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Items: 1 to 20 of 295

1.

Traumatic brain injury precipitates cognitive impairment and extracellular Aβ aggregation in Alzheimer's disease transgenic mice.

Tajiri N, Kellogg SL, Shimizu T, Arendash GW, Borlongan CV.

PLoS One. 2013 Nov 4;8(11):e78851. doi: 10.1371/journal.pone.0078851. eCollection 2013.

2.

Soluble Aβ levels correlate with cognitive deficits in the 12-month-old APPswe/PS1dE9 mouse model of Alzheimer's disease.

Zhang W, Hao J, Liu R, Zhang Z, Lei G, Su C, Miao J, Li Z.

Behav Brain Res. 2011 Sep 23;222(2):342-50. doi: 10.1016/j.bbr.2011.03.072. Epub 2011 Apr 14.

PMID:
21513747
3.

Traumatic Brain Injury Increases the Expression of Nos1, Aβ Clearance, and Epileptogenesis in APP/PS1 Mouse Model of Alzheimer's Disease.

Miszczuk D, Dębski KJ, Tanila H, Lukasiuk K, Pitkänen A.

Mol Neurobiol. 2016 Dec;53(10):7010-7027. Epub 2015 Dec 15.

PMID:
26671618
4.

Electroacupuncture decreases cognitive impairment and promotes neurogenesis in the APP/PS1 transgenic mice.

Li X, Guo F, Zhang Q, Huo T, Liu L, Wei H, Xiong L, Wang Q.

BMC Complement Altern Med. 2014 Jan 22;14:37. doi: 10.1186/1472-6882-14-37.

5.

Traumatic brain injury accelerates amyloid-β deposition and impairs spatial learning in the triple-transgenic mouse model of Alzheimer's disease.

Shishido H, Kishimoto Y, Kawai N, Toyota Y, Ueno M, Kubota T, Kirino Y, Tamiya T.

Neurosci Lett. 2016 Aug 26;629:62-67. doi: 10.1016/j.neulet.2016.06.066. Epub 2016 Jun 29.

PMID:
27373531
6.

L-3-n-butylphthalide improves cognitive impairment and reduces amyloid-beta in a transgenic model of Alzheimer's disease.

Peng Y, Sun J, Hon S, Nylander AN, Xia W, Feng Y, Wang X, Lemere CA.

J Neurosci. 2010 Jun 16;30(24):8180-9. doi: 10.1523/JNEUROSCI.0340-10.2010.

7.

Repetitive mild brain trauma accelerates Abeta deposition, lipid peroxidation, and cognitive impairment in a transgenic mouse model of Alzheimer amyloidosis.

Uryu K, Laurer H, McIntosh T, Praticò D, Martinez D, Leight S, Lee VM, Trojanowski JQ.

J Neurosci. 2002 Jan 15;22(2):446-54.

8.

The effect of focal brain injury on beta-amyloid plaque deposition, inflammation and synapses in the APP/PS1 mouse model of Alzheimer's disease.

Collins JM, King AE, Woodhouse A, Kirkcaldie MT, Vickers JC.

Exp Neurol. 2015 May;267:219-29. doi: 10.1016/j.expneurol.2015.02.034. Epub 2015 Mar 4.

PMID:
25747037
9.

Lithium reduces BACE1 overexpression, β amyloid accumulation, and spatial learning deficits in mice with traumatic brain injury.

Yu F, Zhang Y, Chuang DM.

J Neurotrauma. 2012 Sep;29(13):2342-51. doi: 10.1089/neu.2012.2449. Epub 2012 Jul 2.

10.

Effects of accelerated senescence on learning and memory, locomotion and anxiety-like behavior in APP/PS1 mouse model of Alzheimer's disease.

Lok K, Zhao H, Zhang C, He N, Shen H, Wang Z, Zhao W, Yin M.

J Neurol Sci. 2013 Dec 15;335(1-2):145-54. doi: 10.1016/j.jns.2013.09.018. Epub 2013 Sep 21.

