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Items: 1 to 20 of 142

1.

High-density lipoproteins augment hypoxia-induced angiogenesis via regulation of post-translational modulation of hypoxia-inducible factor 1α.

Tan JT, Prosser HC, Vanags LZ, Monger SA, Ng MK, Bursill CA.

FASEB J. 2014 Jan;28(1):206-17. doi: 10.1096/fj.13-233874. Epub 2013 Sep 10.

2.

High-Density Lipoproteins Rescue Diabetes-Impaired Angiogenesis via Scavenger Receptor Class B Type I.

Tan JT, Prosser HC, Dunn LL, Vanags LZ, Ridiandries A, Tsatralis T, Leece L, Clayton ZE, Yuen SC, Robertson S, Lam YT, Celermajer DS, Ng MK, Bursill CA.

Diabetes. 2016 Oct;65(10):3091-103. doi: 10.2337/db15-1668. Epub 2016 Jun 9. Erratum in: Diabetes. 2017 Feb 13;:. Diabetes. 2017 Apr;66(4):1097.

3.

Hypoxia-inducible factors in the first trimester human lung.

Groenman F, Rutter M, Caniggia I, Tibboel D, Post M.

J Histochem Cytochem. 2007 Apr;55(4):355-63. Epub 2006 Dec 22.

PMID:
17189520
4.
6.

Siah: new players in the cellular response to hypoxia.

Nakayama K, Ronai Z.

Cell Cycle. 2004 Nov;3(11):1345-7. Epub 2004 Nov 6.

PMID:
15492505
8.

Ubiquitin ligase Siah1 promotes the migration and invasion of human glioma cells by regulating HIF-1α signaling under hypoxia.

Shi H, Zheng B, Wu Y, Tang Y, Wang L, Gao Y, Gong H, Du J, Yu R.

Oncol Rep. 2015 Mar;33(3):1185-90. doi: 10.3892/or.2014.3695. Epub 2014 Dec 23.

PMID:
25572001
9.

Multifunctional regulation of angiogenesis by high-density lipoproteins.

Prosser HC, Tan JT, Dunn LL, Patel S, Vanags LZ, Bao S, Ng MK, Bursill CA.

Cardiovasc Res. 2014 Jan 1;101(1):145-54. doi: 10.1093/cvr/cvt234. Epub 2013 Oct 15.

PMID:
24130189
10.

Prolyl hydroxylase domain 2 deficiency promotes skeletal muscle fiber-type transition via a calcineurin/NFATc1-dependent pathway.

Shin J, Nunomiya A, Kitajima Y, Dan T, Miyata T, Nagatomi R.

Skelet Muscle. 2016 Mar 5;6:5. doi: 10.1186/s13395-016-0079-5. eCollection 2016.

11.

The prolyl hydroxylase enzymes are positively associated with hypoxia-inducible factor-1α and vascular endothelial growth factor in human breast cancer and alter in response to primary systemic treatment with epirubicin and tamoxifen.

Fox SB, Generali D, Berruti A, Brizzi MP, Campo L, Bonardi S, Bersiga A, Allevi G, Milani M, Aguggini S, Mele T, Dogliotti L, Bottini A, Harris AL.

Breast Cancer Res. 2011 Feb 3;13(1):R16. doi: 10.1186/bcr2825.

12.
13.

Expression of prolyl hydroxylases (PHDs) is selectively controlled by HIF-1 and HIF-2 proteins in nucleus pulposus cells of the intervertebral disc: distinct roles of PHD2 and PHD3 proteins in controlling HIF-1α activity in hypoxia.

Fujita N, Markova D, Anderson DG, Chiba K, Toyama Y, Shapiro IM, Risbud MV.

J Biol Chem. 2012 May 11;287(20):16975-86. doi: 10.1074/jbc.M111.334466. Epub 2012 Mar 26.

14.

Regulation of HIF prolyl hydroxylases by hypoxia-inducible factors.

Aprelikova O, Chandramouli GV, Wood M, Vasselli JR, Riss J, Maranchie JK, Linehan WM, Barrett JC.

J Cell Biochem. 2004 Jun 1;92(3):491-501. Erratum in: J Cell Biochem. 2004 Oct 15;93(3):639.

PMID:
15156561
15.

Fasudil-induced hypoxia-inducible factor-1alpha degradation disrupts a hypoxia-driven vascular endothelial growth factor autocrine mechanism in endothelial cells.

Takata K, Morishige K, Takahashi T, Hashimoto K, Tsutsumi S, Yin L, Ohta T, Kawagoe J, Takahashi K, Kurachi H.

Mol Cancer Ther. 2008 Jun;7(6):1551-61. doi: 10.1158/1535-7163.MCT-07-0428.

16.

Gambogic acid inhibits angiogenesis through inhibiting PHD2-VHL-HIF-1α pathway.

Lu N, Hui H, Yang H, Zhao K, Chen Y, You QD, Guo QL.

Eur J Pharm Sci. 2013 May 13;49(2):220-6. doi: 10.1016/j.ejps.2013.02.018. Epub 2013 Mar 15.

PMID:
23501055
17.

Endothelin-1 inhibits prolyl hydroxylase domain 2 to activate hypoxia-inducible factor-1alpha in melanoma cells.

Spinella F, Rosanò L, Del Duca M, Di Castro V, Nicotra MR, Natali PG, Bagnato A.

PLoS One. 2010 Jun 21;5(6):e11241. doi: 10.1371/journal.pone.0011241.

18.

SIAH ubiquitin ligases regulate breast cancer cell migration and invasion independent of the oxygen status.

Adam MG, Matt S, Christian S, Hess-Stumpp H, Haegebarth A, Hofmann TG, Algire C.

Cell Cycle. 2015;14(23):3734-47. doi: 10.1080/15384101.2015.1104441.

19.

Carbon monoxide promotes VEGF expression by increasing HIF-1alpha protein level via two distinct mechanisms, translational activation and stabilization of HIF-1alpha protein.

Choi YK, Kim CK, Lee H, Jeoung D, Ha KS, Kwon YG, Kim KW, Kim YM.

J Biol Chem. 2010 Oct 15;285(42):32116-25. doi: 10.1074/jbc.M110.131284. Epub 2010 Aug 19.

20.

HIF prolyl-hydroxylase 2 is the key oxygen sensor setting low steady-state levels of HIF-1alpha in normoxia.

Berra E, Benizri E, Ginouvès A, Volmat V, Roux D, Pouysségur J.

EMBO J. 2003 Aug 15;22(16):4082-90.

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