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Items: 1 to 20 of 121

1.

Tenc1-deficient mice develop glomerular disease in a strain-specific manner.

Uchio-Yamada K, Sawada K, Tamura K, Katayama S, Monobe Y, Yamamoto Y, Ogura A, Manabe N.

Nephron Exp Nephrol. 2013;123(3-4):22-33. doi: 10.1159/000354058. Epub 2013 Aug 23.

PMID:
23988887
2.

Tensin2-deficient mice on FVB/N background develop severe glomerular disease.

Uchio-Yamada K, Monobe Y, Akagi K, Yamamoto Y, Ogura A, Manabe N.

J Vet Med Sci. 2016 Jun 1;78(5):811-8. doi: 10.1292/jvms.15-0442. Epub 2016 Feb 5.

3.

Deletion of CD151 results in a strain-dependent glomerular disease due to severe alterations of the glomerular basement membrane.

Baleato RM, Guthrie PL, Gubler MC, Ashman LK, Roselli S.

Am J Pathol. 2008 Oct;173(4):927-37. doi: 10.2353/ajpath.2008.071149. Epub 2008 Sep 11.

4.
5.

Forced expression of laminin beta1 in podocytes prevents nephrotic syndrome in mice lacking laminin beta2, a model for Pierson syndrome.

Suh JH, Jarad G, VanDeVoorde RG, Miner JH.

Proc Natl Acad Sci U S A. 2011 Sep 13;108(37):15348-53. doi: 10.1073/pnas.1108269108. Epub 2011 Aug 29.

6.

Deficiency of the tensin2 gene in the ICGN mouse: an animal model for congenital nephrotic syndrome.

Cho AR, Uchio-Yamada K, Torigai T, Miyamoto T, Miyoshi I, Matsuda J, Kurosawa T, Kon Y, Asano A, Sasaki N, Agui T.

Mamm Genome. 2006 May;17(5):407-16.

PMID:
16688531
7.

Neurexin-1, a presynaptic adhesion molecule, localizes at the slit diaphragm of the glomerular podocytes in kidneys.

Saito A, Miyauchi N, Hashimoto T, Karasawa T, Han GD, Kayaba M, Sumi T, Tomita M, Ikezumi Y, Suzuki K, Koitabashi Y, Shimizu F, Kawachi H.

Am J Physiol Regul Integr Comp Physiol. 2011 Feb;300(2):R340-8. doi: 10.1152/ajpregu.00640.2009. Epub 2010 Nov 3.

8.

Genetic background-dependent diversity in renal failure caused by the tensin2 gene deficiency in the mouse.

Sasaki H, Marusugi K, Kimura J, Kitamura H, Nagasaki K, Torigoe D, Agui T, Sasaki N.

Biomed Res. 2015;36(5):323-30. doi: 10.2220/biomedres.36.323.

9.

Immunoexpression of podocyte-associated proteins in acquired human glomerulopathies with nephrotic syndrome.

Wagrowska-Danilewicz M, Stasikowska O, Danilewicz M.

Pol J Pathol. 2006;57(1):17-21.

PMID:
16739878
10.

Upregulated expression of integrin α1 in mesangial cells and integrin α3 and vimentin in podocytes of Col4a3-null (Alport) mice.

Steenhard BM, Vanacore R, Friedman D, Zelenchuk A, Stroganova L, Isom K, St John PL, Hudson BG, Abrahamson DR.

PLoS One. 2012;7(12):e50745. doi: 10.1371/journal.pone.0050745. Epub 2012 Dec 7.

11.

Decreases in podocin, CD2-associated protein (CD2AP) and tensin2 may be involved in albuminuria during septic acute renal failure.

Kato T, Mizuno-Horikawa Y, Mizuno S.

J Vet Med Sci. 2011 Dec;73(12):1579-84. Epub 2011 Jul 29.

12.
13.

Gitelman syndrome and glomerular proteinuria: a link between loss of sodium-chloride cotransporter and podocyte dysfunction?

Demoulin N, Aydin S, Cosyns JP, Dahan K, Cornet G, Auberger I, Loffing J, Devuyst O.

Nephrol Dial Transplant. 2014 Sep;29 Suppl 4:iv117-20. doi: 10.1093/ndt/gfu075.

PMID:
25165177
14.

Integrin beta1-mediated matrix assembly and signaling are critical for the normal development and function of the kidney glomerulus.

Kanasaki K, Kanda Y, Palmsten K, Tanjore H, Lee SB, Lebleu VS, Gattone VH Jr, Kalluri R.

Dev Biol. 2008 Jan 15;313(2):584-93. Epub 2007 Nov 12.

15.

Laminin compensation in collagen alpha3(IV) knockout (Alport) glomeruli contributes to permeability defects.

Abrahamson DR, Isom K, Roach E, Stroganova L, Zelenchuk A, Miner JH, St John PL.

J Am Soc Nephrol. 2007 Sep;18(9):2465-72. Epub 2007 Aug 15.

16.

Expression of agrin, dystroglycan, and utrophin in normal renal tissue and in experimental glomerulopathies.

Raats CJ, van den Born J, Bakker MA, Oppers-Walgreen B, Pisa BJ, Dijkman HB, Assmann KJ, Berden JH.

Am J Pathol. 2000 May;156(5):1749-65.

17.

Localization of extracellular matrix receptors in ICGN mice, a strain of mice with hereditary nephrotic syndrome.

Uchio-Yamada K, Manabe N, Yamaguchi M, Akashi N, Goto Y, Yamamoto Y, Ogura A, Miyamoto H.

J Vet Med Sci. 2001 Nov;63(11):1171-8.

18.

Light microscopic features of membranous nephropathy with unusual changes of the podocytes and glomerular basement membrane in a patient with sudden onset of nephrotic syndrome.

Nagayama Y, Morita H, Kawashima E, Yoshimura A.

Am J Kidney Dis. 2010 May;55(5):962-6. doi: 10.1053/j.ajkd.2009.11.018. No abstract available.

PMID:
20138416
19.

Role of the podocyte (and glomerular endothelium) in building the GBM.

Abrahamson DR.

Semin Nephrol. 2012 Jul;32(4):342-9. doi: 10.1016/j.semnephrol.2012.06.005. Review.

20.

Abnormalities of extracellular matrices and transforming growth factor beta1 localization in the kidney of the hereditary nephrotic mice (ICGN strain).

Uchio K, Manabe N, Kinoshita A, Tamura K, Miyamoto M, Ogura A, Yamamoto Y, Miyamoto H.

J Vet Med Sci. 1999 Jul;61(7):769-76.

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