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Items: 1 to 20 of 126

1.

Transforming growth factor-β-induced cross talk between p53 and a microRNA in the pathogenesis of diabetic nephropathy.

Deshpande SD, Putta S, Wang M, Lai JY, Bitzer M, Nelson RG, Lanting LL, Kato M, Natarajan R.

Diabetes. 2013 Sep;62(9):3151-62. doi: 10.2337/db13-0305. Epub 2013 May 6.

2.

MicroRNA-192 in diabetic kidney glomeruli and its function in TGF-beta-induced collagen expression via inhibition of E-box repressors.

Kato M, Zhang J, Wang M, Lanting L, Yuan H, Rossi JJ, Natarajan R.

Proc Natl Acad Sci U S A. 2007 Feb 27;104(9):3432-7. Epub 2007 Feb 20.

3.

Transforming growth factor β1 (TGF-β1) enhances expression of profibrotic genes through a novel signaling cascade and microRNAs in renal mesangial cells.

Castro NE, Kato M, Park JT, Natarajan R.

J Biol Chem. 2014 Oct 17;289(42):29001-13. doi: 10.1074/jbc.M114.600783. Epub 2014 Sep 9.

4.

A microRNA circuit mediates transforming growth factor-β1 autoregulation in renal glomerular mesangial cells.

Kato M, Arce L, Wang M, Putta S, Lanting L, Natarajan R.

Kidney Int. 2011 Aug;80(4):358-68. doi: 10.1038/ki.2011.43. Epub 2011 Mar 9.

5.

Inhibiting microRNA-192 ameliorates renal fibrosis in diabetic nephropathy.

Putta S, Lanting L, Sun G, Lawson G, Kato M, Natarajan R.

J Am Soc Nephrol. 2012 Mar;23(3):458-69. doi: 10.1681/ASN.2011050485. Epub 2012 Jan 5.

6.

MicroRNA-21 in glomerular injury.

Lai JY, Luo J, O'Connor C, Jing X, Nair V, Ju W, Randolph A, Ben-Dov IZ, Matar RN, Briskin D, Zavadil J, Nelson RG, Tuschl T, Brosius FC 3rd, Kretzler M, Bitzer M.

J Am Soc Nephrol. 2015 Apr;26(4):805-16. doi: 10.1681/ASN.2013121274. Epub 2014 Aug 21.

7.

MicroRNA-26a inhibits TGF-β-induced extracellular matrix protein expression in podocytes by targeting CTGF and is downregulated in diabetic nephropathy.

Koga K, Yokoi H, Mori K, Kasahara M, Kuwabara T, Imamaki H, Ishii A, Mori KP, Kato Y, Ohno S, Toda N, Saleem MA, Sugawara A, Nakao K, Yanagita M, Mukoyama M.

Diabetologia. 2015 Sep;58(9):2169-80. doi: 10.1007/s00125-015-3642-4. Epub 2015 Jun 11.

PMID:
26063197
8.

miR-21 promotes renal fibrosis in diabetic nephropathy by targeting PTEN and SMAD7.

McClelland AD, Herman-Edelstein M, Komers R, Jha JC, Winbanks CE, Hagiwara S, Gregorevic P, Kantharidis P, Cooper ME.

Clin Sci (Lond). 2015 Dec;129(12):1237-49. doi: 10.1042/CS20150427. Epub 2015 Sep 28.

PMID:
26415649
9.

FOG2 protein down-regulation by transforming growth factor-β1-induced microRNA-200b/c leads to Akt kinase activation and glomerular mesangial hypertrophy related to diabetic nephropathy.

Park JT, Kato M, Yuan H, Castro N, Lanting L, Wang M, Natarajan R.

J Biol Chem. 2013 Aug 2;288(31):22469-80. doi: 10.1074/jbc.M113.453043. Epub 2013 Jun 20.

10.

Enhanced TGF-beta/Smad signaling in the early stage of diabetic nephropathy is independent of the AT1a receptor.

Okazaki Y, Yamasaki Y, Uchida HA, Okamoto K, Satoh M, Maruyama K, Maeshima Y, Sugiyama H, Sugaya T, Kashihara N, Makino H.

Clin Exp Nephrol. 2007 Mar;11(1):77-87. Epub 2007 Mar 28.

PMID:
17385003
11.

Abated microRNA-195 expression protected mesangial cells from apoptosis in early diabetic renal injury in mice.

