Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 107

1.

Suppression of inflammatory gene expression in T cells by Porphyromonas gingivalis is mediated by targeting MAPK signaling.

Khalaf H, Demirel I, Bengtsson T.

Cell Mol Immunol. 2013 Sep;10(5):413-22. doi: 10.1038/cmi.2013.23. Epub 2013 Jul 29.

4.

Role of the phosphatidylinositol 3 kinase-Akt pathway in the regulation of IL-10 and IL-12 by Porphyromonas gingivalis lipopolysaccharide.

Martin M, Schifferle RE, Cuesta N, Vogel SN, Katz J, Michalek SM.

J Immunol. 2003 Jul 15;171(2):717-25.

5.

Porphyromonas gingivalis induces receptor activator of NF-kappaB ligand expression in osteoblasts through the activator protein 1 pathway.

Okahashi N, Inaba H, Nakagawa I, Yamamura T, Kuboniwa M, Nakayama K, Hamada S, Amano A.

Infect Immun. 2004 Mar;72(3):1706-14.

6.

Differential regulation of cytokine genes in gingival epithelial cells challenged by Fusobacterium nucleatum and Porphyromonas gingivalis.

Huang GT, Zhang HB, Dang HN, Haake SK.

Microb Pathog. 2004 Dec;37(6):303-12. Epub 2004 Dec 8.

PMID:
15619426
7.

Differential cytokine regulation by NF-kappaB and AP-1 in Jurkat T-cells.

Khalaf H, Jass J, Olsson PE.

BMC Immunol. 2010 May 27;11:26. doi: 10.1186/1471-2172-11-26.

8.

Transcriptional regulation of lysophosphatidic acid-induced interleukin-8 expression and secretion by p38 MAPK and JNK in human bronchial epithelial cells.

Saatian B, Zhao Y, He D, Georas SN, Watkins T, Spannhake EW, Natarajan V.

Biochem J. 2006 Feb 1;393(Pt 3):657-68.

9.
10.

Altered T-cell responses by the periodontal pathogen Porphyromonas gingivalis.

Khalaf H, Bengtsson T.

PLoS One. 2012;7(9):e45192. doi: 10.1371/journal.pone.0045192. Epub 2012 Sep 12.

11.
12.
13.

Hemoglobin receptor protein from Porphyromonas gingivalis induces interleukin-8 production in human gingival epithelial cells through stimulation of the mitogen-activated protein kinase and NF-κB signal transduction pathways.

Fujita Y, Nakayama M, Naito M, Yamachika E, Inoue T, Nakayama K, Iida S, Ohara N.

Infect Immun. 2014 Jan;82(1):202-11. doi: 10.1128/IAI.01140-12. Epub 2013 Oct 14.

14.

E-selectin expression induced by Porphyromonas gingivalis in human endothelial cells via nucleotide-binding oligomerization domain-like receptors and Toll-like receptors.

Wan M, Liu JR, Wu D, Chi XP, Ouyang XY.

Mol Oral Microbiol. 2015 Oct;30(5):399-410. doi: 10.1111/omi.12102. Epub 2015 Jun 24.

PMID:
25939768
16.

Porphorymonas gingivalis induces intracellular adhesion molecule-1 expression in endothelial cells through the nuclear factor-kappaB pathway, but not through the p38 MAPK pathway.

Zhang D, Zheng H, Zhao J, Lin L, Li C, Liu J, Pan Y.

J Periodontal Res. 2011 Feb;46(1):31-8. doi: 10.1111/j.1600-0765.2010.01305.x. Epub 2010 Sep 3.

PMID:
20825555
17.

Tormentic acid inhibits LPS-induced inflammatory response in human gingival fibroblasts via inhibition of TLR4-mediated NF-κB and MAPK signalling pathway.

Jian CX, Li MZ, Zheng WY, He Y, Ren Y, Wu ZM, Fan QS, Hu YH, Li CJ.

Arch Oral Biol. 2015 Sep;60(9):1327-32. doi: 10.1016/j.archoralbio.2015.05.005. Epub 2015 May 22.

PMID:
26123747
18.

ROCK activity affects IL-1-induced signaling possibly through MKK4 and p38 MAPK in Caco-2 cells.

Banerjee S, McGee DW.

In Vitro Cell Dev Biol Anim. 2016 Sep;52(8):878-84. doi: 10.1007/s11626-016-0050-0. Epub 2016 May 12.

PMID:
27173611
19.

Increasing maternal body mass index is associated with systemic inflammation in the mother and the activation of distinct placental inflammatory pathways.

Aye IL, Lager S, Ramirez VI, Gaccioli F, Dudley DJ, Jansson T, Powell TL.

Biol Reprod. 2014 Jun;90(6):129. doi: 10.1095/biolreprod.113.116186. Epub 2014 Apr 23.

20.

Endotoxin tolerance induced by lipopolysaccharides derived from Porphyromonas gingivalis and Escherichia coli: alternations in Toll-like receptor 2 and 4 signaling pathway.

Sun Y, Li H, Sun MJ, Zheng YY, Gong DJ, Xu Y.

Inflammation. 2014 Feb;37(1):268-76. doi: 10.1007/s10753-013-9737-5.

PMID:
24132662

Supplemental Content

Support Center