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Items: 1 to 20 of 79

1.

Glucose regulation of load-induced mTOR signaling and ER stress in mammalian heart.

Sen S, Kundu BK, Wu HC, Hashmi SS, Guthrie P, Locke LW, Roy RJ, Matherne GP, Berr SS, Terwelp M, Scott B, Carranza S, Frazier OH, Glover DK, Dillmann WH, Gambello MJ, Entman ML, Taegtmeyer H.

J Am Heart Assoc. 2013 May 17;2(3):e004796. doi: 10.1161/JAHA.113.004796.

2.

Remodeling of glucose metabolism precedes pressure overload-induced left ventricular hypertrophy: review of a hypothesis.

Kundu BK, Zhong M, Sen S, Davogustto G, Keller SR, Taegtmeyer H.

Cardiology. 2015;130(4):211-20. doi: 10.1159/000369782. Epub 2015 Mar 12. Review.

3.

Improved insulin sensitivity by rapamycin is associated with reduction of mTOR and S6K1 activities in L6 myotubes.

Hwang SL, Li X, Lee JY, Chang HW.

Biochem Biophys Res Commun. 2012 Feb 10;418(2):402-7. doi: 10.1016/j.bbrc.2012.01.038. Epub 2012 Jan 18.

PMID:
22281494
4.

AMPK activation prevents excess nutrient-induced hepatic lipid accumulation by inhibiting mTORC1 signaling and endoplasmic reticulum stress response.

Li H, Min Q, Ouyang C, Lee J, He C, Zou MH, Xie Z.

Biochim Biophys Acta. 2014 Sep;1842(9):1844-54. doi: 10.1016/j.bbadis.2014.07.002. Epub 2014 Jul 10.

5.

Chronic inhibition of cGMP-specific phosphodiesterase 5 suppresses endoplasmic reticulum stress in heart failure.

Gong W, Duan Q, Cai Z, Chen C, Ni L, Yan M, Wang X, Cianflone K, Wang DW.

Br J Pharmacol. 2013 Dec;170(7):1396-409. doi: 10.1111/bph.12346.

6.

Cadmium toxicity induces ER stress and apoptosis via impairing energy homoeostasis in cardiomyocytes.

Chen CY, Zhang SL, Liu ZY, Tian Y, Sun Q.

Biosci Rep. 2015 Apr 10;35(3). pii: e00214. doi: 10.1042/BSR20140170.

7.

Bidirectional crosstalk between endoplasmic reticulum stress and mTOR signaling.

Appenzeller-Herzog C, Hall MN.

Trends Cell Biol. 2012 May;22(5):274-82. doi: 10.1016/j.tcb.2012.02.006. Epub 2012 Mar 21. Review.

PMID:
22444729
8.

Mitochondrial aldehyde dehydrogenase obliterates endoplasmic reticulum stress-induced cardiac contractile dysfunction via correction of autophagy.

Zhang B, Zhang Y, La Cour KH, Richmond KL, Wang XM, Ren J.

Biochim Biophys Acta. 2013 Apr;1832(4):574-84. doi: 10.1016/j.bbadis.2013.01.013. Epub 2013 Jan 23.

9.

Inhibition of the mTOR/p70S6K pathway is not involved in the insulin-sensitizing effect of AMPK on cardiac glucose uptake.

Ginion A, Auquier J, Benton CR, Mouton C, Vanoverschelde JL, Hue L, Horman S, Beauloye C, Bertrand L.

Am J Physiol Heart Circ Physiol. 2011 Aug;301(2):H469-77. doi: 10.1152/ajpheart.00986.2010. Epub 2011 May 20.

PMID:
21602475
10.

Isoproterenol instigates cardiomyocyte apoptosis and heart failure via AMPK inactivation-mediated endoplasmic reticulum stress.

Zhuo XZ, Wu Y, Ni YJ, Liu JH, Gong M, Wang XH, Wei F, Wang TZ, Yuan Z, Ma AQ, Song P.

Apoptosis. 2013 Jul;18(7):800-10. doi: 10.1007/s10495-013-0843-5.

PMID:
23620435
11.

Rapamycin attenuates visible light-induced injury in retinal photoreceptor cells via inhibiting endoplasmic reticulum stress.

Li GY, Fan B, Jiao YY.

Brain Res. 2014 May 14;1563:1-12. doi: 10.1016/j.brainres.2014.02.020. Epub 2014 Mar 4.

PMID:
24607296
12.

Activation of mTOR modulates SREBP-2 to induce foam cell formation through increased retinoblastoma protein phosphorylation.

Ma KL, Liu J, Wang CX, Ni J, Zhang Y, Wu Y, Lv LL, Ruan XZ, Liu BC.

Cardiovasc Res. 2013 Dec 1;100(3):450-60. doi: 10.1093/cvr/cvt203. Epub 2013 Sep 25.

PMID:
24068000
13.
14.

ER stress negatively regulates AKT/TSC/mTOR pathway to enhance autophagy.

Qin L, Wang Z, Tao L, Wang Y.

Autophagy. 2010 Feb;6(2):239-47. Epub 2010 Mar 1.

PMID:
20104019
16.

Inhibitory cross-talk between the AMPK and ERK pathways mediates endoplasmic reticulum stress-induced insulin resistance in skeletal muscle.

Hwang SL, Jeong YT, Li X, Kim YD, Lu Y, Chang YC, Lee IK, Chang HW.

Br J Pharmacol. 2013 May;169(1):69-81. doi: 10.1111/bph.12124.

17.

Glucose phosphorylation is required for insulin-dependent mTOR signalling in the heart.

Sharma S, Guthrie PH, Chan SS, Haq S, Taegtmeyer H.

Cardiovasc Res. 2007 Oct 1;76(1):71-80. Epub 2007 May 10.

18.

Deficiency of cardiac Acyl-CoA synthetase-1 induces diastolic dysfunction, but pathologic hypertrophy is reversed by rapamycin.

Paul DS, Grevengoed TJ, Pascual F, Ellis JM, Willis MS, Coleman RA.

Biochim Biophys Acta. 2014 Jun;1841(6):880-7. doi: 10.1016/j.bbalip.2014.03.001. Epub 2014 Mar 12.

19.

Thyroid hormone improves the mechanical performance of the post-infarcted diabetic myocardium: a response associated with up-regulation of Akt/mTOR and AMPK activation.

Mourouzis I, Giagourta I, Galanopoulos G, Mantzouratou P, Kostakou E, Kokkinos AD, Tentolouris N, Pantos C.

Metabolism. 2013 Oct;62(10):1387-93. doi: 10.1016/j.metabol.2013.05.008. Epub 2013 Jun 15.

PMID:
23773982
20.

NAD blocks high glucose induced mesangial hypertrophy via activation of the sirtuins-AMPK-mTOR pathway.

Zhuo L, Fu B, Bai X, Zhang B, Wu L, Cui J, Cui S, Wei R, Chen X, Cai G.

Cell Physiol Biochem. 2011;27(6):681-90. doi: 10.1159/000330077. Epub 2011 Jun 17.

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