Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 137

1.

Perturbed mononuclear phagocyte system in severely burned and septic patients.

Xiu F, Jeschke MG.

Shock. 2013 Aug;40(2):81-8. doi: 10.1097/SHK.0b013e318299f774. Review.

2.
3.

The effects and mechanisms of insulin on systemic inflammatory response and immune cells in severe trauma, burn injury, and sepsis.

Deng HP, Chai JK.

Int Immunopharmacol. 2009 Oct;9(11):1251-9. doi: 10.1016/j.intimp.2009.07.009. Epub 2009 Jul 30. Review.

PMID:
19647101
4.

High MafB expression following burn augments monocyte commitment and inhibits DC differentiation in hemopoietic progenitors.

Howell K, Posluszny J, He LK, Szilagyi A, Halerz J, Gamelli RL, Shankar R, Muthu K.

J Leukoc Biol. 2012 Jan;91(1):69-81. doi: 10.1189/jlb.0711338. Epub 2011 Oct 7.

5.

Host defence mechanisms: evaluation and roles of acquired defects and immunotherapy.

Meakins JL.

Can J Surg. 1975 May;18(3):259-68. Review.

PMID:
1092446
6.

What are the immunological alterations induced by burn injury?

Winkelstein A.

J Trauma. 1984 Sep;24(9 Suppl):S72-83. No abstract available.

PMID:
6332922
7.

Major injury leads to predominance of the T helper-2 lymphocyte phenotype and diminished interleukin-12 production associated with decreased resistance to infection.

O'Sullivan ST, Lederer JA, Horgan AF, Chin DH, Mannick JA, Rodrick ML.

Ann Surg. 1995 Oct;222(4):482-90; discussion 490-2.

8.

[Immunological alterations after extensive burn injury].

Kawakami M, Okada Y.

Nihon Geka Gakkai Zasshi. 1998 Jan;99(1):26-30. Review. Japanese.

PMID:
9547744
9.

Severity of burn injury and sepsis determines the cytokine responses of bone marrow progenitor-derived macrophages.

Cohen MJ, Carroll C, He LK, Muthu K, Gamelli RL, Jones SB, Shankar R.

J Trauma. 2007 Apr;62(4):858-67.

PMID:
17426540
10.

Circulating dendritic cells following burn.

D'Arpa N, Accardo-Palumbo A, Amato G, D'Amelio L, Pileri D, Cataldo V, Mogavero R, Lombardo C, Napoli B, Conte F.

Burns. 2009 Jun;35(4):513-8. doi: 10.1016/j.burns.2008.05.027. Epub 2009 Mar 6.

PMID:
19269101
11.

Th17 cells: critical mediators of host responses to burn injury and sepsis.

Rendon JL, Choudhry MA.

J Leukoc Biol. 2012 Sep;92(3):529-38. doi: 10.1189/jlb.0212083. Epub 2012 Jun 29. Review.

13.

Role of cellular events in the pathophysiology of sepsis.

Bhan C, Dipankar P, Chakraborty P, Sarangi PP.

Inflamm Res. 2016 Nov;65(11):853-868. Epub 2016 Jul 8. Review.

PMID:
27392441
15.

Differentiation and heterogeneity in the mononuclear phagocyte system.

Hume DA.

Mucosal Immunol. 2008 Nov;1(6):432-41. doi: 10.1038/mi.2008.36. Epub 2008 Aug 27. Review.

PMID:
19079210
16.

Major injury induces increased production of interleukin-10 by cells of the immune system with a negative impact on resistance to infection.

Lyons A, Kelly JL, Rodrick ML, Mannick JA, Lederer JA.

Ann Surg. 1997 Oct;226(4):450-8; discussion 458-60.

17.

Interaction between the innate and adaptive immune systems is required to survive sepsis and control inflammation after injury.

Shelley O, Murphy T, Paterson H, Mannick JA, Lederer JA.

Shock. 2003 Aug;20(2):123-9.

PMID:
12865655
18.

Antimicrobial peptides in burns and wounds.

Bhat S, Milner S.

Curr Protein Pept Sci. 2007 Oct;8(5):506-20. Review.

PMID:
17979765
19.

[Study on the relationship between the human leucocyte antigen-DR expression on CD14+ monocytes and sepsis].

Yang HM, Yu Y, Chai JK, Hu S, Lei SH, Zhang DQ, Tao BJ, Zhang GA, He LX, Yao YM.

Zhonghua Shao Shang Za Zhi. 2007 Aug;23(4):272-5. Chinese.

PMID:
18095551
20.

Prostaglandin E2 alterations during sepsis are partially mediated by endotoxin-induced inhibition of prostaglandin 15-hydroxydehydrogenase.

Hahn EL, Clancy KD, Tai HH, Ricken JD, He LK, Gamelli RL.

J Trauma. 1998 May;44(5):777-81; discussion 781-2.

PMID:
9603077

Supplemental Content

Support Center