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Items: 1 to 20 of 136

1.

[The regulation of Akt signaling on axonal density after hypoxic-ischemic brain injury in neonatal rat].

Xiong T, Chen HJ, Qu Y, Luo BR, Mu DZ.

Sichuan Da Xue Xue Bao Yi Xue Ban. 2013 Mar;44(2):274-9. Chinese.

PMID:
23745271
2.

Involvement of the Akt/GSK-3β/CRMP-2 pathway in axonal injury after hypoxic-ischemic brain damage in neonatal rat.

Xiong T, Tang J, Zhao J, Chen H, Zhao F, Li J, Qu Y, Ferriero D, Mu D.

Neuroscience. 2012 Aug 2;216:123-32. doi: 10.1016/j.neuroscience.2012.04.052. Epub 2012 Apr 30.

PMID:
22554777
3.

[The Involvement of CRMP-2 on Axonal and Dendrictic Injury after Hypoxic-Ischemic Brain Injury in Neonatal Rat].

Xiong T, Chen HJ, Qu Y, Mu DZ.

Sichuan Da Xue Xue Bao Yi Xue Ban. 2015 Jul;46(4):513-8. Chinese.

PMID:
26480649
4.

G-CSF ameliorates neuronal apoptosis through GSK-3β inhibition in neonatal hypoxia-ischemia in rats.

Li L, Klebe D, Doycheva D, McBride DW, Krafft PR, Flores J, Zhou C, Zhang JH, Tang J.

Exp Neurol. 2015 Jan;263:141-9. doi: 10.1016/j.expneurol.2014.10.004. Epub 2014 Oct 18.

5.

Protective effect of proanthocyanidins on anoxia-reoxygenation injury of myocardial cells mediated by the PI3K/Akt/GSK-3β pathway and mitochondrial ATP-sensitive potassium channel.

Hu Y, Li L, Yin W, Shen L, You B, Gao H.

Mol Med Rep. 2014 Oct;10(4):2051-8. doi: 10.3892/mmr.2014.2459. Epub 2014 Aug 6.

PMID:
25109283
6.

GSK-3β inhibitor TDZD-8 reduces neonatal hypoxic-ischemic brain injury in mice.

Huang S, Wang H, Turlova E, Abussaud A, Ji X, Britto LR, Miller SP, Martinez A, Sun HS, Feng ZP.

CNS Neurosci Ther. 2017 May;23(5):405-415. doi: 10.1111/cns.12683. Epub 2017 Mar 2.

PMID:
28256059
7.

G-CSF attenuates neuroinflammation and stabilizes the blood-brain barrier via the PI3K/Akt/GSK-3β signaling pathway following neonatal hypoxia-ischemia in rats.

Li L, McBride DW, Doycheva D, Dixon BJ, Krafft PR, Zhang JH, Tang J.

Exp Neurol. 2015 Oct;272:135-44. doi: 10.1016/j.expneurol.2014.12.020. Epub 2015 Jan 10.

8.

Resveratrol prevents CA1 neurons against ischemic injury by parallel modulation of both GSK-3β and CREB through PI3-K/Akt pathways.

Simão F, Matté A, Pagnussat AS, Netto CA, Salbego CG.

Eur J Neurosci. 2012 Oct;36(7):2899-905. doi: 10.1111/j.1460-9568.2012.08229.x. Epub 2012 Jul 22.

PMID:
22817531
9.

Limb ischemic preconditioning protects against contrast-induced acute kidney injury in rats via phosphorylation of GSK-3β.

Liu T, Fang Y, Liu S, Yu X, Zhang H, Liang M, Ding X.

Free Radic Biol Med. 2015 Apr;81:170-82. doi: 10.1016/j.freeradbiomed.2014.10.509. Epub 2014 Oct 31.

PMID:
25451640
10.

Activation of Akt/GSK-3beta signaling pathway is involved in intermedin(1-53) protection against myocardial apoptosis induced by ischemia/reperfusion.

Song JQ, Teng X, Cai Y, Tang CS, Qi YF.

