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Items: 1 to 20 of 358

1.

Porphyromonas gingivalis peptidylarginine deiminase substrate specificity.

Abdullah SN, Farmer EA, Spargo L, Logan R, Gully N.

Anaerobe. 2013 Oct;23:102-8. doi: 10.1016/j.anaerobe.2013.07.001. Epub 2013 Jul 13.

PMID:
23856045
2.

Peptidylarginine deiminase from Porphyromonas gingivalis citrullinates human fibrinogen and α-enolase: implications for autoimmunity in rheumatoid arthritis.

Wegner N, Wait R, Sroka A, Eick S, Nguyen KA, Lundberg K, Kinloch A, Culshaw S, Potempa J, Venables PJ.

Arthritis Rheum. 2010 Sep;62(9):2662-72. doi: 10.1002/art.27552.

3.

Porphyromonas gingivalis may play an important role in the pathogenesis of periodontitis-associated rheumatoid arthritis.

Liao F, Li Z, Wang Y, Shi B, Gong Z, Cheng X.

Med Hypotheses. 2009 Jun;72(6):732-5. doi: 10.1016/j.mehy.2008.12.040. Epub 2009 Feb 25.

PMID:
19246161
4.

Effect of Porphyromonas gingivalis on citrullination of proteins by macrophages in vitro.

Marchant C, Smith MD, Proudman S, Haynes DR, Bartold PM.

J Periodontol. 2013 Sep;84(9):1272-80. doi: 10.1902/jop.2012.120103. Epub 2012 Oct 29.

PMID:
23106505
5.

Crystal structure of Porphyromonas gingivalis peptidylarginine deiminase: implications for autoimmunity in rheumatoid arthritis.

Montgomery AB, Kopec J, Shrestha L, Thezenas ML, Burgess-Brown NA, Fischer R, Yue WW, Venables PJ.

Ann Rheum Dis. 2016 Jun;75(6):1255-61. doi: 10.1136/annrheumdis-2015-207656. Epub 2015 Jul 24.

6.

The human peptidylarginine deiminases type 2 and type 4 have distinct substrate specificities.

Assohou-Luty C, Raijmakers R, Benckhuijsen WE, Stammen-Vogelzangs J, de Ru A, van Veelen PA, Franken KL, Drijfhout JW, Pruijn GJ.

Biochim Biophys Acta. 2014 Apr;1844(4):829-36. doi: 10.1016/j.bbapap.2014.02.019. Epub 2014 Mar 2.

PMID:
24594197
7.

Lessons to be learned from periodontitis.

Janssen KM, Vissink A, de Smit MJ, Westra J, Brouwer E.

Curr Opin Rheumatol. 2013 Mar;25(2):241-7. doi: 10.1097/BOR.0b013e32835d833d. Review.

PMID:
23370377
8.

Theoretical insights into the protonation states of active site cysteine and citrullination mechanism of Porphyromonas gingivalis peptidylarginine deiminase.

Zhao C, Ling B, Dong L, Liu Y.

Proteins. 2017 Aug;85(8):1518-1528. doi: 10.1002/prot.25313. Epub 2017 May 25.

PMID:
28486790
9.

Defining the role of Porphyromonas gingivalis peptidylarginine deiminase (PPAD) in rheumatoid arthritis through the study of PPAD biology.

Konig MF, Paracha AS, Moni M, Bingham CO 3rd, Andrade F.

Ann Rheum Dis. 2015 Nov;74(11):2054-61. doi: 10.1136/annrheumdis-2014-205385. Epub 2014 May 26.

10.

Rheumatoid arthritis-specific autoantibodies to peptidyl arginine deiminase type 4 inhibit citrullination of fibrinogen.

Auger I, Martin M, Balandraud N, Roudier J.

Arthritis Rheum. 2010 Jan;62(1):126-31. doi: 10.1002/art.27230.

11.

Peptidyl arginine deiminase from Porphyromonas gingivalis abolishes anaphylatoxin C5a activity.

Bielecka E, Scavenius C, Kantyka T, Jusko M, Mizgalska D, Szmigielski B, Potempa B, Enghild JJ, Prossnitz ER, Blom AM, Potempa J.

J Biol Chem. 2014 Nov 21;289(47):32481-7. doi: 10.1074/jbc.C114.617142. Epub 2014 Oct 16.

12.

Expression of peptidylarginine deiminase from Porphyromonas gingivalis in Escherichia coli: enzyme purification and characterization.

Rodríguez SB, Stitt BL, Ash DE.

Arch Biochem Biophys. 2009 Aug 1;488(1):14-22. doi: 10.1016/j.abb.2009.06.010. Epub 2009 Jun 21.

13.

Extracellular Proteome and Citrullinome of the Oral Pathogen Porphyromonas gingivalis.

Stobernack T, Glasner C, Junker S, Gabarrini G, de Smit M, de Jong A, Otto A, Becher D, van Winkelhoff AJ, van Dijl JM.

J Proteome Res. 2016 Dec 2;15(12):4532-4543. Epub 2016 Oct 19.

PMID:
27712078
14.

Citrullination in the periodontium--a possible link between periodontitis and rheumatoid arthritis.

Laugisch O, Wong A, Sroka A, Kantyka T, Koziel J, Neuhaus K, Sculean A, Venables PJ, Potempa J, Möller B, Eick S.

Clin Oral Investig. 2016 May;20(4):675-83. doi: 10.1007/s00784-015-1556-7. Epub 2015 Aug 13.

15.

Autoimmunity to specific citrullinated proteins gives the first clues to the etiology of rheumatoid arthritis.

Wegner N, Lundberg K, Kinloch A, Fisher B, Malmström V, Feldmann M, Venables PJ.

Immunol Rev. 2010 Jan;233(1):34-54. doi: 10.1111/j.0105-2896.2009.00850.x. Review.

PMID:
20192991
16.

Porphyromonas gingivalis facilitates the development and progression of destructive arthritis through its unique bacterial peptidylarginine deiminase (PAD).

Maresz KJ, Hellvard A, Sroka A, Adamowicz K, Bielecka E, Koziel J, Gawron K, Mizgalska D, Marcinska KA, Benedyk M, Pyrc K, Quirke AM, Jonsson R, Alzabin S, Venables PJ, Nguyen KA, Mydel P, Potempa J.

PLoS Pathog. 2013 Sep;9(9):e1003627. doi: 10.1371/journal.ppat.1003627. Epub 2013 Sep 12.

17.

The peptidylarginine deiminase gene is a conserved feature of Porphyromonas gingivalis.

Gabarrini G, de Smit M, Westra J, Brouwer E, Vissink A, Zhou K, Rossen JW, Stobernack T, van Dijl JM, van Winkelhoff AJ.

Sci Rep. 2015 Sep 25;5:13936. doi: 10.1038/srep13936.

18.
19.

Citrullination of synovial proteins in murine models of rheumatoid arthritis.

Vossenaar ER, Nijenhuis S, Helsen MM, van der Heijden A, Senshu T, van den Berg WB, van Venrooij WJ, Joosten LA.

Arthritis Rheum. 2003 Sep;48(9):2489-500.

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