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Items: 1 to 20 of 107

1.

Sleep duration does not mediate or modify association of common genetic variants with type 2 diabetes.

Tare A, Lane JM, Cade BE, Grant SF, Chen TH, Punjabi NM, Lauderdale DS, Zee PC, Gharib SA, Gottlieb DJ, Scheer FA, Redline S, Saxena R.

Diabetologia. 2014 Feb;57(2):339-46. doi: 10.1007/s00125-013-3110-y. Epub 2013 Nov 27.

2.

Gene-Environment Interactions of Circadian-Related Genes for Cardiometabolic Traits.

Dashti HS, Follis JL, Smith CE, Tanaka T, Garaulet M, Gottlieb DJ, Hruby A, Jacques PF, Kiefte-de Jong JC, Lamon-Fava S, Scheer FA, Bartz TM, Kovanen L, Wojczynski MK, Frazier-Wood AC, Ahluwalia TS, Perälä MM, Jonsson A, Muka T, Kalafati IP, Mikkilä V, Ordovás JM; CHARGE Nutrition Study Group.

Diabetes Care. 2015 Aug;38(8):1456-66. doi: 10.2337/dc14-2709. Epub 2015 Jun 17. Erratum in: Diabetes Care. 2017 Aug 25;:.

3.

Association of sleep duration with type 2 diabetes and impaired glucose tolerance.

Chaput JP, Després JP, Bouchard C, Tremblay A.

Diabetologia. 2007 Nov;50(11):2298-304. Epub 2007 Aug 24.

PMID:
17717644
4.

Multiple genetic variants explain measurable variance in type 2 diabetes-related traits in Pakistanis.

Islam M, Jafar TH, Wood AR, De Silva NM, Caulfield M, Chaturvedi N, Frayling TM.

Diabetologia. 2012 Aug;55(8):2193-204. doi: 10.1007/s00125-012-2560-y. Epub 2012 Apr 28.

PMID:
22538361
5.

Habitual sleep duration is associated with BMI and macronutrient intake and may be modified by CLOCK genetic variants.

Dashti HS, Follis JL, Smith CE, Tanaka T, Cade BE, Gottlieb DJ, Hruby A, Jacques PF, Lamon-Fava S, Richardson K, Saxena R, Scheer FA, Kovanen L, Bartz TM, Perälä MM, Jonsson A, Frazier-Wood AC, Kalafati IP, Mikkilä V, Partonen T, Lemaitre RN, Lahti J, Hernandez DG, Toft U, Johnson WC, Kanoni S, Raitakari OT, Perola M, Psaty BM, Ferrucci L, Grarup N, Highland HM, Rallidis L, Kähönen M, Havulinna AS, Siscovick DS, Räikkönen K, Jørgensen T, Rotter JI, Deloukas P, Viikari JS, Mozaffarian D, Linneberg A, Seppälä I, Hansen T, Salomaa V, Gharib SA, Eriksson JG, Bandinelli S, Pedersen O, Rich SS, Dedoussis G, Lehtimäki T, Ordovás JM.

Am J Clin Nutr. 2015 Jan;101(1):135-43. doi: 10.3945/ajcn.114.095026. Epub 2014 Nov 26.

6.

Common genetic determinants of glucose homeostasis in healthy children: the European Youth Heart Study.

Kelliny C, Ekelund U, Andersen LB, Brage S, Loos RJ, Wareham NJ, Langenberg C.

Diabetes. 2009 Dec;58(12):2939-45. doi: 10.2337/db09-0374. Epub 2009 Sep 9.

7.

Impaired pancreatic beta cell compensatory function is the main cause of type 2 diabetes in individuals with high genetic risk: a 9 year prospective cohort study in the Chinese population.

Yan J, Peng D, Jiang F, Zhang R, Chen M, Wang T, Yan D, Wang S, Hou X, Bao Y, Hu C, Jia W.

Diabetologia. 2016 Jul;59(7):1458-1462. doi: 10.1007/s00125-016-3939-y. Epub 2016 Mar 23.

PMID:
27008621
8.

Previously associated type 2 diabetes variants may interact with physical activity to modify the risk of impaired glucose regulation and type 2 diabetes: a study of 16,003 Swedish adults.

Brito EC, Lyssenko V, Renström F, Berglund G, Nilsson PM, Groop L, Franks PW.

Diabetes. 2009 Jun;58(6):1411-8. doi: 10.2337/db08-1623. Epub 2009 Mar 26.

9.

Contribution of 24 obesity-associated genetic variants to insulin resistance, pancreatic beta-cell function and type 2 diabetes risk in the French population.

Robiou-du-Pont S, Bonnefond A, Yengo L, Vaillant E, Lobbens S, Durand E, Weill J, Lantieri O, Balkau B, Charpentier G, Marre M, Froguel P, Meyre D.

Int J Obes (Lond). 2013 Jul;37(7):980-5. doi: 10.1038/ijo.2012.175. Epub 2012 Oct 23.

PMID:
23090577
10.

Type 2 diabetes-related genetic risk scores associated with variations in fasting plasma glucose and development of impaired glucose homeostasis in the prospective DESIR study.

