Similar articles for PMID: 24141050

Year	Number of Results
2000	2
2001	2
2002	3
2003	1
2005	5
2006	7
2007	6
2008	3
2009	8
2010	7
2011	8
2012	5
2013	18
2014	11
2015	8
2016	5
2017	3
2018	7
2019	2
2020	2
2021	3
2022	1
2023	3
2024	2

1

UCP2 overexpression worsens mitochondrial dysfunction and accelerates disease progression in a mouse model of amyotrophic lateral sclerosis.

Peixoto PM, Kim HJ, Sider B, Starkov A, Horvath TL, Manfredi G. Peixoto PM, et al. Mol Cell Neurosci. 2013 Nov;57:104-10. doi: 10.1016/j.mcn.2013.10.002. Epub 2013 Oct 17. Mol Cell Neurosci. 2013. PMID: 24141050 Free PMC article.

2

Xiao Y, Karam C, Yi J, Zhang L, Li X, Yoon D, Wang H, Dhakal K, Ramlow P, Yu T, Mo Z, Ma J, Zhou J. Xiao Y, et al. Pharmacol Res. 2018 Dec;138:25-36. doi: 10.1016/j.phrs.2018.09.008. Epub 2018 Sep 18. Pharmacol Res. 2018. PMID: 30236524 Free PMC article.

3

Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.

Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC. Jaarsma D, et al. Neurobiol Dis. 2000 Dec;7(6 Pt B):623-43. doi: 10.1006/nbdi.2000.0299. Neurobiol Dis. 2000. PMID: 11114261

4

Increased mitochondrial antioxidative activity or decreased oxygen free radical propagation prevent mutant SOD1-mediated motor neuron cell death and increase amyotrophic lateral sclerosis-like transgenic mouse survival.

Liu R, Li B, Flanagan SW, Oberley LW, Gozal D, Qiu M. Liu R, et al. J Neurochem. 2002 Feb;80(3):488-500. doi: 10.1046/j.0022-3042.2001.00720.x. J Neurochem. 2002. PMID: 11905995 Free article.

5

Progressive impairment of CaV1.1 function in the skeletal muscle of mice expressing a mutant type 1 Cu/Zn superoxide dismutase (G93A) linked to amyotrophic lateral sclerosis.

Beqollari D, Romberg CF, Dobrowolny G, Martini M, Voss AA, Musarò A, Bannister RA. Beqollari D, et al. Skelet Muscle. 2016 Jun 23;6:24. doi: 10.1186/s13395-016-0094-6. eCollection 2016. Skelet Muscle. 2016. PMID: 27340545 Free PMC article.

6

Progressive Mitochondrial SOD1^G93A Accumulation Causes Severe Structural, Metabolic and Functional Aberrations through OPA1 Down-Regulation in a Mouse Model of Amyotrophic Lateral Sclerosis.

Méndez-López I, Sancho-Bielsa FJ, Engel T, García AG, Padín JF. Méndez-López I, et al. Int J Mol Sci. 2021 Jul 30;22(15):8194. doi: 10.3390/ijms22158194. Int J Mol Sci. 2021. PMID: 34360957 Free PMC article.

7

Amyotrophic lateral sclerosis-associated mutant SOD1 inhibits anterograde axonal transport of mitochondria by reducing Miro1 levels.

Moller A, Bauer CS, Cohen RN, Webster CP, De Vos KJ. Moller A, et al. Hum Mol Genet. 2017 Dec 1;26(23):4668-4679. doi: 10.1093/hmg/ddx348. Hum Mol Genet. 2017. PMID: 28973175 Free PMC article.

8

Cytochrome c association with the inner mitochondrial membrane is impaired in the CNS of G93A-SOD1 mice.

Kirkinezos IG, Bacman SR, Hernandez D, Oca-Cossio J, Arias LJ, Perez-Pinzon MA, Bradley WG, Moraes CT. Kirkinezos IG, et al. J Neurosci. 2005 Jan 5;25(1):164-72. doi: 10.1523/JNEUROSCI.3829-04.2005. J Neurosci. 2005. PMID: 15634778 Free PMC article.

9

Mitochondria-targeted catalase reverts the neurotoxicity of hSOD1G⁹³A astrocytes without extending the survival of ALS-linked mutant hSOD1 mice.

Pehar M, Beeson G, Beeson CC, Johnson JA, Vargas MR. Pehar M, et al. PLoS One. 2014 Jul 23;9(7):e103438. doi: 10.1371/journal.pone.0103438. eCollection 2014. PLoS One. 2014. PMID: 25054289 Free PMC article.

10

The mitochondrial calcium regulator cyclophilin D is an essential component of oestrogen-mediated neuroprotection in amyotrophic lateral sclerosis.

Kim HJ, Magranè J, Starkov AA, Manfredi G. Kim HJ, et al. Brain. 2012 Sep;135(Pt 9):2865-74. doi: 10.1093/brain/aws208. Brain. 2012. PMID: 22961554 Free PMC article.

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