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Items: 1 to 20 of 175

1.

M2 Kupffer cells promote M1 Kupffer cell apoptosis: a protective mechanism against alcoholic and nonalcoholic fatty liver disease.

Wan J, Benkdane M, Teixeira-Clerc F, Bonnafous S, Louvet A, Lafdil F, Pecker F, Tran A, Gual P, Mallat A, Lotersztajn S, Pavoine C.

Hepatology. 2014 Jan;59(1):130-42. doi: 10.1002/hep.26607.

PMID:
23832548
2.

Cannabinoid CB2 receptors protect against alcoholic liver disease by regulating Kupffer cell polarization in mice.

Louvet A, Teixeira-Clerc F, Chobert MN, Deveaux V, Pavoine C, Zimmer A, Pecker F, Mallat A, Lotersztajn S.

Hepatology. 2011 Oct;54(4):1217-26. doi: 10.1002/hep.24524.

PMID:
21735467
3.

M2 kupffer cells promote hepatocyte senescence: an IL-6-dependent protective mechanism against alcoholic liver disease.

Wan J, Benkdane M, Alons E, Lotersztajn S, Pavoine C.

Am J Pathol. 2014 Jun;184(6):1763-72. doi: 10.1016/j.ajpath.2014.02.014.

PMID:
24713392
4.

Bias in macrophage activation pattern influences non-alcoholic steatohepatitis (NASH) in mice.

Maina V, Sutti S, Locatelli I, Vidali M, Mombello C, Bozzola C, Albano E.

Clin Sci (Lond). 2012 Jun;122(11):545-53. doi: 10.1042/CS20110366.

PMID:
22142284
5.

Impact of PPAR-α induction on glucose homoeostasis in alcohol-fed mice.

Lebrun V, Molendi-Coste O, Lanthier N, Sempoux C, Cani PD, van Rooijen N, Stärkel P, Horsmans Y, Leclercq IA.

Clin Sci (Lond). 2013 Dec;125(11):501-11. doi: 10.1042/CS20130064.

PMID:
23758436
6.

[Activation of Kupffer cell and related signal pathway proteins in the liver of high fat and high fructose diet induced NAFLD mice].

Zhu ML, Liu J, Liu YL, Yang WJ, Luo Y, Zhuang ZJ, Jiao QB, Chen JY, Yan J, Bian DX, Ma XJ, Xun YH, Shi JP.

Zhonghua Shi Yan He Lin Chuang Bing Du Xue Za Zhi. 2013 Oct;27(5):325-7. Chinese.

PMID:
24645313
7.

Prevention and reversal of lipotoxicity-induced hepatic insulin resistance and steatohepatitis in mice by an antioxidant carotenoid, β-cryptoxanthin.

Ni Y, Nagashimada M, Zhan L, Nagata N, Kobori M, Sugiura M, Ogawa K, Kaneko S, Ota T.

Endocrinology. 2015 Mar;156(3):987-99. doi: 10.1210/en.2014-1776.

PMID:
25562616
8.

Depletion of liver Kupffer cells prevents the development of diet-induced hepatic steatosis and insulin resistance.

Huang W, Metlakunta A, Dedousis N, Zhang P, Sipula I, Dube JJ, Scott DK, O'Doherty RM.

Diabetes. 2010 Feb;59(2):347-57. doi: 10.2337/db09-0016.

9.

Chronic Alcohol Consumption Causes Liver Injury in High-Fructose-Fed Male Mice Through Enhanced Hepatic Inflammatory Response.

Song M, Chen T, Prough RA, Cave MC, McClain CJ.

Alcohol Clin Exp Res. 2016 Mar;40(3):518-28. doi: 10.1111/acer.12994.

PMID:
26858005
10.

Fat-laden macrophages modulate lobular inflammation in nonalcoholic steatohepatitis (NASH).

Jindal A, Bruzzì S, Sutti S, Locatelli I, Bozzola C, Paternostro C, Parola M, Albano E.

Exp Mol Pathol. 2015 Aug;99(1):155-62. doi: 10.1016/j.yexmp.2015.06.015.

PMID:
26112094
11.

Role of Kupffer cells in failure of fatty livers following liver transplantation and alcoholic liver injury.

Thurman RG, Gao W, Connor HD, Adachi Y, Stachlewitz RF, Zhong Z, Knecht KT, Bradford BU, Mason RP, Lemasters JJ.

J Gastroenterol Hepatol. 1995;10 Suppl 1:S24-30.

PMID:
8589336
12.

Impaired macrophage autophagy increases the immune response in obese mice by promoting proinflammatory macrophage polarization.

Liu K, Zhao E, Ilyas G, Lalazar G, Lin Y, Haseeb M, Tanaka KE, Czaja MJ.

Autophagy. 2015;11(2):271-84. doi: 10.1080/15548627.2015.1009787.

13.

Pro-inflammatory activated Kupffer cells by lipids induce hepatic NKT cells deficiency through activation-induced cell death.

Tang T, Sui Y, Lian M, Li Z, Hua J.

PLoS One. 2013 Dec 3;8(12):e81949. doi: 10.1371/journal.pone.0081949.

14.

Adipogenic changes of hepatocytes in a high-fat diet-induced fatty liver mice model and non-alcoholic fatty liver disease patients.

Pan X, Wang P, Luo J, Wang Z, Song Y, Ye J, Hou X.

Endocrine. 2015 Apr;48(3):834-47. doi: 10.1007/s12020-014-0384-x.

PMID:
25138963
15.

Super paramagnetic iron oxide MRI shows defective Kupffer cell uptake function in non-alcoholic fatty liver disease.

Asanuma T, Ono M, Kubota K, Hirose A, Hayashi Y, Saibara T, Inanami O, Ogawa Y, Enzan H, Onishi S, Kuwabara M, Oben JA.

Gut. 2010 Feb;59(2):258-66. doi: 10.1136/gut.2009.176651.

PMID:
19919948
16.

Arginase 2 deficiency results in spontaneous steatohepatitis: a novel link between innate immune activation and hepatic de novo lipogenesis.

Navarro LA, Wree A, Povero D, Berk MP, Eguchi A, Ghosh S, Papouchado BG, Erzurum SC, Feldstein AE.

J Hepatol. 2015 Feb;62(2):412-20. doi: 10.1016/j.jhep.2014.09.015.

17.

Cooperation of liver cells in health and disease.

Kmieć Z.

Adv Anat Embryol Cell Biol. 2001;161:III-XIII, 1-151. Review.

PMID:
11729749
18.

Pharmacological inhibition of adipocyte fatty acid binding protein alleviates both acute liver injury and non-alcoholic steatohepatitis in mice.

Hoo RL, Lee IP, Zhou M, Wong JY, Hui X, Xu A, Lam KS.

J Hepatol. 2013 Feb;58(2):358-64. doi: 10.1016/j.jhep.2012.10.022.

PMID:
23108115
19.

Activation of toll-like receptor 3 attenuates alcoholic liver injury by stimulating Kupffer cells and stellate cells to produce interleukin-10 in mice.

Byun JS, Suh YG, Yi HS, Lee YS, Jeong WI.

J Hepatol. 2013 Feb;58(2):342-9. doi: 10.1016/j.jhep.2012.09.016.

PMID:
23023014
20.

Depletion of Kupffer cells modulates ethanol-induced hepatocyte DNA synthesis in C57Bl/6 mice.

Owumi SE, Corthals SM, Uwaifo AO, Kamendulis LM, Klaunig JE.

Environ Toxicol. 2014 Aug;29(8):867-75. doi: 10.1002/tox.21814.

PMID:
22996800
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