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Items: 1 to 20 of 79

1.

Chronic noise exposure and Alzheimer disease: Is there an etiological association?

Cui B, Li K.

Med Hypotheses. 2013 Oct;81(4):623-6. doi: 10.1016/j.mehy.2013.07.017. Epub 2013 Jul 27.

PMID:
23899629
2.

Alzheimer's disease.

De-Paula VJ, Radanovic M, Diniz BS, Forlenza OV.

Subcell Biochem. 2012;65:329-52. doi: 10.1007/978-94-007-5416-4_14. Review.

PMID:
23225010
3.

[Alzheimer disease: cellular and molecular aspects].

Octave JN.

Bull Mem Acad R Med Belg. 2005;160(10-12):445-9; discussion 450-1. French.

PMID:
16768248
4.

Alzheimer's disease and amyloid: culprit or coincidence?

Skaper SD.

Int Rev Neurobiol. 2012;102:277-316. doi: 10.1016/B978-0-12-386986-9.00011-9. Review.

PMID:
22748834
5.

Involvement of WAVE accumulation in Abeta/APP pathology-dependent tangle modification in Alzheimer's disease.

Takata K, Kitamura Y, Nakata Y, Matsuoka Y, Tomimoto H, Taniguchi T, Shimohama S.

Am J Pathol. 2009 Jul;175(1):17-24. doi: 10.2353/ajpath.2009.080908. Epub 2009 Jun 4.

6.

A{beta} accelerates the spatiotemporal progression of tau pathology and augments tau amyloidosis in an Alzheimer mouse model.

Hurtado DE, Molina-Porcel L, Iba M, Aboagye AK, Paul SM, Trojanowski JQ, Lee VM.

Am J Pathol. 2010 Oct;177(4):1977-88. doi: 10.2353/ajpath.2010.100346. Epub 2010 Aug 27.

7.

Roles of glycogen synthase kinase 3 in Alzheimer's disease.

Cai Z, Zhao Y, Zhao B.

Curr Alzheimer Res. 2012 Sep;9(7):864-79. Review.

PMID:
22272620
8.

Non-tau based neuronal degeneration in Alzheimer's disease -- an immunocytochemical and quantitative study in the supragranular layers of the middle temporal neocortex.

van de Nes JA, Nafe R, Schlote W.

Brain Res. 2008 Jun 5;1213:152-65. doi: 10.1016/j.brainres.2008.03.043. Epub 2008 Apr 1.

PMID:
18455153
10.

Neurofibrillary tangles and tau phosphorylation.

Brion JP, Anderton BH, Authelet M, Dayanandan R, Leroy K, Lovestone S, Octave JN, Pradier L, Touchet N, Tremp G.

Biochem Soc Symp. 2001;(67):81-8.

PMID:
11447842
11.

A critical analysis of the 'amyloid cascade hypothesis'.

Armstrong RA.

Folia Neuropathol. 2014;52(3):211-25. Review.

12.

Abnormal interaction of VDAC1 with amyloid beta and phosphorylated tau causes mitochondrial dysfunction in Alzheimer's disease.

Manczak M, Reddy PH.

Hum Mol Genet. 2012 Dec 1;21(23):5131-46. doi: 10.1093/hmg/dds360. Epub 2012 Aug 27.

13.

Alpha1-antichymotrypsin, an inflammatory protein overexpressed in Alzheimer's disease brain, induces tau phosphorylation in neurons.

Padmanabhan J, Levy M, Dickson DW, Potter H.

Brain. 2006 Nov;129(Pt 11):3020-34. Epub 2006 Sep 20.

PMID:
16987932
14.

Beta-amyloid, neuronal death and Alzheimer's disease.

Carter J, Lippa CF.

Curr Mol Med. 2001 Dec;1(6):733-7. Review.

PMID:
11899259
15.

[Pathological changes induced by amyloid-β in Alzheimer's disease].

Takata K, Kitamura Y, Taniguchi T.

Yakugaku Zasshi. 2011 Jan;131(1):3-11. Review. Japanese.

16.

Chronic Noise Exposure Acts Cumulatively to Exacerbate Alzheimer's Disease-Like Amyloid-β Pathology and Neuroinflammation in the Rat Hippocampus.

Cui B, Li K, Gai Z, She X, Zhang N, Xu C, Chen X, An G, Ma Q, Wang R.

Sci Rep. 2015 Aug 7;5:12943. doi: 10.1038/srep12943.

18.

Untangling amyloid-β, tau, and metals in Alzheimer's disease.

Savelieff MG, Lee S, Liu Y, Lim MH.

ACS Chem Biol. 2013 May 17;8(5):856-65. doi: 10.1021/cb400080f. Epub 2013 Mar 18. Review.

PMID:
23506614
19.

Reduced Reelin expression accelerates amyloid-beta plaque formation and tau pathology in transgenic Alzheimer's disease mice.

Kocherhans S, Madhusudan A, Doehner J, Breu KS, Nitsch RM, Fritschy JM, Knuesel I.

J Neurosci. 2010 Jul 7;30(27):9228-40. doi: 10.1523/JNEUROSCI.0418-10.2010.

20.

Connective tissue growth factor (CTGF) expression in the brain is a downstream effector of insulin resistance- associated promotion of Alzheimer's disease beta-amyloid neuropathology.

Zhao Z, Ho L, Wang J, Qin W, Festa ED, Mobbs C, Hof P, Rocher A, Masur S, Haroutunian V, Pasinetti GM.

FASEB J. 2005 Dec;19(14):2081-2. Epub 2005 Sep 26.

PMID:
16186174

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