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Items: 1 to 20 of 96

1.

Mitochondrial ROS in the prohypertensive immune response.

Nazarewicz RR, Dikalov SI.

Am J Physiol Regul Integr Comp Physiol. 2013 Jul 15;305(2):R98-100. doi: 10.1152/ajpregu.00208.2013. Epub 2013 May 8. Review.

2.
3.

Mitochondrial ROS, uncoupled from ATP synthesis, determine endothelial activation for both physiological recruitment of patrolling cells and pathological recruitment of inflammatory cells.

Li X, Fang P, Yang WY, Chan K, Lavallee M, Xu K, Gao T, Wang H, Yang X.

Can J Physiol Pharmacol. 2017 Mar;95(3):247-252. doi: 10.1139/cjpp-2016-0515. Epub 2016 Nov 5. Review.

4.

TNF-alpha/cycloheximide-induced apoptosis in intestinal epithelial cells requires Rac1-regulated reactive oxygen species.

Jin S, Ray RM, Johnson LR.

Am J Physiol Gastrointest Liver Physiol. 2008 Apr;294(4):G928-37. doi: 10.1152/ajpgi.00219.2007. Epub 2008 Jan 24.

6.

Administration of interleukin-17 soluble receptor C suppresses TH17 cells, oxidative stress, and hypertension in response to placental ischemia during pregnancy.

Cornelius DC, Hogg JP, Scott J, Wallace K, Herse F, Moseley J, Wallukat G, Dechend R, LaMarca B.

Hypertension. 2013 Dec;62(6):1068-73. doi: 10.1161/HYPERTENSIONAHA.113.01514. Epub 2013 Sep 23.

7.

Redox signaling (cross-talk) from and to mitochondria involves mitochondrial pores and reactive oxygen species.

Daiber A.

Biochim Biophys Acta. 2010 Jun-Jul;1797(6-7):897-906. doi: 10.1016/j.bbabio.2010.01.032. Epub 2010 Feb 1. Review.

8.

Role of mitochondrial oxidative stress in hypertension.

Dikalov SI, Ungvari Z.

Am J Physiol Heart Circ Physiol. 2013 Nov 15;305(10):H1417-27. doi: 10.1152/ajpheart.00089.2013. Epub 2013 Sep 16. Review.

9.

Elevated mitochondrial reactive oxygen species generation affects the immune response via hypoxia-inducible factor-1alpha in long-lived Mclk1+/- mouse mutants.

Wang D, Malo D, Hekimi S.

J Immunol. 2010 Jan 15;184(2):582-90. doi: 10.4049/jimmunol.0902352. Epub 2009 Dec 9.

10.

Mitochondria and NADPH oxidases are the major sources of TNF-α/cycloheximide-induced oxidative stress in murine intestinal epithelial MODE-K cells.

Babu D, Leclercq G, Goossens V, Vanden Berghe T, Van Hamme E, Vandenabeele P, Lefebvre RA.

Cell Signal. 2015 Jun;27(6):1141-58. doi: 10.1016/j.cellsig.2015.02.019. Epub 2015 Feb 26.

PMID:
25725292
11.

The putative role of mitochondrial dysfunction in hypertension.

Puddu P, Puddu GM, Cravero E, De Pascalis S, Muscari A.

Clin Exp Hypertens. 2007 Oct;29(7):427-34. Review.

PMID:
17994352
12.

Interrelation of oxidative stress and inflammation in neurodegenerative disease: role of TNF.

Fischer R, Maier O.

Oxid Med Cell Longev. 2015;2015:610813. doi: 10.1155/2015/610813. Epub 2015 Mar 5. Review.

13.

Mitochondrial ROS govern the LPS-induced pro-inflammatory response in microglia cells by regulating MAPK and NF-κB pathways.

Park J, Min JS, Kim B, Chae UB, Yun JW, Choi MS, Kong IK, Chang KT, Lee DS.

Neurosci Lett. 2015 Jan 1;584:191-6. doi: 10.1016/j.neulet.2014.10.016. Epub 2014 Oct 22.

PMID:
25459294
14.

Mitochondrial reactive oxygen species and risk of atherosclerosis.

Hulsmans M, Van Dooren E, Holvoet P.

Curr Atheroscler Rep. 2012 Jun;14(3):264-76. doi: 10.1007/s11883-012-0237-0. Review.

PMID:
22350585
15.

Differential phenotypes of tissue-infiltrating T cells during angiotensin II-induced hypertension in mice.

Wei Z, Spizzo I, Diep H, Drummond GR, Widdop RE, Vinh A.

PLoS One. 2014 Dec 11;9(12):e114895. doi: 10.1371/journal.pone.0114895. eCollection 2014.

16.

Visualization of vascular Ca2+ signaling triggered by paracrine derived ROS.

Mallilankaraman K, Gandhirajan RK, Hawkins BJ, Madesh M.

J Vis Exp. 2011 Dec 21;(58). pii: 3511. doi: 10.3791/3511.

17.

Mitochondrial reactive oxygen species promote production of proinflammatory cytokines and are elevated in TNFR1-associated periodic syndrome (TRAPS).

Bulua AC, Simon A, Maddipati R, Pelletier M, Park H, Kim KY, Sack MN, Kastner DL, Siegel RM.

J Exp Med. 2011 Mar 14;208(3):519-33. doi: 10.1084/jem.20102049. Epub 2011 Jan 31.

19.

Pro-inflammatory cytokines increase reactive oxygen species through mitochondria and NADPH oxidase in cultured RPE cells.

Yang D, Elner SG, Bian ZM, Till GO, Petty HR, Elner VM.

Exp Eye Res. 2007 Oct;85(4):462-72. Epub 2007 Jun 27.

20.

Angiotensin II-induced production of mitochondrial reactive oxygen species: potential mechanisms and relevance for cardiovascular disease.

Dikalov SI, Nazarewicz RR.

Antioxid Redox Signal. 2013 Oct 1;19(10):1085-94. doi: 10.1089/ars.2012.4604. Epub 2012 May 21. Review.

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