Chen YQ, Wang XX, Yao XM, Zhang DL, Yang XF, Tian SF, Wang NS.

J Nephrol. 2012 Jul-Aug;25(4):566-76. doi: 10.5301/jn.5000034.

PMID:
21983986
12.

Cellular basis of diabetic nephropathy: II. The transforming growth factor-beta system and diabetic nephropathy lesions in type 1 diabetes.

Huang C, Kim Y, Caramori ML, Fish AJ, Rich SS, Miller ME, Russell GB, Mauer M.

Diabetes. 2002 Dec;51(12):3577-81.

13.

Post-transcriptional up-regulation of Tsc-22 by Ybx1, a target of miR-216a, mediates TGF-{beta}-induced collagen expression in kidney cells.

Kato M, Wang L, Putta S, Wang M, Yuan H, Sun G, Lanting L, Todorov I, Rossi JJ, Natarajan R.

J Biol Chem. 2010 Oct 29;285(44):34004-15. doi: 10.1074/jbc.M110.165027. Epub 2010 Aug 16.

14.

miR-21 is a key therapeutic target for renal injury in a mouse model of type 2 diabetes.

Zhong X, Chung AC, Chen HY, Dong Y, Meng XM, Li R, Yang W, Hou FF, Lan HY.

Diabetologia. 2013 Mar;56(3):663-74. doi: 10.1007/s00125-012-2804-x. Epub 2013 Jan 5.

PMID:
23292313
15.

miR-21 overexpression enhances TGF-β1-induced epithelial-to-mesenchymal transition by target smad7 and aggravates renal damage in diabetic nephropathy.

Wang JY, Gao YB, Zhang N, Zou DW, Wang P, Zhu ZY, Li JY, Zhou SN, Wang SC, Wang YY, Yang JK.

Mol Cell Endocrinol. 2014 Jul 5;392(1-2):163-72. doi: 10.1016/j.mce.2014.05.018. Epub 2014 Jun 2.

PMID:
24887517
16.

Absence of miR-146a in Podocytes Increases Risk of Diabetic Glomerulopathy via Up-regulation of ErbB4 and Notch-1.

Lee HW, Khan SQ, Khaliqdina S, Altintas MM, Grahammer F, Zhao JL, Koh KH, Tardi NJ, Faridi MH, Geraghty T, Cimbaluk DJ, Susztak K, Moita LF, Baltimore D, Tharaux PL, Huber TB, Kretzler M, Bitzer M, Reiser J, Gupta V.

J Biol Chem. 2017 Jan 13;292(2):732-747. doi: 10.1074/jbc.M116.753822. Epub 2016 Dec 2.

PMID:
27913625
17.

Transforming growth factor-β1-mediated renal fibrosis is dependent on the regulation of transforming growth factor receptor 1 expression by let-7b.

Wang B, Jha JC, Hagiwara S, McClelland AD, Jandeleit-Dahm K, Thomas MC, Cooper ME, Kantharidis P.

Kidney Int. 2014 Feb;85(2):352-61. doi: 10.1038/ki.2013.372. Epub 2013 Oct 2.

PMID:
24088962
18.

miR-378 reduces mesangial hypertrophy and kidney tubular fibrosis via MAPK signalling.

Wang B, Yao K, Wise AF, Lau R, Shen HH, Tesch GH, Ricardo SD.

Clin Sci (Lond). 2017 Mar 1;131(5):411-423. doi: 10.1042/CS20160571. Epub 2017 Jan 4.

PMID:
28053239
19.

MicroRNA-23b Targets Ras GTPase-Activating Protein SH3 Domain-Binding Protein 2 to Alleviate Fibrosis and Albuminuria in Diabetic Nephropathy.

Zhao B, Li H, Liu J, Han P, Zhang C, Bai H, Yuan X, Wang X, Li L, Ma H, Jin X, Chu Y.

J Am Soc Nephrol. 2016 Sep;27(9):2597-608. doi: 10.1681/ASN.2015030300. Epub 2016 Feb 2.

PMID:
26839366
20.

MicroRNA-29b inhibits diabetic nephropathy in db/db mice.

Chen HY, Zhong X, Huang XR, Meng XM, You Y, Chung AC, Lan HY.

Mol Ther. 2014 Apr;22(4):842-53. doi: 10.1038/mt.2013.235. Epub 2013 Dec 6.

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