Apoptosis. 2009 Sep;14(9):1061-9. doi: 10.1007/s10495-009-0382-2.

PMID:
19634012
11.

Glycogen synthase kinase-3beta is involved in the process of myocardial hypertrophy stimulated by insulin-like growth factor-1.

Seimi SK, Seinosuke K, Tsuyoshi S, Tomomi U, Tetsuaki H, Miki K, Ryuji T, Kenji I, Mitsuhiro Y.

Circ J. 2004 Mar;68(3):247-53.

12.

Ischemic postconditioning mediates cardioprotection via PI3K/GSK-3β/β-catenin signaling pathway in ischemic rat myocardium.

Wu QL, Shen T, Shao LL, Ma H, Wang JK.

Shock. 2012 Aug;38(2):165-9. doi: 10.1097/SHK.0b013e31825b5633.

PMID:
22576003
13.

Progesterone alleviates hypoxic-ischemic brain injury via the Akt/GSK-3β signaling pathway.

Li X, Zhang J, Chai S, Wang X.

Exp Ther Med. 2014 Oct;8(4):1241-1246. Epub 2014 Jul 21.

14.

Role of the Akt/GSK-3β/CRMP-2 pathway in axon degeneration of dopaminergic neurons resulting from MPP+ toxicity.

Fang W, Gao G, Zhao H, Xia Y, Guo X, Li N, Li Y, Yang Y, Chen L, Wang Q, Li L.

Brain Res. 2015 Mar 30;1602:9-19. doi: 10.1016/j.brainres.2014.08.030. Epub 2014 Aug 21.

PMID:
25150591
15.

Activation of Akt/GSK-3beta signaling pathway is involved in intermedin(1-53) protection against myocardial apoptosis induced by ischemia/reperfusion.

Song JQ, Teng X, Cai Y, Tang CS, Qi YF.

Apoptosis. 2009 Nov;14(11):1299-307. doi: 10.1007/s10495-009-0398-7.

PMID:
19757065
16.

Glycogen synthase kinase-3β is involved in electroacupuncture pretreatment via the cannabinoid CB1 receptor in ischemic stroke.

Wei H, Yao X, Yang L, Wang S, Guo F, Zhou H, Marsicano G, Wang Q, Xiong L.

Mol Neurobiol. 2014 Feb;49(1):326-36. doi: 10.1007/s12035-013-8524-5. Epub 2013 Aug 14.

PMID:
23943518
17.

Sevoflurane postconditioning improves long-term learning and memory of neonatal hypoxia-ischemia brain damage rats via the PI3K/Akt-mPTP pathway.

Lai Z, Zhang L, Su J, Cai D, Xu Q.

Brain Res. 2016 Jan 1;1630:25-37. doi: 10.1016/j.brainres.2015.10.050. Epub 2015 Nov 2.

PMID:
26541582
18.

Neuroprotection induced by sevoflurane-delayed post-conditioning is attributable to increased phosphorylation of mitochondrial GSK-3β through the PI3K/Akt survival pathway.

Ye Z, Xia P, Cheng ZG, Guo Q.

J Neurol Sci. 2015 Jan 15;348(1-2):216-25. doi: 10.1016/j.jns.2014.12.011. Epub 2014 Dec 18.

PMID:
25555490
19.

Neuronal pentraxin 1 induction in hypoxic-ischemic neuronal death is regulated via a glycogen synthase kinase-3α/β dependent mechanism.

Russell JC, Kishimoto K, O'Driscoll C, Hossain MA.

Cell Signal. 2011 Apr;23(4):673-82. doi: 10.1016/j.cellsig.2010.11.021. Epub 2010 Dec 3.

20.

[Role of integrin-linked kinase signaling pathway in skin lesions and wound healing in diabetic rats].

Zhou R, Li Y, Li G, Lin W, Sun Je, Zhou W.

Zhonghua Shao Shang Za Zhi. 2016 Apr;32(4):216-23. doi: 10.3760/cma.j.issn.1009-2587.2016.04.006. Chinese.

PMID:
27093933

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