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Items: 1 to 20 of 107


Effects of dipeptidyl peptidase-4 inhibition in an animal model of experimental asthma: a matter of dose, route, and time.

Stephan M, Suhling H, Schade J, Wittlake M, Tasic T, Klemann C, Pabst R, Jurawitz MC, Raber KA, Hoymann HG, Braun A, Glaab T, Hoffmann T, Schmiedl A, von Hörsten S.

Physiol Rep. 2013 Oct;1(5):e00095. doi: 10.1002/phy2.95. Epub 2013 Oct 2.


Dipeptidyl peptidase IV (DPP4) deficiency increases Th1-driven allergic contact dermatitis.

Tasic T, Bäumer W, Schmiedl A, Schwichtenhövel F, Pabst R, Raap U, von Hörsten S, Stephan M.

Clin Exp Allergy. 2011 Aug;41(8):1098-107. doi: 10.1111/j.1365-2222.2011.03778.x. Epub 2011 Jun 14.


Dose-dependent recruitment of CD25+ and CD26+ T cells in a novel F344 rat model of asthma.

Skripuletz T, Schmiedl A, Schade J, Bedoui S, Glaab T, Pabst R, von Hörsten S, Stephan M.

Am J Physiol Lung Cell Mol Physiol. 2007 Jun;292(6):L1564-71. Epub 2007 Mar 9.


DPP4 inhibitors increase differentially the expression of surfactant proteins in Fischer 344 rats.

Schmiedl A, Grützner D, Hoffmann T, von Hörsten S, Stephan M.

Acta Physiol (Oxf). 2014 Nov;212(3):248-61. doi: 10.1111/apha.12350. Epub 2014 Sep 15.


Reduced airway inflammation in CD26/DPP4-deficient F344 rats is associated with altered recruitment patterns of regulatory T cells and expression of pulmonary surfactant proteins.

Schmiedl A, Krainski J, Schwichtenhövel F, Schade J, Klemann C, Raber KA, Zscheppang K, Beekmann T, Acevedo C, Glaab T, Wedekind D, Pabst R, von Hörsten S, Stephan M.

Clin Exp Allergy. 2010 Dec;40(12):1794-808. doi: 10.1111/j.1365-2222.2010.03547.x.


Effects of concentrated ambient particles and diesel engine exhaust on allergic airway disease in Brown Norway rats.

Harkema JR, Wagner JG, Kaminski NE, Morishita M, Keeler GJ, McDonald JD, Barrett EG; HEI Health Review Committee.

Res Rep Health Eff Inst. 2009 Nov;(145):5-55.


CD26 (dipeptidyl-peptidase IV)-dependent recruitment of T cells in a rat asthma model.

Kruschinski C, Skripuletz T, Bedoui S, Tschernig T, Pabst R, Nassenstein C, Braun A, von Hörsten S.

Clin Exp Immunol. 2005 Jan;139(1):17-24. Erratum in: Clin Exp Immunol. 2005 Apr;140(1):192.


[Protective effects of ketamine on allergen-induced airway inflammatory injure and high airway reactivity in asthma: experiment with rats].

Zhu MM, Qian YN, Zhu W, Xu YM, Rong HB, Ding ZN, Fu CZ.

Zhonghua Yi Xue Za Zhi. 2007 May 22;87(19):1308-13. Chinese.


Differential OVA-induced pulmonary inflammation and unspecific reaction in Dark Agouti (DA) rats contingent on CD26/DPPIV deficiency.

Tasic T, Stephan M, von Hörsten S, Pabst R, Schmiedl A.

Immunobiology. 2014 Nov;219(11):888-900. doi: 10.1016/j.imbio.2014.07.007. Epub 2014 Jul 22.


Functional inhibition of PAR2 alleviates allergen-induced airway hyperresponsiveness and inflammation.

Asaduzzaman M, Nadeem A, Arizmendi N, Davidson C, Nichols HL, Abel M, Ionescu LI, Puttagunta L, Thebaud B, Gordon J, DeFea K, Hollenberg MD, Vliagoftis H.

Clin Exp Allergy. 2015 Dec;45(12):1844-55. doi: 10.1111/cea.12628.


CD26/dipeptidyl peptidase 4-deficiency alters thymic emigration patterns and leukcocyte subsets in F344-rats age-dependently.

Klemann C, Schade J, Pabst R, Leitner S, Stiller J, von Hörsten S, Stephan M.

Clin Exp Immunol. 2009 Feb;155(2):357-65. doi: 10.1111/j.1365-2249.2008.03839.x. Epub 2008 Dec 1.


Effects of concentrated ambient particles on normal and hypersecretory airways in rats.

Harkema JR, Keeler G, Wagner J, Morishita M, Timm E, Hotchkiss J, Marsik F, Dvonch T, Kaminski N, Barr E.

Res Rep Health Eff Inst. 2004 Aug;(120):1-68; discussion 69-79.


Dipeptidyl peptidase-4 is highly expressed in bronchial epithelial cells of untreated asthma and it increases cell proliferation along with fibronectin production in airway constitutive cells.

Shiobara T, Chibana K, Watanabe T, Arai R, Horigane Y, Nakamura Y, Hayashi Y, Shimizu Y, Takemasa A, Ishii Y.

Respir Res. 2016 Mar 14;17:28. doi: 10.1186/s12931-016-0342-7.


Engineered silica nanoparticles act as adjuvants to enhance allergic airway disease in mice.

Brandenberger C, Rowley NL, Jackson-Humbles DN, Zhang Q, Bramble LA, Lewandowski RP, Wagner JG, Chen W, Kaplan BL, Kaminski NE, Baker GL, Worden RM, Harkema JR.

Part Fibre Toxicol. 2013 Jul 1;10:26. doi: 10.1186/1743-8977-10-26.


Inhibition of neutrophil elastase attenuates airway hyperresponsiveness and inflammation in a mouse model of secondary allergen challenge: neutrophil elastase inhibition attenuates allergic airway responses.

Koga H, Miyahara N, Fuchimoto Y, Ikeda G, Waseda K, Ono K, Tanimoto Y, Kataoka M, Gelfand EW, Tanimoto M, Kanehiro A.

Respir Res. 2013 Jan 24;14:8. doi: 10.1186/1465-9921-14-8.


Role of IgE in the development of allergic airway inflammation and airway hyperresponsiveness--a murine model.

Hamelmann E, Tadeda K, Oshiba A, Gelfand EW.

Allergy. 1999 Apr;54(4):297-305. Review.


Spleen tyrosine kinase inhibition attenuates airway hyperresponsiveness and pollution-induced enhanced airway response in a chronic mouse model of asthma.

Penton PC, Wang X, Amatullah H, Cooper J, Godri K, North ML, Khanna N, Scott JA, Chow CW.

J Allergy Clin Immunol. 2013 Feb;131(2):512-20.e1-10. doi: 10.1016/j.jaci.2012.07.039. Epub 2012 Sep 13.


Inhibition of Pim1 kinase activation attenuates allergen-induced airway hyperresponsiveness and inflammation.

Shin YS, Takeda K, Shiraishi Y, Jia Y, Wang M, Jackson L, Wright AD, Carter L, Robinson J, Hicken E, Gelfand EW.

Am J Respir Cell Mol Biol. 2012 Apr;46(4):488-97. doi: 10.1165/rcmb.2011-0190OC. Epub 2011 Nov 10.


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