PMID:
24095271
11.

Self-assembling nanofibers alter the processing of amyloid precursor protein in a transgenic mouse model of Alzheimer's disease.

Yang H, Yang H, Xie Z, Wang P, Bi J.

Neurol Res. 2015 Jan;37(1):84-91. doi: 10.1179/1743132814Y.0000000417. Epub 2014 Jul 9.

PMID:
25005263
12.

Gender difference in valproic acid-induced neuroprotective effects on APP/PS1 double transgenic mice modeling Alzheimer's disease.

Long Z, Zeng Q, Wang K, Sharma A, He G.

Acta Biochim Biophys Sin (Shanghai). 2016 Oct;48(10):930-938. Epub 2016 Sep 10.

PMID:
27614317
13.

[Effects of huannao yicong recipe extract on the learning and memory and related factors of Abeta generation in the brain of APP transgenic mice].

Li H, Liu MF, Liu JG, Liu LT, Guan J, Cai LL, Hu J, Wei Y.

Zhongguo Zhong Xi Yi Jie He Za Zhi. 2013 Jan;33(1):90-4. Chinese.

PMID:
23596795
14.

Learning impairment by minimal cortical injury in a mouse model of Alzheimer's disease.

Zou J, Wang M, Uchiumi O, Shui Y, Ishigaki Y, Liu X, Tajima N, Akai T, Iizuka H, Kato N.

Brain Res. 2016 Apr 15;1637:56-63. doi: 10.1016/j.brainres.2016.02.017. Epub 2016 Feb 11.

PMID:
26876740
15.

Treadmill exercise enhances synaptic plasticity, but does not alter β-amyloid deposition in hippocampi of aged APP/PS1 transgenic mice.

Zhao G, Liu HL, Zhang H, Tong XJ.

Neuroscience. 2015 Jul 9;298:357-66. doi: 10.1016/j.neuroscience.2015.04.038. Epub 2015 Apr 23.

PMID:
25917310
16.

Overexpression of Ubiquilin-1 Alleviates Alzheimer's Disease-Caused Cognitive and Motor Deficits and Reduces Amyloid-β Accumulation in Mice.

Adegoke OO, Qiao F, Liu Y, Longley K, Feng S, Wang H.

J Alzheimers Dis. 2017;59(2):575-590. doi: 10.3233/JAD-170173.

17.

Mitochondrial amyloid-beta levels are associated with the extent of mitochondrial dysfunction in different brain regions and the degree of cognitive impairment in Alzheimer's transgenic mice.

Dragicevic N, Mamcarz M, Zhu Y, Buzzeo R, Tan J, Arendash GW, Bradshaw PC.

J Alzheimers Dis. 2010;20 Suppl 2:S535-50. doi: 10.3233/JAD-2010-100342.

PMID:
20463404
18.

Colivelin Ameliorates Impairments in Cognitive Behaviors and Synaptic Plasticity in APP/PS1 Transgenic Mice.

Wu M, Shi H, He Y, Yuan L, Qu X, Zhang J, Wang Z, Cai H, Qi J.

J Alzheimers Dis. 2017;59(3):1067-1078. doi: 10.3233/JAD-170307.

PMID:
28731445
19.

Apolipoprotein E4 influences amyloid deposition but not cell loss after traumatic brain injury in a mouse model of Alzheimer's disease.

Hartman RE, Laurer H, Longhi L, Bales KR, Paul SM, McIntosh TK, Holtzman DM.

J Neurosci. 2002 Dec 1;22(23):10083-7.

20.

Conditional inactivation of presenilin 1 prevents amyloid accumulation and temporarily rescues contextual and spatial working memory impairments in amyloid precursor protein transgenic mice.

Saura CA, Chen G, Malkani S, Choi SY, Takahashi RH, Zhang D, Gouras GK, Kirkwood A, Morris RG, Shen J.

J Neurosci. 2005 Jul 20;25(29):6755-64.

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