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Items: 1 to 20 of 188

1.

The Rb1 gene inhibits the viability of retinoblastoma cells by regulating homologous recombination.

Yang Y, Tian S, Brown B, Chen P, Hu H, Xia L, Zhang J, Cai X, Chen Z, Pan X, Ge J, Yu K, Zhuang J.

Int J Mol Med. 2013 Jul;32(1):137-43. doi: 10.3892/ijmm.2013.1374.

PMID:
23670186
2.

The efficiency of homologous recombination and non-homologous end joining systems in repairing double-strand breaks during cell cycle progression.

Bee L, Fabris S, Cherubini R, Mognato M, Celotti L.

PLoS One. 2013 Jul 11;8(7):e69061. doi: 10.1371/journal.pone.0069061.

3.
4.

Deregulation of DNA double-strand break repair in multiple myeloma: implications for genome stability.

Herrero AB, San Miguel J, Gutierrez NC.

PLoS One. 2015 Mar 19;10(3):e0121581. doi: 10.1371/journal.pone.0121581.

5.

Homologous recombination protects mammalian cells from replication-associated DNA double-strand breaks arising in response to methyl methanesulfonate.

Nikolova T, Ensminger M, Löbrich M, Kaina B.

DNA Repair (Amst). 2010 Oct 5;9(10):1050-63. doi: 10.1016/j.dnarep.2010.07.005.

PMID:
20708982
6.

Regulation of DNA double-strand break repair pathway choice.

Shrivastav M, De Haro LP, Nickoloff JA.

Cell Res. 2008 Jan;18(1):134-47. Review.

8.

Inhibition of homologous recombination by hyperthermia shunts early double strand break repair to non-homologous end-joining.

Bergs JW, Krawczyk PM, Borovski T, ten Cate R, Rodermond HM, Stap J, Medema JP, Haveman J, Essers J, van Bree C, Stalpers LJ, Kanaar R, Aten JA, Franken NA.

DNA Repair (Amst). 2013 Jan 1;12(1):38-45. doi: 10.1016/j.dnarep.2012.10.008.

PMID:
23237939
9.

Non-homologous end joining is the responsible pathway for the repair of fludarabine-induced DNA double strand breaks in mammalian cells.

de Campos-Nebel M, Larripa I, González-Cid M.

Mutat Res. 2008 Nov 10;646(1-2):8-16. doi: 10.1016/j.mrfmmm.2008.08.013.

PMID:
18812179
10.

Transcriptomic analysis provides insights on hexavalent chromium induced DNA double strand breaks and their possible repair in midgut cells of Drosophila melanogaster larvae.

Mishra M, Sharma A, Shukla AK, Pragya P, Murthy RC, de Pomerai D, Dwivedi UN, Chowdhuri DK.

Mutat Res. 2013 Jul-Aug;747-748:28-39. doi: 10.1016/j.mrfmmm.2013.04.005.

PMID:
23628323
11.

Increased sensitivity to ionizing radiation by targeting the homologous recombination pathway in glioma initiating cells.

Lim YC, Roberts TL, Day BW, Stringer BW, Kozlov S, Fazry S, Bruce ZC, Ensbey KS, Walker DG, Boyd AW, Lavin MF.

Mol Oncol. 2014 Dec;8(8):1603-15. doi: 10.1016/j.molonc.2014.06.012.

12.

BMS-345541 sensitizes MCF-7 breast cancer cells to ionizing radiation by selective inhibition of homologous recombinational repair of DNA double-strand breaks.

Wu L, Shao L, Li M, Zheng J, Wang J, Feng W, Chang J, Wang Y, Hauer-Jensen M, Zhou D.

Radiat Res. 2013 Feb;179(2):160-70. doi: 10.1667/RR3034.1.

13.

Homologous recombination is a primary pathway to repair DNA double-strand breaks generated during DNA rereplication.

Truong LN, Li Y, Sun E, Ang K, Hwang PY, Wu X.

J Biol Chem. 2014 Oct 17;289(42):28910-23. doi: 10.1074/jbc.M114.576488.

14.

Expression of the TrkA or TrkB receptor tyrosine kinase alters the double-strand break (DSB) repair capacity of SY5Y neuroblastoma cells.

Schulte JH, Kuhfittig-Kulle S, Klein-Hitpass L, Schramm A, Biard DS, Pfeiffer P, Eggert A.

DNA Repair (Amst). 2008 Oct 1;7(10):1757-64. doi: 10.1016/j.dnarep.2008.07.004.

PMID:
18678285
15.

Constitutional genomic instability, chromosome aberrations in tumor cells and retinoblastoma.

Amare Kadam PS, Ghule P, Jose J, Bamne M, Kurkure P, Banavali S, Sarin R, Advani S.

Cancer Genet Cytogenet. 2004 Apr 1;150(1):33-43.

PMID:
15041221
16.

Canonical non-homologous end joining in mitosis induces genome instability and is suppressed by M-phase-specific phosphorylation of XRCC4.

Terasawa M, Shinohara A, Shinohara M.

PLoS Genet. 2014 Aug 28;10(8):e1004563. doi: 10.1371/journal.pgen.1004563.

17.
18.

Loss of heterozygosity and mutations are the major mechanisms of RB1 gene inactivation in Chinese with sporadic retinoblastoma.

Choy KW, Pang CP, Yu CB, Wong HL, Ng JS, Fan DS, Lo KW, Chai JT, Wang J, Fu W, Lam DS.

Hum Mutat. 2002 Nov;20(5):408.

PMID:
12402348
19.

RB1 gene inactivation by chromothripsis in human retinoblastoma.

McEvoy J, Nagahawatte P, Finkelstein D, Richards-Yutz J, Valentine M, Ma J, Mullighan C, Song G, Chen X, Wilson M, Brennan R, Pounds S, Becksfort J, Huether R, Lu C, Fulton RS, Fulton LL, Hong X, Dooling DJ, Ochoa K, Mardis ER, Wilson RK, Easton J, Zhang J, Downing JR, Ganguly A, Dyer MA.

Oncotarget. 2014 Jan 30;5(2):438-50.

20.

Direct involvement of retinoblastoma family proteins in DNA repair by non-homologous end-joining.

Cook R, Zoumpoulidou G, Luczynski MT, Rieger S, Moquet J, Spanswick VJ, Hartley JA, Rothkamm K, Huang PH, Mittnacht S.

Cell Rep. 2015 Mar 31;10(12):2006-18. doi: 10.1016/j.celrep.2015.02.059.

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