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Items: 1 to 20 of 98

1.
2.

A possible role for inflammation in mediating apoptosis of oligodendrocytes as induced by the Lyme disease spirochete Borrelia burgdorferi.

Ramesh G, Benge S, Pahar B, Philipp MT.

J Neuroinflammation. 2012 Apr 23;9:72. doi: 10.1186/1742-2094-9-72.

3.
4.

The Lyme disease spirochete Borrelia burgdorferi induces inflammation and apoptosis in cells from dorsal root ganglia.

Ramesh G, Santana-Gould L, Inglis FM, England JD, Philipp MT.

J Neuroinflammation. 2013 Jul 18;10:88. doi: 10.1186/1742-2094-10-88.

5.

Interaction of the Lyme disease spirochete Borrelia burgdorferi with brain parenchyma elicits inflammatory mediators from glial cells as well as glial and neuronal apoptosis.

Ramesh G, Borda JT, Dufour J, Kaushal D, Ramamoorthy R, Lackner AA, Philipp MT.

Am J Pathol. 2008 Nov;173(5):1415-27. doi: 10.2353/ajpath.2008.080483. Epub 2008 Oct 2.

6.

Non-viable Borrelia burgdorferi induce inflammatory mediators and apoptosis in human oligodendrocytes.

Parthasarathy G, Fevrier HB, Philipp MT.

Neurosci Lett. 2013 Nov 27;556:200-3. doi: 10.1016/j.neulet.2013.10.032. Epub 2013 Oct 22.

7.

Effects of dexamethasone and meloxicam on Borrelia burgdorferi-induced inflammation in glial and neuronal cells of the central nervous system.

Ramesh G, Martinez AN, Martin DS, Philipp MT.

J Neuroinflammation. 2017 Feb 2;14(1):28. doi: 10.1186/s12974-017-0806-9.

9.

Persisting atypical and cystic forms of Borrelia burgdorferi and local inflammation in Lyme neuroborreliosis.

Miklossy J, Kasas S, Zurn AD, McCall S, Yu S, McGeer PL.

J Neuroinflammation. 2008 Sep 25;5:40. doi: 10.1186/1742-2094-5-40.

10.

Microglia are mediators of Borrelia burgdorferi-induced apoptosis in SH-SY5Y neuronal cells.

Myers TA, Kaushal D, Philipp MT.

PLoS Pathog. 2009 Nov;5(11):e1000659. doi: 10.1371/journal.ppat.1000659. Epub 2009 Nov 13.

11.

Antagonist of the neurokinin-1 receptor curbs neuroinflammation in ex vivo and in vitro models of Lyme neuroborreliosis.

Martinez AN, Ramesh G, Jacobs MB, Philipp MT.

J Neuroinflammation. 2015 Dec 30;12:243. doi: 10.1186/s12974-015-0453-y.

12.

Possible role of glial cells in the onset and progression of Lyme neuroborreliosis.

Ramesh G, Borda JT, Gill A, Ribka EP, Morici LA, Mottram P, Martin DS, Jacobs MB, Didier PJ, Philipp MT.

J Neuroinflammation. 2009 Aug 25;6:23. doi: 10.1186/1742-2094-6-23.

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14.

Borrelia burgdorferi basic membrane protein A could induce chemokine production in murine microglia cell line BV2.

Zhao H, Liu A, Cui Y, Liang Z, Li B, Bao F.

Microb Pathog. 2017 Oct;111:174-181. doi: 10.1016/j.micpath.2017.08.036. Epub 2017 Sep 1.

PMID:
28867633
15.

Borrelia burgdorferi induces inflammatory mediator production by murine microglia.

Rasley A, Anguita J, Marriott I.

J Neuroimmunol. 2002 Sep;130(1-2):22-31.

PMID:
12225885
16.

Toll-like receptors: insights into their possible role in the pathogenesis of lyme neuroborreliosis.

Bernardino AL, Myers TA, Alvarez X, Hasegawa A, Philipp MT.

Infect Immun. 2008 Oct;76(10):4385-95. doi: 10.1128/IAI.00394-08. Epub 2008 Aug 11.

17.

Inflammation in the pathogenesis of lyme neuroborreliosis.

Ramesh G, Didier PJ, England JD, Santana-Gould L, Doyle-Meyers LA, Martin DS, Jacobs MB, Philipp MT.

Am J Pathol. 2015 May;185(5):1344-60. doi: 10.1016/j.ajpath.2015.01.024. Epub 2015 Apr 16.

18.

Expression of Fas receptor on human T lymphocytes under stimulation with Borrelia burgdorferi sensu lato - preliminary results.

Grygorczuk S, Osada J, Swierzbińska R, Zajkowska J, Kondrusik M, Pancewicz S, Dąbrowska M.

Adv Med Sci. 2010;55(2):228-34. doi: 10.2478/v10039-010-0025-4.

PMID:
20639186
20.

Aprepitant limits in vivo neuroinflammatory responses in a rhesus model of Lyme neuroborreliosis.

Martinez AN, Burmeister AR, Ramesh G, Doyle-Meyers L, Marriott I, Philipp MT.

J Neuroinflammation. 2017 Feb 15;14(1):37. doi: 10.1186/s12974-017-0813-x.

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