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Items: 1 to 20 of 132

1.

Angiotensin II is a new component involved in splenic T lymphocyte responses during Plasmodium berghei ANKA infection.

Silva-Filho JL, Souza MC, Ferreira-Dasilva CT, Silva LS, Costa MF, Padua TA, Henriques Md, Morrot A, Savino W, Caruso-Neves C, Pinheiro AA.

PLoS One. 2013 Apr 30;8(4):e62999. doi: 10.1371/journal.pone.0062999. Print 2013.

2.

Targeting Angiotensin II Type-1 Receptor (AT1R) Inhibits the Harmful Phenotype of Plasmodium-Specific CD8+ T Cells during Blood-Stage Malaria.

Silva-Filho JL, Caruso-Neves C, Pinheiro AA.

Front Cell Infect Microbiol. 2017 Feb 16;7:42. doi: 10.3389/fcimb.2017.00042. eCollection 2017.

3.

Malaria-specific and nonspecific activation of CD8+ T cells during blood stage of Plasmodium berghei infection.

Miyakoda M, Kimura D, Yuda M, Chinzei Y, Shibata Y, Honma K, Yui K.

J Immunol. 2008 Jul 15;181(2):1420-8.

4.

The transcription factor T-bet regulates parasitemia and promotes pathogenesis during Plasmodium berghei ANKA murine malaria.

Oakley MS, Sahu BR, Lotspeich-Cole L, Solanki NR, Majam V, Pham PT, Banerjee R, Kozakai Y, Derrick SC, Kumar S, Morris SL.

J Immunol. 2013 Nov 1;191(9):4699-708. doi: 10.4049/jimmunol.1300396. Epub 2013 Sep 27.

5.

Pre-existing Schistosoma japonicum infection alters the immune response to Plasmodium berghei infection in C57BL/6 mice.

Wang ML, Cao YM, Luo EJ, Zhang Y, Guo YJ.

Malar J. 2013 Sep 14;12:322. doi: 10.1186/1475-2875-12-322.

6.

Differential role of T regulatory and Th17 in Swiss mice infected with Plasmodium berghei ANKA and Plasmodium yoelii.

Keswani T, Bhattacharyya A.

Exp Parasitol. 2014 Jun;141:82-92. doi: 10.1016/j.exppara.2014.03.003. Epub 2014 Mar 24.

PMID:
24675415
7.

Perforin-dependent brain-infiltrating cytotoxic CD8+ T lymphocytes mediate experimental cerebral malaria pathogenesis.

Nitcheu J, Bonduelle O, Combadiere C, Tefit M, Seilhean D, Mazier D, Combadiere B.

J Immunol. 2003 Feb 15;170(4):2221-8.

8.

AT1 receptor-mediated angiotensin II activation and chemotaxis of T lymphocytes.

Silva-Filho JL, Souza MC, Henriques Md, Morrot A, Savino W, Nunes MP, Caruso-Neves C, Pinheiro AA.

Mol Immunol. 2011 Sep;48(15-16):1835-43. doi: 10.1016/j.molimm.2011.05.008. Epub 2011 Jun 8.

PMID:
21641648
9.

Protein kinase C θ deficiency increases resistance of C57BL/6J mice to Plasmodium berghei infection-induced cerebral malaria.

Ohayon A, Golenser J, Sinay R, Tamir A, Altman A, Pollack Y, Isakov N.

Infect Immun. 2010 Oct;78(10):4195-205. doi: 10.1128/IAI.00465-10. Epub 2010 Jul 26.

10.

Heterogeneous and tissue-specific regulation of effector T cell responses by IFN-gamma during Plasmodium berghei ANKA infection.

Villegas-Mendez A, de Souza JB, Murungi L, Hafalla JC, Shaw TN, Greig R, Riley EM, Couper KN.

J Immunol. 2011 Sep 15;187(6):2885-97. doi: 10.4049/jimmunol.1100241. Epub 2011 Aug 31.

11.

NK cells and conventional dendritic cells engage in reciprocal activation for the induction of inflammatory responses during Plasmodium berghei ANKA infection.

Ryg-Cornejo V, Nie CQ, Bernard NJ, Lundie RJ, Evans KJ, Crabb BS, Schofield L, Hansen DS.

Immunobiology. 2013 Feb;218(2):263-71. doi: 10.1016/j.imbio.2012.05.018. Epub 2012 May 23.

PMID:
22704523
12.

Type I interferons contribute to experimental cerebral malaria development in response to sporozoite or blood-stage Plasmodium berghei ANKA.

Palomo J, Fauconnier M, Coquard L, Gilles M, Meme S, Szeremeta F, Fick L, Franetich JF, Jacobs M, Togbe D, Beloeil JC, Mazier D, Ryffel B, Quesniaux VF.

Eur J Immunol. 2013 Oct;43(10):2683-95. doi: 10.1002/eji.201343327. Epub 2013 Jul 19.

14.

Ligation of B and T lymphocyte attenuator prevents the genesis of experimental cerebral malaria.

Lepenies B, Pfeffer K, Hurchla MA, Murphy TL, Murphy KM, Oetzel J, Fleischer B, Jacobs T.

J Immunol. 2007 Sep 15;179(6):4093-100.

15.

Parasite densities modulate susceptibility of mice to cerebral malaria during co-infection with Schistosoma japonicum and Plasmodium berghei.

Wang ML, Feng YH, Pang W, Qi ZM, Zhang Y, Guo YJ, Luo EJ, Cao YM.

Malar J. 2014 Mar 26;13:116. doi: 10.1186/1475-2875-13-116.

16.

NK cells stimulate recruitment of CXCR3+ T cells to the brain during Plasmodium berghei-mediated cerebral malaria.

Hansen DS, Bernard NJ, Nie CQ, Schofield L.

J Immunol. 2007 May 1;178(9):5779-88.

17.

Cutting edge: selective blockade of LIGHT-lymphotoxin beta receptor signaling protects mice from experimental cerebral malaria caused by Plasmodium berghei ANKA.

Randall LM, Amante FH, Zhou Y, Stanley AC, Haque A, Rivera F, Pfeffer K, Scheu S, Hill GR, Tamada K, Engwerda CR.

J Immunol. 2008 Dec 1;181(11):7458-62.

18.
19.

CXCR3 determines strain susceptibility to murine cerebral malaria by mediating T lymphocyte migration toward IFN-gamma-induced chemokines.

Van den Steen PE, Deroost K, Van Aelst I, Geurts N, Martens E, Struyf S, Nie CQ, Hansen DS, Matthys P, Van Damme J, Opdenakker G.

Eur J Immunol. 2008 Apr;38(4):1082-95. doi: 10.1002/eji.200737906.

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