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Items: 1 to 20 of 128

1.

Combined effects of aging and in vitro non-steroid anti-inflammatory drugs on kidney and liver mitochondrial physiology.

Rocha-Rodrigues S, Santos-Alves E, Coxito PM, Marques-Aleixo I, Passos E, Guimarães JT, Martins MJ, Oliveira PJ, Magalhães J, Ascensão A.

Life Sci. 2013 Sep 3;93(8):329-37. doi: 10.1016/j.lfs.2013.07.004. Epub 2013 Jul 17.

PMID:
23872100
2.

In vitro salicylate does not further impair aging-induced brain mitochondrial dysfunction.

Marques-Aleixo I, Rocha-Rodrigues S, Santos-Alves E, Coxito PM, Passos E, Oliveira PJ, Magalhães J, Ascensão A.

Toxicology. 2012 Dec 8;302(1):51-9. doi: 10.1016/j.tox.2012.07.018. Epub 2012 Aug 8.

PMID:
22967791
3.

Exercise mitigates diclofenac-induced liver mitochondrial dysfunction.

Santos-Alves E, Marques-Aleixo I, Coxito P, Balça MM, Rizo-Roca D, Rocha-Rodrigues S, Martins S, Torrella JR, Oliveira PJ, Moreno AJ, Magalhães J, Ascensão A.

Eur J Clin Invest. 2014 Jul;44(7):668-77. doi: 10.1111/eci.12285.

PMID:
24889192
4.

Acute exercise protects against calcium-induced cardiac mitochondrial permeability transition pore opening in doxorubicin-treated rats.

Ascensão A, Lumini-Oliveira J, Machado NG, Ferreira RM, Gonçalves IO, Moreira AC, Marques F, Sardão VA, Oliveira PJ, Magalhães J.

Clin Sci (Lond). 2011 Jan;120(1):37-49. doi: 10.1042/CS20100254.

PMID:
20666733
5.

Effects of all-trans-retinoic acid on the permeability transition and bioenergetic functions of rat liver mitochondria in combination with endoxifen.

Ribeiro MP, Santos AE, Santos MS, Custódio JB.

Life Sci. 2013 Jul 30;93(2-3):96-107. doi: 10.1016/j.lfs.2013.05.027. Epub 2013 Jun 11.

PMID:
23764110
6.

Influence of nonsteroidal anti-inflammatory drugs on calcium efflux in isolated rat renal cortex mitochondria and aspects of the mechanisms involved.

Pigoso AA, Uyemura SA, Santos AC, Rodrigues T, Mingatto FE, Curti C.

Int J Biochem Cell Biol. 1998 Sep;30(9):961-5.

PMID:
9785459
7.

Unaltered hepatic oxidative phosphorylation and mitochondrial permeability transition in wistar rats treated with nimesulide: Relevance for nimesulide toxicity characterization.

Moreno AJ, Oliveira PJ, Nova CD, Alvaro AR, Moreira RA, Santos SM, Macedo T.

J Biochem Mol Toxicol. 2007;21(2):53-61.

PMID:
17427176
8.

Physical exercise improves brain cortex and cerebellum mitochondrial bioenergetics and alters apoptotic, dynamic and auto(mito)phagy markers.

Marques-Aleixo I, Santos-Alves E, Balça MM, Rizo-Roca D, Moreira PI, Oliveira PJ, Magalhães J, Ascensão A.

Neuroscience. 2015 Aug 20;301:480-95. doi: 10.1016/j.neuroscience.2015.06.027. Epub 2015 Jun 24.

PMID:
26116519
9.
10.

Endurance training reverts heart mitochondrial dysfunction, permeability transition and apoptotic signaling in long-term severe hyperglycemia.

Lumini-Oliveira J, Magalhães J, Pereira CV, Moreira AC, Oliveira PJ, Ascensão A.

Mitochondrion. 2011 Jan;11(1):54-63. doi: 10.1016/j.mito.2010.07.005. Epub 2010 Jul 21.

PMID:
20654738
11.

Role of mitochondrial permeability transition in diclofenac-induced hepatocyte injury in rats.

Masubuchi Y, Nakayama S, Horie T.

Hepatology. 2002 Mar;35(3):544-51.

PMID:
11870366
12.

Oxidative stress is responsible for mitochondrial permeability transition induction by salicylate in liver mitochondria.

Battaglia V, Salvi M, Toninello A.

J Biol Chem. 2005 Oct 7;280(40):33864-72. Epub 2005 Aug 12.

13.
14.

Diclofenac induced in vivo nephrotoxicity may involve oxidative stress-mediated massive genomic DNA fragmentation and apoptotic cell death.

Hickey EJ, Raje RR, Reid VE, Gross SM, Ray SD.

Free Radic Biol Med. 2001 Jul 15;31(2):139-52.

PMID:
11440826
15.

Endurance training and chronic intermittent hypoxia modulate in vitro salicylate-induced hepatic mitochondrial dysfunction.

Ascensão A, Gonçalves IO, Lumini-Oliveira J, Marques-Aleixo I, Dos Passos E, Rocha-Rodrigues S, Machado NG, Moreira AC, Oliveira PJ, Torrella JR, Magalhães J.

Mitochondrion. 2012 Nov;12(6):607-16. doi: 10.1016/j.mito.2012.10.007. Epub 2012 Oct 12.

PMID:
23069012
16.

Histological changes and impairment of liver mitochondrial bioenergetics after long-term treatment with alpha-naphthyl-isothiocyanate (ANIT).

Palmeira CM, Ferreira FM, Rolo AP, Oliveira PJ, Santos MS, Moreno AJ, Cipriano MA, Martins MI, Seiça R.

Toxicology. 2003 Aug 28;190(3):185-96.

PMID:
12927374
17.

Modulation of cardiac mitochondrial permeability transition and apoptotic signaling by endurance training and intermittent hypobaric hypoxia.

Magalhães J, Gonçalves IO, Lumini-Oliveira J, Marques-Aleixo I, Passos E, Rocha-Rodrigues S, Machado NG, Moreira AC, Rizo D, Viscor G, Oliveira PJ, Torrella JR, Ascensão A.

Int J Cardiol. 2014 Apr 15;173(1):40-5. doi: 10.1016/j.ijcard.2014.02.011. Epub 2014 Feb 20.

PMID:
24602319
18.

Diclofenac induces apoptosis in hepatocytes.

Gómez-Lechón MJ, Ponsoda X, O'Connor E, Donato T, Jover R, Castell JV.

Toxicol In Vitro. 2003 Oct-Dec;17(5-6):675-80.

PMID:
14599462
19.

Bax-mediated mitochondrial outer membrane permeabilization (MOMP), distinct from the mitochondrial permeability transition, is a key mechanism in diclofenac-induced hepatocyte injury: Multiple protective roles of cyclosporin A.

Siu WP, Pun PB, Latchoumycandane C, Boelsterli UA.

Toxicol Appl Pharmacol. 2008 Mar 15;227(3):451-61. doi: 10.1016/j.taap.2007.11.030. Epub 2007 Dec 14.

PMID:
18191430
20.

Diclofenac sodium and mefenamic acid: potent inducers of the membrane permeability transition in renal cortex mitochondria.

Uyemura SA, Santos AC, Mingatto FE, Jordani MC, Curti C.

Arch Biochem Biophys. 1997 Jun 15;342(2):231-5.

PMID:
9186483

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