Vaxillaire M, Yengo L, Lobbens S, Rocheleau G, Eury E, Lantieri O, Marre M, Balkau B, Bonnefond A, Froguel P.

Diabetologia. 2014 Aug;57(8):1601-10. doi: 10.1007/s00125-014-3277-x. Epub 2014 Jun 4.

PMID:
24893864
11.

Association Between Sleep Timing, Obesity, Diabetes: The Hispanic Community Health Study/Study of Latinos (HCHS/SOL) Cohort Study.

Knutson KL, Wu D, Patel SR, Loredo JS, Redline S, Cai J, Gallo LC, Mossavar-Rahmani Y, Ramos AR, Teng Y, Daviglus ML, Zee PC.

Sleep. 2017 Apr 1;40(4). doi: 10.1093/sleep/zsx014.

PMID:
28329091
12.

Genetic variants at CDC123/CAMK1D and SPRY2 are associated with susceptibility to type 2 diabetes in the Japanese population.

Imamura M, Iwata M, Maegawa H, Watada H, Hirose H, Tanaka Y, Tobe K, Kaku K, Kashiwagi A, Kawamori R, Nakamura Y, Maeda S.

Diabetologia. 2011 Dec;54(12):3071-7. doi: 10.1007/s00125-011-2293-3. Epub 2011 Sep 10.

PMID:
21909839
13.

Variants of ADRA2A are associated with fasting glucose, blood pressure, body mass index and type 2 diabetes risk: meta-analysis of four prospective studies.

Talmud PJ, Cooper JA, Gaunt T, Holmes MV, Shah S, Palmen J, Drenos F, Shah T, Kumari M, Kivimaki M, Whittaker J, Lawlor DA, Day IN, Hingorani AD, Casas JP, Humphries SE.

Diabetologia. 2011 Jul;54(7):1710-9. doi: 10.1007/s00125-011-2108-6. Epub 2011 Apr 1.

14.

Genetic risk score of 46 type 2 diabetes risk variants associates with changes in plasma glucose and estimates of pancreatic β-cell function over 5 years of follow-up.

Andersson EA, Allin KH, Sandholt CH, Borglykke A, Lau CJ, Ribel-Madsen R, Sparsø T, Justesen JM, Harder MN, Jørgensen ME, Jørgensen T, Hansen T, Pedersen O.

Diabetes. 2013 Oct;62(10):3610-7. doi: 10.2337/db13-0362. Epub 2013 Jul 8.

15.

Variants from GIPR, TCF7L2, DGKB, MADD, CRY2, GLIS3, PROX1, SLC30A8 and IGF1 are associated with glucose metabolism in the Chinese.

Hu C, Zhang R, Wang C, Wang J, Ma X, Hou X, Lu J, Yu W, Jiang F, Bao Y, Xiang K, Jia W.

PLoS One. 2010 Nov 17;5(11):e15542. doi: 10.1371/journal.pone.0015542.

16.

Investigation of type 2 diabetes risk alleles support CDKN2A/B, CDKAL1, and TCF7L2 as susceptibility genes in a Han Chinese cohort.

Wen J, Rönn T, Olsson A, Yang Z, Lu B, Du Y, Groop L, Ling C, Hu R.

PLoS One. 2010 Feb 10;5(2):e9153. doi: 10.1371/journal.pone.0009153.

17.

The p53 codon 72 (Arg72Pro) polymorphism is associated with the degree of insulin resistance in type 2 diabetic subjects: a cross-sectional study.

Bonfigli AR, Sirolla C, Testa R, Cucchi M, Spazzafumo L, Salvioli S, Ceriello A, Olivieri F, Festa R, Procopio AD, Brandoni G, Boemi M, Marra M, Franceschi C.

Acta Diabetol. 2013 Jun;50(3):429-36. doi: 10.1007/s00592-012-0450-x. Epub 2012 Dec 27.

PMID:
23269546
18.

Effects of 16 genetic variants on fasting glucose and type 2 diabetes in South Asians: ADCY5 and GLIS3 variants may predispose to type 2 diabetes.

Rees SD, Hydrie MZ, O'Hare JP, Kumar S, Shera AS, Basit A, Barnett AH, Kelly MA.

PLoS One. 2011;6(9):e24710. doi: 10.1371/journal.pone.0024710. Epub 2011 Sep 20.

19.

A common variant in MTNR1B, encoding melatonin receptor 1B, is associated with type 2 diabetes and fasting plasma glucose in Han Chinese individuals.

Rönn T, Wen J, Yang Z, Lu B, Du Y, Groop L, Hu R, Ling C.

Diabetologia. 2009 May;52(5):830-3. doi: 10.1007/s00125-009-1297-8. Epub 2009 Feb 25.

PMID:
19241057
20.

The rs553668 polymorphism of the ADRA2A gene predicts the worsening of fasting glucose values in a cohort of subjects without diabetes. A population-based study.

Bo S, Cassader M, Cavallo-Perin P, Durazzo M, Rosato R, Gambino R.

Diabet Med. 2012 Apr;29(4):549-52. doi: 10.1111/j.1464-5491.2011.03522.x.

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