelaar B, den Ouden K, Pasterkamp G, Lim SK, de Kleijn DP.

J Mol Cell Cardiol. 2013 Mar;56:116-28. doi: 10.1016/j.yjmcc.2012.12.012. Epub 2012 Dec 26.

PMID:
23274064

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