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Items: 1 to 20 of 65

1.

A salty taste to autoimmunity.

van der Meer JW, Netea MG.

N Engl J Med. 2013 Jun 27;368(26):2520-1. doi: 10.1056/NEJMcibr1303292. No abstract available.

PMID:
23802520
2.

Autoimmunity: Rubbing salt in the wound.

O'Shea JJ, Jones RG.

Nature. 2013 Apr 25;496(7446):437-9. doi: 10.1038/nature11959. Epub 2013 Mar 6. No abstract available.

PMID:
23467087
3.

PD research round-up: Sodium sensing: link to (auto)immunity.

Devuyst O.

Perit Dial Int. 2013 Jul-Aug;33(4):348. doi: 10.3747/pdi.2013.00143. No abstract available.

4.

High sodium chloride consumption enhances the effects of smoking but does not interact with SGK1 polymorphisms in the development of ACPA-positive status in patients with RA.

Jiang X, Sundström B, Alfredsson L, Klareskog L, Rantapää-Dahlqvist S, Bengtsson C.

Ann Rheum Dis. 2016 May;75(5):943-6. doi: 10.1136/annrheumdis-2015-209009. Epub 2016 Feb 22. No abstract available.

PMID:
26903441
5.

Low dose CP-690,550 (tofacitinib), a pan-JAK inhibitor, accelerates the onset of experimental autoimmune encephalomyelitis by potentiating Th17 differentiation.

Yoshida H, Kimura A, Fukaya T, Sekiya T, Morita R, Shichita T, Inoue H, Yoshimura A.

Biochem Biophys Res Commun. 2012 Feb 10;418(2):234-40. doi: 10.1016/j.bbrc.2011.12.156. Epub 2012 Jan 9.

PMID:
22252297
6.

Estrogen receptor α signaling in T lymphocytes is required for estradiol-mediated inhibition of Th1 and Th17 cell differentiation and protection against experimental autoimmune encephalomyelitis.

Lélu K, Laffont S, Delpy L, Paulet PE, Périnat T, Tschanz SA, Pelletier L, Engelhardt B, Guéry JC.

J Immunol. 2011 Sep 1;187(5):2386-93. doi: 10.4049/jimmunol.1101578. Epub 2011 Aug 1.

7.

Soluble IL-2Rα (sCD25) exacerbates autoimmunity and enhances the development of Th17 responses in mice.

Russell SE, Moore AC, Fallon PG, Walsh PT.

PLoS One. 2012;7(10):e47748. doi: 10.1371/journal.pone.0047748. Epub 2012 Oct 15.

8.

The gap-junction inhibitor carbenoxolone suppresses the differentiation of Th17 cells through inhibition of IL-23 expression in antigen presenting cells.

Endong L, Shijie J, Sonobe Y, Di M, Hua L, Kawanokuchi J, Mizuno T, Suzumura A.

J Neuroimmunol. 2011 Dec 15;240-241:58-64. doi: 10.1016/j.jneuroim.2011.09.012. Epub 2011 Oct 28.

PMID:
22036952
9.

Immunology: A helping hand against autoimmunity.

Jetten AM.

Nature. 2011 Apr 28;472(7344):421-2. doi: 10.1038/472421a. No abstract available.

10.

The deviated balance between regulatory T cell and Th17 in autoimmunity.

Jadidi-Niaragh F, Mirshafiey A.

Immunopharmacol Immunotoxicol. 2012 Oct;34(5):727-39. doi: 10.3109/08923973.2011.619987. Epub 2012 Feb 9. Review.

PMID:
22316060
11.

1,25-Dihydroxyvitamin D3 inhibits the differentiation and migration of T(H)17 cells to protect against experimental autoimmune encephalomyelitis.

Chang JH, Cha HR, Lee DS, Seo KY, Kweon MN.

PLoS One. 2010 Sep 23;5(9):e12925. doi: 10.1371/journal.pone.0012925. Erratum in: PLoS One. 2010;5(12) doi: 10.1371/annotation/6da7b65e-dda2-467d-bcb3-82d5669f6bc6.

12.

Interferon-γ orchestrates the number and function of Th17 cells in experimental autoimmune encephalomyelitis.

Berghmans N, Nuyts A, Uyttenhove C, Van Snick J, Opdenakker G, Heremans H.

J Interferon Cytokine Res. 2011 Jul;31(7):575-87. doi: 10.1089/jir.2010.0137. Epub 2011 Feb 25.

PMID:
21348780
13.

Prostaglandin I2 signaling drives Th17 differentiation and exacerbates experimental autoimmune encephalomyelitis.

Zhou W, Dowell DR, Huckabee MM, Newcomb DC, Boswell MG, Goleniewska K, Lotz MT, Toki S, Yin H, Yao S, Natarajan C, Wu P, Sriram S, Breyer RM, Fitzgerald GA, Peebles RS Jr.

PLoS One. 2012;7(5):e33518. doi: 10.1371/journal.pone.0033518. Epub 2012 May 10.

14.

Protection of Tregs, suppression of Th1 and Th17 cells, and amelioration of experimental allergic encephalomyelitis by a physically-modified saline.

Mondal S, Martinson JA, Ghosh S, Watson R, Pahan K.

PLoS One. 2012;7(12):e51869. doi: 10.1371/journal.pone.0051869. Epub 2012 Dec 20.

15.

4-1BB triggering ameliorates experimental autoimmune encephalomyelitis by modulating the balance between Th17 and regulatory T cells.

Kim YH, Choi BK, Shin SM, Kim CH, Oh HS, Park SH, Lee DG, Lee MJ, Kim KH, Vinay DS, Kwon BS.

J Immunol. 2011 Aug 1;187(3):1120-8. doi: 10.4049/jimmunol.1002681. Epub 2011 Jun 29.

16.

Functional and pathogenic differences of Th1 and Th17 cells in experimental autoimmune encephalomyelitis.

Domingues HS, Mues M, Lassmann H, Wekerle H, Krishnamoorthy G.

PLoS One. 2010 Nov 29;5(11):e15531. doi: 10.1371/journal.pone.0015531.

17.

Translational mini-review series on Th17 cells: are T helper 17 cells really pathogenic in autoimmunity?

Koenders MI, van den Berg WB.

Clin Exp Immunol. 2010 Feb;159(2):131-6. doi: 10.1111/j.1365-2249.2009.04039.x. Epub 2009 Nov 11. Review.

18.

Calcitonin gene-related peptide enhances experimental autoimmune encephalomyelitis by promoting Th17-cell functions.

Mikami N, Watanabe K, Hashimoto N, Miyagi Y, Sueda K, Fukada S, Yamamoto H, Tsujikawa K.

Int Immunol. 2012 Nov;24(11):681-91. doi: 10.1093/intimm/dxs075. Epub 2012 Jul 27.

PMID:
22843730
19.

The NF-κB transcription factor c-Rel is required for Th17 effector cell development in experimental autoimmune encephalomyelitis.

Chen G, Hardy K, Pagler E, Ma L, Lee S, Gerondakis S, Daley S, Shannon MF.

J Immunol. 2011 Nov 1;187(9):4483-91. doi: 10.4049/jimmunol.1101757. Epub 2011 Sep 21.

20.

IL-21 and IL-21R are not required for development of Th17 cells and autoimmunity in vivo.

Sonderegger I, Kisielow J, Meier R, King C, Kopf M.

Eur J Immunol. 2008 Jul;38(7):1833-8. doi: 10.1002/eji.200838511.

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