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Items: 1 to 20 of 100

1.

Mitochondrial reactive oxygen species (ROS) as signaling molecules of intracellular pathways triggered by the cardiac renin-angiotensin II-aldosterone system (RAAS).

De Giusti VC, Caldiz CI, Ennis IL, Pérez NG, Cingolani HE, Aiello EA.

Front Physiol. 2013 May 30;4:126. doi: 10.3389/fphys.2013.00126. eCollection 2013.

2.

Role of reactive oxygen species (ROS) in angiotensin II-induced stimulation of the cardiac Na+/HCO3- cotransport.

De Giusti VC, Garciarena CD, Aiello EA.

J Mol Cell Cardiol. 2009 Nov;47(5):716-22. doi: 10.1016/j.yjmcc.2009.07.023. Epub 2009 Jul 30.

PMID:
19646989
3.

Modulation of the cardiac sodium/bicarbonate cotransporter by the renin angiotensin aldosterone system: pathophysiological consequences.

De Giusti VC, Ciancio MC, Orlowski A, Aiello EA.

Front Physiol. 2014 Jan 17;4:411. doi: 10.3389/fphys.2013.00411. eCollection 2013. Review.

4.

Aldosterone stimulates the cardiac sodium/bicarbonate cotransporter via activation of the g protein-coupled receptor gpr30.

De Giusti VC, Orlowski A, Ciancio MC, Espejo MS, Gonano LA, Caldiz CI, Vila Petroff MG, Villa-Abrille MC, Aiello EA.

J Mol Cell Cardiol. 2015 Dec;89(Pt B):260-7. doi: 10.1016/j.yjmcc.2015.10.024. Epub 2015 Oct 21.

PMID:
26497404
5.

Na+/H+ exchanger-1 inhibitors decrease myocardial superoxide production via direct mitochondrial action.

Garciarena CD, Caldiz CI, Correa MV, Schinella GR, Mosca SM, Chiappe de Cingolani GE, Cingolani HE, Ennis IL.

J Appl Physiol (1985). 2008 Dec;105(6):1706-13. doi: 10.1152/japplphysiol.90616.2008. Epub 2008 Sep 18.

6.

The positive inotropic effect of endothelin-1 is mediated by mitochondrial reactive oxygen species.

De Giusti VC, Correa MV, Villa-Abrille MC, Beltrano C, Yeves AM, de Cingolani GE, Cingolani HE, Aiello EA.

Life Sci. 2008 Aug 15;83(7-8):264-71. doi: 10.1016/j.lfs.2008.06.008. Epub 2008 Jun 24.

PMID:
18625248
7.

Reactive oxygen species partially mediate high dose angiotensin II-induced positive inotropic effect in cat ventricular myocytes.

Yeves AM, Caldiz CI, Aiello EA, Villa-Abrille MC, Ennis IL.

Cardiovasc Pathol. 2015 Jul-Aug;24(4):236-40. doi: 10.1016/j.carpath.2015.01.002. Epub 2015 Jan 28.

PMID:
25670255
8.

Aldosterone stimulates the cardiac Na(+)/H(+) exchanger via transactivation of the epidermal growth factor receptor.

De Giusti VC, Nolly MB, Yeves AM, Caldiz CI, Villa-Abrille MC, Chiappe de Cingolani GE, Ennis IL, Cingolani HE, Aiello EA.

Hypertension. 2011 Nov;58(5):912-9. doi: 10.1161/HYPERTENSIONAHA.111.176024.

9.

Role of mitochondria in angiotensin II-induced reactive oxygen species and mitogen-activated protein kinase activation.

Zhang GX, Lu XM, Kimura S, Nishiyama A.

Cardiovasc Res. 2007 Nov 1;76(2):204-12. Epub 2007 Jul 28. Review.

PMID:
17698051
10.

Mitochondrial reactive oxygen species: which ROS signals cardioprotection?

Garlid AO, Jaburek M, Jacobs JP, Garlid KD.

Am J Physiol Heart Circ Physiol. 2013 Oct 1;305(7):H960-8. doi: 10.1152/ajpheart.00858.2012. Epub 2013 Aug 2.

11.

Methods in the evaluation of cardiovascular renin angiotensin aldosterone activation and oxidative stress.

Manrique C, Lastra G, Habibi J, Wei Y, Morris EM, Stump CS, Sowers JR.

Methods Mol Med. 2007;139:163-79.

PMID:
18287671
12.
13.

Mitochondrial reactive oxygen species activate the slow force response to stretch in feline myocardium.

Caldiz CI, Garciarena CD, Dulce RA, Novaretto LP, Yeves AM, Ennis IL, Cingolani HE, Chiappe de Cingolani G, Pérez NG.

J Physiol. 2007 Nov 1;584(Pt 3):895-905. Epub 2007 Sep 6.

15.

Diazoxide prevents reactive oxygen species and mitochondrial damage, leading to anti-hypertrophic effects.

Lucas AM, Caldas FR, da Silva AP, Ventura MM, Leite IM, Filgueiras AB, Silva CG, Kowaltowski AJ, Facundo HT.

Chem Biol Interact. 2017 Jan 5;261:50-55. doi: 10.1016/j.cbi.2016.11.012. Epub 2016 Nov 17.

PMID:
27867086
17.

Redox signaling (cross-talk) from and to mitochondria involves mitochondrial pores and reactive oxygen species.

Daiber A.

Biochim Biophys Acta. 2010 Jun-Jul;1797(6-7):897-906. doi: 10.1016/j.bbabio.2010.01.032. Epub 2010 Feb 1. Review.

18.
19.

Mitochondrial K(ATP) channel openers activate the ERK kinase by an oxidant-dependent mechanism.

Samavati L, Monick MM, Sanlioglu S, Buettner GR, Oberley LW, Hunninghake GW.

Am J Physiol Cell Physiol. 2002 Jul;283(1):C273-81.

20.

Activation of CaMKII as a key regulator of reactive oxygen species production in diabetic rat heart.

Nishio S, Teshima Y, Takahashi N, Thuc LC, Saito S, Fukui A, Kume O, Fukunaga N, Hara M, Nakagawa M, Saikawa T.

J Mol Cell Cardiol. 2012 May;52(5):1103-11. doi: 10.1016/j.yjmcc.2012.02.006. Epub 2012 Feb 25.

PMID:
22394624

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